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anti-Human MAPK3 Anticorps:
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Human Polyclonal MAPK3 Primary Antibody pour WB - ABIN1881527
Munshi, Wu, Mukhopadhyay, Ottaviano, Sassano, Koblinski, Platanias, Stack et al.: Differential regulation of membrane type 1-matrix metalloproteinase activity by ERK 1/2- and p38 MAPK-modulated tissue inhibitor of metalloproteinases 2 expression controls transforming growth ... dans The Journal of biological chemistry 2004
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Human Polyclonal MAPK3 Primary Antibody pour ICC, IHC (p) - ABIN3044377
Li, Zhu, Liu, Liu, Wang, Xiong, Shen, Hu, Zheng: ZFX knockdown inhibits growth and migration of non-small cell lung carcinoma cell line H1299. dans International journal of clinical and experimental pathology 2013
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Chicken Monoclonal MAPK3 Primary Antibody pour IF, IP - ABIN967700
Ackerley, Grierson, Brownlees, Thornhill, Anderton, Leigh, Shaw, Miller: Glutamate slows axonal transport of neurofilaments in transfected neurons. dans The Journal of cell biology 2000
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Chicken Monoclonal MAPK3 Primary Antibody pour IF, IP - ABIN967701
Aguirre-Ghiso, Liu, Mignatti, Kovalski, Ossowski: Urokinase receptor and fibronectin regulate the ERK(MAPK) to p38(MAPK) activity ratios that determine carcinoma cell proliferation or dormancy in vivo. dans Molecular biology of the cell 2001
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Chicken Polyclonal MAPK3 Primary Antibody pour ICC, FACS - ABIN361833
Boulton, Gregory, Cobb: Purification and properties of extracellular signal-regulated kinase 1, an insulin-stimulated microtubule-associated protein 2 kinase. dans Biochemistry 1991
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Chicken Monoclonal MAPK3 Primary Antibody pour IF, IP - ABIN967952
Boulton, Cobb: Identification of multiple extracellular signal-regulated kinases (ERKs) with antipeptide antibodies. dans Cell regulation 1991
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Mouse (Murine) Polyclonal MAPK3 Primary Antibody pour IHC, WB - ABIN3020725
Fan, Zhang, Hu, Li, Zhang: Activation of AKT/ERK confers non-small cell lung cancer cells resistance to vinorelbine. dans International journal of clinical and experimental pathology 2014
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Human Polyclonal MAPK3 Primary Antibody pour WB - ABIN2801963
McLaughlin, Kumar, McDonnell, Van Horn, Lee, Livi, Young: Identification of mitogen-activated protein (MAP) kinase-activated protein kinase-3, a novel substrate of CSBP p38 MAP kinase. dans The Journal of biological chemistry 1996
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Human Polyclonal MAPK3 Primary Antibody pour IF (p), IHC (p) - ABIN744143
Zhao, Zhang, Liu, Zhang, Hao, Li, Chen, Shen, Tang, Min, Meng, Wang, Yi, Zhang: Hydrogen Sulfide and/or Ammonia Reduces Spermatozoa Motility through AMPK/AKT Related Pathways. dans Scientific reports 2016
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Human Monoclonal MAPK3 Primary Antibody pour WB - ABIN1539804
Lei, Chen, Huang, Wu, Lin, Lai: Proteomic analysis of the effect of extracellular calcium ions on human mesenchymal stem cells: Implications for bone tissue engineering. dans Chemico-biological interactions 2015
Data report that MPK3/MPK6 and their substrate ERF6 promote the biosynthesis of IGSs and the conversion of I3G to 4MI3G, a target of PEN2/PEN3-dependent chemical defenses in plant immunity.
Data show that the protein kinases MPK3 and MPK6 (Montrer MAPK6 Anticorps) can both interact with SPOROCYTELESS/NOZZLE (SPL (Montrer SGPL1 Anticorps)) in vitro and in vivo and can phosphorylate the SPL (Montrer SGPL1 Anticorps) protein in vitro.
MKK4 (Montrer MAP2K4 Anticorps), MKK5 (Montrer MAP2K5 Anticorps), MKK7 (Montrer MAP2K7 Anticorps), and MKK9, are responsible for the activation of MPK3 and MPK6 (Montrer MAPK6 Anticorps) by melatonin, indicating that melatonin-mediated innate immunity is triggered by MAPK (Montrer MAPK1 Anticorps) signaling through MKK4 (Montrer MAP2K4 Anticorps)/5/7/9-MPK3/6 cascades.
Phosphatase AP2C1, as well as AP2C1-targeted MPK3 and MPK6 (Montrer MAPK6 Anticorps), are important regulators of plant-nematode interaction, where the co-ordinated action of these signalling components ensures the timely activation of plant defence.
