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anti-Human Tumor Necrosis Factor Anticorps:
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Rat (Rattus) Polyclonal Tumor Necrosis Factor Primary Antibody pour WB - ABIN3042522
Zhang, Liu, Goldstein, Li, Ge, He, Fei, Wang, Ruiz: Role of the JAK/STAT signaling pathway in the pathogenesis of acute myocardial infarction in rats and its effect on NF-?B expression. dans Molecular medicine reports 2013
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Mouse (Murine) Polyclonal Tumor Necrosis Factor Primary Antibody pour ELISA, IHC (p) - ABIN3042817
Liu, Shan, Dong, Liu, Ma, Liu: Combined early fluid resuscitation and hydrogen inhalation attenuates lung and intestine injury. dans World journal of gastroenterology 2013
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Mouse (Murine) Polyclonal Tumor Necrosis Factor Primary Antibody pour WB - ABIN3043313
Qin, Ma, Yang, Hu, Zhou, Fu, Tian, Liu, Xu, Shen: A Triterpenoid Inhibited Hormone-Induced Adipocyte Differentiation and Alleviated Dexamethasone-Induced Insulin Resistance in 3T3-L1 adipocytes. dans Natural products and bioprospecting 2015
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Mouse (Murine) Polyclonal Tumor Necrosis Factor Primary Antibody pour CyTOF, ELISA (Capture) - ABIN4900489
Ramkhelawon, Hennessy, Ménager, Ray, Sheedy, Hutchison, Wanschel, Oldebeken, Geoffrion, Spiro, Miller, McPherson, Rayner, Moore: Netrin-1 promotes adipose tissue macrophage retention and insulin resistance in obesity. dans Nature medicine 2014
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Human Monoclonal Tumor Necrosis Factor Primary Antibody pour - ABIN1383955
Hervé, Racadot, Wijdenes, Flesch, Tiberghien, Bordigoni, Holler, Powles, Bourdeau, Wilmer: Monoclonal anti TNF alpha antibody in the treatment of acute GvHD refractory both to corticosteroids and anti IL-2 R antibody. dans Bone marrow transplantation 1991
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Human Polyclonal Tumor Necrosis Factor Primary Antibody pour ICC, IF - ABIN446837
Kwon, Jung, Hwang, Choi: Evaluation of Bronchiolar and Alveolar Cell Injuries Induced by Short- and Long-term Exposure to Sidestream Smoke. dans Korean journal of pathology 2012
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Human Polyclonal Tumor Necrosis Factor Primary Antibody pour IF (p), IHC (p) - ABIN677318
Morishita, Oku, Horie, Tonari, Kida, Okubo, Sugiyama, Takai, Hara, Ikeda: Systemic simvastatin rescues retinal ganglion cells from optic nerve injury possibly through suppression of astroglial NF-?B activation. dans PLoS ONE 2014
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Human Polyclonal Tumor Necrosis Factor Primary Antibody pour IHC (p), IHC - ABIN153474
Roth, Städler, Lemann, Hösli, Simon, Simon: Distinct eosinophil cytokine expression patterns in skin diseases - the possible existence of functionally different eosinophil subpopulations. dans Allergy 2011
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Chicken Polyclonal Tumor Necrosis Factor Primary Antibody pour IHC, ELISA - ABIN1582141
Choi, Lim, Hwang, Lee, Cho, Kim: Anti-ischemic and anti-inflammatory activity of (S)-cis-verbenol. dans Free radical research 2010
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Human Monoclonal Tumor Necrosis Factor Primary Antibody pour FACS - ABIN4898910
Glässner, Eisenhardt, Krämer, Körner, Coenen, Sauerbruch, Spengler, Nattermann: NK cells from HCV-infected patients effectively induce apoptosis of activated primary human hepatic stellate cells in a TRAIL-, FasL- and NKG2D-dependent manner. dans Laboratory investigation; a journal of technical methods and pathology 2012
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Serum TNF-alpha and IL-6 (Montrer IL6 Anticorps) levels might be negatively correlated with cognitive function, and we suspect that serum IL-6 (Montrer IL6 Anticorps) levels could be biomarkers for AD(Alzheimer's disease).
TNF-alpha-308 showed no statistically signi fi cant al- lelic or genotypic association with Type 2 Diabetes Mellitus.
VCAM-1 (Montrer VCAM1 Anticorps) knockdown reduced TNFalpha-induced HUVEC migration and tube formation.