Results demonstrated the contribution of MPK3 and MPK6 (Montrer MAPK6 Anticorps) to riboflavin-induced resistance.
These results indicate that the MVB pathway is positively regulated by pathogen-responsive MPK3/6 through LIP5 phosphorylation and plays a critical role in plant immune system
MKK3-MPK6 is activated by blue light in a MYC2-dependent manner.
MPK3 and MPK6 (Montrer MAPK6 Anticorps) target a subclass of 'VQ-motif'-containing proteins to regulate immune responses.
MicroRNA biogenesis factor DRB1 (Montrer EPHX1 Anticorps) is a phosphorylation target of mitogen activated protein kinase (Montrer MAPK1 Anticorps) MPK3 in both rice and Arabidopsis
MPK3 represses the constitutive and flg22-induced expression of defence genes in Arabidopsis thaliana.
MAPK3/1 is involved in luteinizing hormone-mediated decrease of C-type natriuretic peptide (Montrer NPPC Anticorps) and this process is related to the EGFR (Montrer EGFR Anticorps) and MAPK3/1 signal pathways
Chronic hypoxia induces Egr-1 (Montrer EGR1 Anticorps) via activation of ERK1/2 (Montrer MAPK1/3 Anticorps) and contributes to pulmonary vascular remodeling.
ER Ca(2 (Montrer CA2 Anticorps)+) release enhances eNOS (Montrer NOS3 Anticorps) Ser (Montrer SIGLEC1 Anticorps)-635 phosphorylation and function via ERK1/2 (Montrer MAPK1/3 Anticorps) activation.
Thrombospondin 1 (Montrer THBS1 Anticorps), fibronectin (Montrer FN1 Anticorps), and vitronectin (Montrer VTN Anticorps) are differentially dependent upon RAS, ERK1/2 (Montrer MAPK1/3 Anticorps), and p38 (Montrer MAPK14 Anticorps) for induction of vascular smooth muscle cell chemotaxis.
results suggest that Nav1.7-Ca2+ influx-protein kinase C-alpha pathway activated ERK1/ERK2 and p38, which increased phosphorylation of glycogen synthase kinase-3beta, decreasing tau phosphorylation
These data suggest that Gab1 (Montrer GAB1 Anticorps)-ERK1/2 (Montrer MAPK1/3 Anticorps) binding and their nuclear translocation play a crucial role in Egr-1 (Montrer EGR1 Anticorps) nuclear accumulation.
data demonstrate that hypoxia-induced adventitial fibroblast proliferation requires activation and interaction of PI3K, Akt (Montrer AKT1 Anticorps), mTOR (Montrer FRAP1 Anticorps), p70S6K (Montrer RPS6KB1 Anticorps), and ERK1/2 (Montrer MAPK1/3 Anticorps).
This study demonstrates for the first time that cyclic mechanical stretch induces the proliferation of bovine satellite cells and suppresses their myogenic differentiation through the activation of ERK (Montrer MAPK1 Anticorps).
findings indicate that exposure to DHEA, at concentrations found in human blood, causes vascular endothelial proliferation by a plasma membrane-initiated activity that is Gi/o and ERK1/2 (Montrer MAPK1/3 Anticorps) dependent.
Results suggest that estrogen receptors and the ERK1/2 (Montrer MAPK1/3 Anticorps) signaling pathway are involved in the anti-apoptotic action of LY117018 in vascular endothelial cells.
The results suggest that the MPK-1 (Montrer MAPK1 Anticorps)/ERK (Montrer MAPK1 Anticorps) regulatory network, including FBF-1 (Montrer FBF1 Anticorps), FBF-2, and LIP-1 (Montrer CENPJ Anticorps), controls the number of sperm by regulating the timing of the sperm-oocyte switch in C. elegans.
Cek2 (Montrer FGFR3 Anticorps) has a cryptic role in cell-wall biogenesis and its role is not entirely redundant to Cek1.
knockdown of SUV420H1 (Montrer SUV420H1 Anticorps) reduced phosphorylated ERK1 and total ERK1 proteins, and interestingly suppressed ERK1 at the transcriptional level
Secreted aspartic protease-mediated proteolytic cleavage of Msb2 is required for activation of the Cek1 mitogen activated protein kinase (Montrer MAPK1 Anticorps) pathway in response to environmental cues.
The authors propose that a Msb2, Cek1 and Ace2 signalling pathway addresses PMT genes as downstream targets and that different modes of regulation have evolved for PMT1 and PMT2/PMT4 genes.
Msb2 is involved in the transmission of the signal toward Cek1 mediated by the Cdc42 (Montrer CDC42 Anticorps) GTPase (Montrer RACGAP1 Anticorps).