The plasma levels of interleukin (IL)-1beta (Montrer IL1B Anticorps), tumor necrosis factor (TNF)-alpha, tissue inhibitor of metalloproteinases (TIMP)-1 (Montrer TIMP1 Anticorps) are increased in myelofibrosis (MF) patients.
an increase in TNF-alpha concentration in CSF (Montrer CSF2 Anticorps) preceded CNS relapse in children with ALL
suggest that the low blood levels of tumor necrosis factor-alpha mRNA in subjects with autism spectrum disorder might be due to impaired social interaction in early childhood
IL-17 (Montrer IL17A Anticorps) and TNF-alpha induced IL-6 (Montrer IL6 Anticorps) and IL-8 (Montrer IL8 Anticorps) expression, although IL-17 (Montrer IL17A Anticorps) alone had a limited effect on endothelial cells compared to synoviocytes. Supernatants from activated T-helper clones also induced IL-6 (Montrer IL6 Anticorps) and IL-8 (Montrer IL8 Anticorps) expression but with discrepancies between endothelial cells and synoviocytes.
we genotyped these polymorphisms within the TNF-a gene in 256 Brazilian subjects infected with Schistosoma mansoni, with different patterns of periportal fibrosis. The genotype (-308) AA was associated with a significant increase in the risk to advanced periportal fibrosis. There was no association between average serum levels of TNF-a and (-G308A) TNF-a polymorphism.
The present study reveals that TLR4 (Montrer TLR4 Anticorps) drives the production of TNFalpha and IL-10 (Montrer IL10 Anticorps) induced by L. braziliensis amastigotes and that the parasites decrease TLR4 (Montrer TLR4 Anticorps) expression on monocyte surface.
Tumor necrosis factor gene polymorphisms in adult patients with autoimmune hemolytic anemia.
Our findings demonstrate that TNF-alpha plays a pivotal role in HF-mediated neuroinflammation and associated alterations of cortical dendritic spine density and has the potential to reveal novel treatment strategies for HF-associated memory deficits.
the present study assessed the combined impacts of estrogens and bacterial infection on the insulin (Montrer INS Anticorps)-like growth factors (IGF) and tumor-necrosis factor (TNF)-alpha.
The expression of miR (Montrer MYLIP Anticorps)-125b-1 regulated by NF-kappaB (Montrer NFKB1 Anticorps) has been reported in diverse cell types under various stimuli, this study found that its expression was also significantly regulated by NF-kappaB (Montrer NFKB1 Anticorps) in TNFalpha-stimulated HeLa and HepG2 cells
In the Han Chinese population, the allele 'A' of TNF-alpha -308 may increase the risk for osteoarthritis (OA), whereas TNF-alpha -238 polymorphisms do not play a role in OA patients.
Letter: serum TNF-alpha was not elevated in chronic spontaneous urticaria.
Metastatic cells can escape the proapoptotic effects of TNF-alpha through increased autocrine IL-6 (Montrer IL6 Anticorps)/STAT3 (Montrer STAT3 Anticorps) signaling
The predicted 3D structures of IL-1beta (Montrer IL1B Anticorps), TNF-alpha, and IL-8 (Montrer IL8 Anticorps) from Oncorhynchus mykiss, Dicentrarchus labrax, and Chionodraco hamatus are reported and the differences between them and their mammalian counterparts investigated.
The mitochondrial dysfunction may be caused by increased brain levels of TNFalpha without physiological stress but will be exacerbated after middle cerebral artery occlusion.
TNF-alpha is released rapidly in trout macrophages after stimulation with lipopolysaccharide and zymosan, and its secretion is dependent on TACE (Montrer ADAM17 Anticorps)-like activity that is regulated by ERK (Montrer MAPK1 Anticorps), JNK (Montrer MAPK8 Anticorps) and p38MAPK (Montrer MAPK14 Anticorps) signaling pathways.
Recombinant, as well as native trout TNF-alpha, directly stimulates glucose uptake in trout muscle cells and provide evidence, for the first time in nonmammalian vertebrates, for a potential regulatory role of TNF-alpha in skeletal muscle metabolism.
Lactosylceramide-Induced Phosphorylation Signaling to Group IVA Phospholipase A2 (Montrer YWHAZ Anticorps) via Reactive Oxygen Species in Tumor Necrosis Factor-alpha-Treated Cells.
The current study demonstrated that honey can stimulate or suppress the mRNA expression of some pro-inflammatory cytokines in mice brains. Furthermore, honey suppresses the TNF-alpha mRNA expression in the presence of T. gondii infection but it stimulates the IL-1beta (Montrer IL1B Anticorps) and IL-6 (Montrer IL6 Anticorps) mRNA expression. Treatment of the mice with honey reduces parasite multiplication in the brain.
aerobic interval training enhanced the anti-inflammatory indices IL-10 (Montrer IL10 Anticorps)/TNF-alpha ratio and IL-15 (Montrer IL15 Anticorps) expression in skeletal muscle in tumor-bearing mice.
findings suggest that activation of Tnf-Aicda (Montrer AICDA Anticorps) axis and co-inhibitory signals to T cells in coordination with Th1 (Montrer HAND1 Anticorps)-type immunity has critical roles in the immune response against Hepatitis B virus infection
Taken together, we speculate that DT-13 inhibits endothelium vascular inflammation through regulating nitric oxide production and the expression of ROS (Montrer ROS1 Anticorps), TNFR (Montrer TNFRSF1A Anticorps), IL-8 (Montrer IL8 Anticorps), MCP-1 (Montrer CPT1B Anticorps), which are associated with inflammation.
TNF signalling is required for the expansion and differentiation of pathogenic IFNgamma+CD4 (Montrer CD4 Anticorps)+ T cells that promote the irreversible loss of bone marrow function.
Drugs targeting XIAP (Montrer XIAP Anticorps) and cIAP1 (Montrer BIRC2 Anticorps)/2 may be effective for osteosarcoma patients whose tumors express abundant RIPK1 (Montrer RIPK1 Anticorps) and contain high levels of TNFalpha.
Taken together, we indicated that anti-IL-6 (Montrer IL6 Anticorps) and anti-TNF-alpha therapy prevent intestinal permeability induced by intestinal inflammation
Following vasectomy, IL1alpha (Montrer IL1A Anticorps), IL1beta (Montrer IL1B Anticorps), IL1ra (Montrer IL1RN Anticorps), IL10 (Montrer IL10 Anticorps), and TNF-alpha may mediate immune reaction in whole epididymis, whereas IL6 (Montrer IL6 Anticorps) and TGF-beta1 (Montrer TGFB1 Anticorps) may mediate regionally different immune response primarily in the lower part of epididymis.
Stat3 (Montrer STAT3 Anticorps) mediates the expression of iNOS (Montrer NOS2 Anticorps) to promote IFNgamma/TNFalpha-induced muscle atrophy.
Data suggest that luteolytic factors (such as TNFa, interferon gamma (Montrer IFNG Anticorps), and PGF2a) control expression of MMP1 (Montrer MMP1 Anticorps), other matrix metalloproteinases, and tissue inhibitors of metalloproteinase in cultured luteal cells.
These results suggest that polymorphism of the TNFalpha-824 A>G gene and mTNFalpha protein expression play an important role in the pathogenesis of enzootic bovine leukosis.
These results suggest that the endometrium might lower the TNF concentration in the blastocyst by (1) regulating TNF secretion into the uterine fluid and (2) inducing decreased TNF and TNFR2 (Montrer TNFRSF1B Anticorps) mRNA transcription in the embryo.
There was a significant association between the proviral load and a low frequency of the G/G genotype of TNF-alpha at position -824.
SNP in the TNF-alpha gene affects immune function and reproductive performance in dairy cows.
Studied genotypic and expression profiling of partial TNF-alpha gene and its association with mastitis susceptibility in 129 crossbred cattle.
Messenger RNA and protein levels of prostaglandin (PG) E synthase (PGES (Montrer PTGES Anticorps)), PGF2alpha receptor (PGFR), tumor necrosis factor-alpha (TNF) and Fas (Montrer FAS Anticorps) were found to be higher in the corpus luteum of pregnancy than in corpus luteum of the cycle.
TNF-alpha up-regulates NaV1.7 (Montrer SCN9A Anticorps) mRNA in both adrenal chromaffin cells and dorsal root ganglia (DRG) neurons, highlighting the peripheral nociceptive mechanism of TNF-alpha
These results provide evidence for a high prevalence of subclinical endometritis in repeat breeding cows as well as the involvement of TNFalpha and iNOS (Montrer NOS2 Anticorps) pathways in the regulation of this pathological condition.