ERK1/2 (Montrer MAPK1/3 Anticorps)-Akt1 (Montrer AKT1 Anticorps) crosstalk regulates arteriogenesis in mice and zebrafish.
eena (Montrer SH3GL1 Anticorps) plays an important role in the development of the myeloid cell through activation of the ERK1/ERK2 (Montrer MAPK1 Anticorps) pathway
ERK1 and ERK2 (Montrer MAPK1 Anticorps) target common and distinct gene sets, confirming diverse roles for these kinases during embryogenesis; for ERK1 different specific genes involved in dorsal-ventral patterning and subsequent embryonic cell migration were identified.
These results demonstrate that induction of Hsp70 (Montrer HSPA1A Anticorps) in response to heat stress is dependent on ERK (Montrer MAPK1 Anticorps) activation in Pac2 (Montrer PSMG2 Anticorps) cells.
Data define distinct roles for ERK1 and ERK2 (Montrer MAPK1 Anticorps) in developmental cell migration processes during zebrafish embryogenesis.
GLUL (Montrer GLUL Anticorps) knockdown markedly inhibited the p38 MAPK (Montrer MAPK14 Anticorps) and ERK1/ERK2 (Montrer MAPK1 Anticorps) signaling pathways in cultured breast cancer cells and reduces their proliferation.
These results suggested that HOXB7 (Montrer HOXB7 Anticorps) stimulates ERK1/2 (Montrer MAPK1/3 Anticorps) phosphorylation and provided evidence that HOXB7 (Montrer HOXB7 Anticorps), besides its role in transcriptional regulation, also promotes cell motility and invasiveness.
High ERK1 expression is associated with castration-resistant prostate cancer.
combined use of butyrate and highly specific Syk (Montrer SYK Anticorps) inhibitor BAY61-3606 does not enhance differentiation and apoptosis of colonocytes. Instead, BAY completely abolishes butyrate-induced differentiation and apoptosis in a Syk (Montrer SYK Anticorps)- and ERK1/2 (Montrer MAPK1/3 Anticorps)-dependent manner.
new findings indicating that canonical FGFR (Montrer FGFR2 Anticorps)-ERK1/2 (Montrer MAPK1/3 Anticorps) signaling entrapped hBMSCs in a pre-committed state and arrested further maturation of committed precursors.
mutually exclusive transcriptional regulation by AP-1 (cjun (Montrer JUN Anticorps)/cfos) and non-canonical NF-kappaB (Montrer NFKB1 Anticorps) (RelB (Montrer RELB Anticorps)/p52 (Montrer FKBP4 Anticorps)) downstream of MEK (Montrer MAP2K1 Anticorps)-ERK (Montrer EPHB2 Anticorps) and NIK (Montrer MAP3K14 Anticorps)-IKK-alpha (Montrer CHUK Anticorps)-NF-kappaB2 (p100 (Montrer CUX1 Anticorps)) phosphorylation, respectively was responsible for persistent Ccl20 (Montrer CCL20 Anticorps) expression in the colonic cells.
LPS (Montrer IRF6 Anticorps)-activated ERK1,2 was at least partly involved in the observed effects on periodontal ligament stem cell differentiation capacity, acquisition of myofibroblastic attributes, and changes of their immunomodulatory features.
The findings indicate that ERK (Montrer EPHB2 Anticorps) and JNK (Montrer MAPK8 Anticorps) signaling pathways, as well as NF-kappaB (Montrer NFKB1 Anticorps)-mediated signaling are important contributors to the pathogenesis of Kashin-Beck disease.
The antitumor activity of scopoletin may be due to its strong anti-angiogenic effect, which may be mediated by its effective inhibition of ERK1, VEGF-A (Montrer VEGFA Anticorps), and FGF-2 (Montrer FGF2 Anticorps).
High ERK1 expression is associated with melanoma.
Cortical neuron-specific deletion of extracellular signal-regulated kinases Erk1 or Erk2 (Montrer MAPK1 Anticorps) significantly increased the duration of wakefulness.
pERK1/2 is a regulator of CD44 (Montrer CD44 Anticorps) expression, and increased CD44 (Montrer CD44 Anticorps) expression leads to a pro-sclerotic and migratory parietal epithelial cell phenotype in focal segmental glomerulosclerosis.
mmLDL increased the serum concentrations and expression of ICAM-1 (Montrer ICAM1 Anticorps) and VCAM-1 (Montrer VCAM1 Anticorps) by activating the ERK1/2 (Montrer MAPK1/3 Anticorps) pathway, resulting in the expression of ETB (Montrer EDNRB Anticorps) receptors and the enhancement of contractile function in vascular smooth muscle.
Angiotensin II regulates dendritic cells through activation of p65 NF-kappaB (Montrer NFkBP65 Anticorps), ERK1, ERK2 (Montrer MAPK1 Anticorps) and STAT1 (Montrer STAT1 Anticorps) pathways.
MAPK3/1 participates in primordial follicle activation through mTORC1-KITL (Montrer KITLG Anticorps) signaling.
At low oxLDL levels LOX-1 (Montrer OLR1 Anticorps) activates the protective Oct-1 (Montrer POU2F1 Anticorps)/SIRT1 (Montrer SIRT1 Anticorps) pathway, while at higher levels of the lipoprotein switches to the thrombogenic ERK1/2 (Montrer MAPK1/3 Anticorps) pathway.
Studies indicate that progesterone receptor (Montrer PGR Anticorps) transgenic (Pgrcre/+) mitogen inducible gene 6 (Mig (Montrer CXCL9 Anticorps)-6over) phosphatase and tensin homolog (Montrer PTEN Anticorps) protein (Ptenf/f) knockout mice exhibited an increase of phospho-ERK1/2 (Montrer MAPK1/3 Anticorps) and its target genes.
Gpr182 reduction led to increased activation of ERK1/2 in basal and challenge models, demonstrating a potential role for this orphan GPCR in regulating the proliferative capacity of the intestine.
ERK1 underexpression is associated with obesity.
retinoschisin (Montrer RS1 Anticorps) is a novel regulator of MAP kinase (Montrer MAPK1 Anticorps) signalling and exerts an anti-apoptotic effect on retinal cells.
Early activation of MAPK p44/42 is involved in deoxynivalenol -induced disruption of intestinal barrier function and tight junction network signaling.
Pseudorabies virus glycoprotein gE-mediated ERK 1/2 phosphorylation also occurs in epithelial cells and in these cells, gE-mediated ERK 1/2 signaling is associated with degradation of the pro-apoptotic protein Bim (Montrer BCL2L11 Anticorps).
Treatment with ERK (Montrer MAPK1 Anticorps) inhibitors or ERK1/2 (Montrer MAPK1/3 Anticorps) knockdown significantly suppressed porcine epidemic diarrhea virus progeny production.
This study reveals a new function of the gE glycoprotein of pseudorabies virus and suggests that pseudorabies virus, through activation of ERK1/2 (Montrer MAPK1/3 Anticorps) signaling, has a substantial impact on T cell behavior.
CSF2 (Montrer CSF2 Anticorps) stimulates proliferation of trophectoderm cells by activation of the PI3K-and ERK1/2 (Montrer MAPK1/3 Anticorps) MAPK (Montrer MAPK1 Anticorps)-dependent MTOR (Montrer FRAP1 Anticorps) signal transduction cascades.
PGRN (Montrer GRN Anticorps) inhibits adipogenesis in porcine preadipocytes partially through ERK (Montrer MAPK1 Anticorps) activation mediated PPARgamma (Montrer PPARG Anticorps) phosphorylation.
Porcine circovirus type 2 (PCV2) might induce autophagy via the AMPK (Montrer PRKAA1 Anticorps)/ERK (Montrer MAPK1 Anticorps)/TSC2 (Montrer TSC2 Anticorps)/mTOR (Montrer FRAP1 Anticorps) signaling pathway in the host cells, representing a pivotal mechanism for PCV2 pathogenesis
Data show that proinflammatory cytokines induction was ERK1/2 (Montrer MAPK1/3 Anticorps) and JNK1 (Montrer MAPK8 Anticorps)/2 dependent.
Saccharomyces cerevisiae inhibits the Enterotoxigenic Escherichia coli-induced expression of pro-inflammatory transcripts and this inhibition was associated to a decrease of ERK1/2 (Montrer MAPK1/3 Anticorps) and p38 MAPK (Montrer MAPK14 Anticorps) phosphorylation
ERK1 phosphorylation in response to Insulin-like Growth Factor-1 (Montrer IGF1 Anticorps) does not require activation of the Insulin-like Growth Factor-1 receptor tyrosine kinase (Montrer IGF1R Anticorps)
The protein encoded by this gene is a member of the MAP kinase family. MAP kinases, also known as extracellular signal-regulated kinases (ERKs), act in a signaling cascade that regulates various cellular processes such as proliferation, differentiation, and cell cycle progression in response to a variety of extracellular signals. This kinase is activated by upstream kinases, resulting in its translocation to the nucleus where it phosphorylates nuclear targets. Alternatively spliced transcript variants encoding different protein isoforms have been described.
extracellular signal-regulated kinase 1
, likely protein kinase
, MAP kinase isoform p44
, MAPK 1
, extracellular signal-related kinase 1
, insulin-stimulated MAP2 kinase
, microtubule-associated protein 2 kinase
, MAP kinase 3
, p44 MAP kinase
, pp42/MAP kinase
, MAP kinase 1
, MAPK 3
, mitogen-activated 3
, mitogen-activated protein kinase 1
, mitogen-activated protein kinase 3
, extracellular signal-regulated kinase-1