Exposure to follicular fluid transiently increased the transcript levels of IL8 (Montrer IL8 Anticorps) and PTGS2 (Montrer PTGS2 Anticorps), and decreased the expression of SOD2 (Montrer SOD2 Anticorps), GPX3 (Montrer GPX3 Anticorps), DAB2 (Montrer DAB2 Anticorps), and NR3C1 (Montrer NR3C1 Anticorps). TNF and IL6 (Montrer IL6 Anticorps) levels were also decreased while those of NAMPT (Montrer NAMPT Anticorps) were unaffected.
these findings provide the direct evidence that ADAM17 (Montrer ADAM17 Anticorps) cleaves porcine TNFalpha, which represents a new view for identifying potential therapeutic targets in anti-porcine reproductive and respiratory syndrome virus therapy
TMZ pretreatment effectively reduced the myocardial damage caused by CME via inhibiting the PDCD4 (Montrer PDCD4 Anticorps)/NF-kappaB (Montrer NFKB1 Anticorps)/ TNF-alpha pathway in cardiomyocytes.
TNF-alpha, secreted from activated Monocytes, mediates the downregulation of OTX2 (Montrer OTX2 Anticorps) and essential retinal pigment epithelium genes.
TNF-alpha induced MMP-13 (Montrer MMP13 Anticorps) expression by condylar cells might be involved in the degradation of the juvenile condyle.
TNF-alpha was able to promote theca interna cell proliferation. Our results suggest that TNF-alpha might play a role in hyperandrogenism, cortex thickness, and the increased ovary volume observed in polycystic ovaries.
This study showed that the -791(C-->T) mutation of the TNF-alpha gene could be considered an important potential genetic marker of enterotoxigenic Escherichia coli F18 (Montrer MAMLD1 Anticorps) resistance.
Both Nsp1beta and Nsp11 of porcine reproductive and respiratory syndrome virus were demonstrated to be responsible for the inhibitory effect on TNF-alpha production in pulmonary alveolar macrophages.
Study presents evidence demonstrating a single species of exotoxin ApxI, derived from A. pleuropneumoniae serotype 10, induces the expression and production of proinflammatory cytokines IL-1beta (Montrer IL1B Anticorps), IL-8 (Montrer IL8 Anticorps) and TNF-alpha in porcine alveolar macrophages.
the role of miR (Montrer MYLIP Anticorps)-181a in adipocyte differentiation by regulation of TNF-alpha
The NOS (Montrer NOS Anticorps) inhibitor, L-NMMA, significantly suppressed the combined effects of HT and CORM-2 on TNFalpha-triggered NFkappaBp65 phosphorylation as well as decreased cell viability.
Using in vivo, time-lapse imaging we show that as centrally-projecting pioneer axons from dorsal root ganglia (DRG (Montrer COL4A1 Anticorps)) enter the spinal cord, they initiate expression of the cytokine TNFalpha. This induction coincides with ensheathment of these axons by associated glia via a TNF receptor 2 (TNFR2 (Montrer TNFRSF1B Anticorps))-mediated process.
The results reveal a crucial role for TNFalpha/TNFR2 (Montrer TNFRSF1B Anticorps) axis in the protection of the skin against DUOX1 (Montrer DUOX1 Anticorps)-mediated oxidative stress.
proinflammatory cytokines IL-1 (Montrer IL1A Anticorps)/TNFalpha trigger a novel antiviral mechanism involving AID to regulate host cell permissiveness to HBV infection.
TNF alpha is the first molecule identified that is produced by dying retinal neurons and is necessary to induce Muller glia to proliferate in the zebrafish retinal regeneration response.
The data suggest that Mycobacterium marinum SecA2 modulates adaptive immunity to promote granuloma stability, perhaps through induction of tumor necrosis factor alpha.
The data suggest that during infectious pancreatic necrosis virus infection, the expression of cytokines and metalloproteinases might be initiated through the TNFalpha/NF-kappaB (Montrer NFKB1 Anticorps)-mediated pathway.
Infectious pancreatic necrosis virus triggers two death pathways via up-stream induction of the pro-inflammatory cytokine TNFalpha.
TNF is not required for tuberculous granuloma formation, but maintains granuloma integrity indirectly by restricting mycobacterial growth within macrophages and preventing their necrosis.
The main proinflammatory effects of fish TNF-alpha are mediated through the activation of endothelial cells and point to the complexity of the evolution that has taken place in the regulation of innate immunity by cytokines.
Antisense knockdown of tnfa rescued hepatic steatosis and liver degeneration in dtp (Montrer AHCY Anticorps) larvae, whereas the overexpression of tnfa and the hepatic phenotype were unchanged in dtp (Montrer AHCY Anticorps) larvae.
These interactions result in the induction of the TNF signaling pathway, activation of apoptosis, and DNA-damage stress response.
Findings showed that both mucosal compartments harbor similar percentages of memory CD4 (Montrer CD4 Anticorps)(+) T cells and displayed comparable cytokine TNF-alpha responses to mitogenic stimulations prior to infection.
Primary role for IL-1beta (Montrer IL1B Anticorps) and TNF-alpha in the triggering of preterm labor associated with inflammation or infection.
The results obtained showed that IL-6 (Montrer IL6 Anticorps) expression in adipose tissue biopsies derived from animals with equine metabolic syndrome was enhanced while TNF-alpha levels of both groups were comparable.
Data indicate that tumor necrosis factor-alpha production is closely related to ovarian steroid actions.
These results indicate that in equine corpus luteum, cytokines TNF, IFNG (Montrer IFNG Anticorps) and FASL (Montrer FASL Anticorps) regulate nitric oxide activity, via eNOS (Montrer NOS3 Anticorps) expression modulation.
These data show the presence of cytokines TNF and IFNG (Montrer IFNG Anticorps), and their receptors, in the equine corpus luteum and indicate their potential involvement in regulation of luteal function.
The contribution of bronchial epithelium to airway inflammation, with focus on mRNA and protein expression of IL-6 (Montrer IL6 Anticorps), IL-10 (Montrer IL10 Anticorps) and TNF-alpha, in horses with recurrent airway obstruction during exacerbation and in remission is reported.
TUNEL staining was positively correlated with TNF-a protein expression. Our findings suggest that apoptosis can be induced in the vocal fold epithelium after 120min of modal intensity phonation. In contrast, shorter durations of vibration exposure do not result in apoptosis signaling.
Inflammatory factors such as TNF-alpha can stimulate MMP-2 (Montrer MMP2 Anticorps)/9 activity in corneal epithelium cells. This may be a potential manipulating mechanism of MMP expression in the pathogenesis of corneal diseases.
The JNK (Montrer MAPK8 Anticorps) pathway plays an important role in mechanical ventilation-stimulated TNF-alpha expression in alveolar macrophages, but the injury-stimulated IL-8 (Montrer IL8 Anticorps) expression may be regulated by other signaling pathways.
The TNFalpha-evoked Cl- current.
IL-1beta (Montrer IL1B Anticorps) and TNF-alpha expression increases significantly during acute lung injury. Ambroxol combined with low-dose heparin inhibits teh release of IL-1beta (Montrer IL1B Anticorps) and TNF-alpha.
Hypercapnia increased expression of TNFa and decreased expression of NFKB in acute lung injury models.
In the early stages of myocardial ischemia, bone marrow stem cells are mobilized and home to ischemic myocardium with a concomitant increase in expression of cytokines VEGF (Montrer VEGFA Anticorps) and TNFalpha.
Ammonium perchlorate can increase gene expressions of types I, III collagens, TGF-beta(1 (Montrer TGFB1 Anticorps)) and TNF-alpha in lung of rabbits.
TNFalpha may either be directly or indirectly involved in vascular damage following an embolic stroke. Moreover, TNFalpha may mediate some of the detrimental effects of tPA (Montrer PLAT Anticorps) on the vascular compartment
Data indicate that three single-nucleotide polymorphisms were identified in tumor necrosis factor-alpha (TNF-alpha) gene.
Macrophage- and neutrophil-derived TNF-alpha instructs skin langerhans cells to prime antiviral immune responses.
This gene encodes a multifunctional proinflammatory cytokine that belongs to the tumor necrosis factor (TNF) superfamily. This cytokine is mainly secreted by macrophages. It can bind to, and thus functions through its receptors TNFRSF1A/TNFR1 and TNFRSF1B/TNFBR. This cytokine is involved in the regulation of a wide spectrum of biological processes including cell proliferation, differentiation, apoptosis, lipid metabolism, and coagulation. This cytokine has been implicated in a variety of diseases, including autoimmune diseases, insulin resistance, and cancer. Knockout studies in mice also suggested the neuroprotective function of this cytokine.
, TNF, macrophage-derived
, TNF, monocyte-derived
, tumor necrosis factor ligand superfamily member 2
, tumor necrosis factor-alpha
, tumor necrosis factor (TNF superfamily, member 2)
, tumor necrosis factor alpha
, tumor necrosis factor
, tumor-necrosis factor
, tumour necrosis factor
, TNF alpha
, ATP-binding cassette, sub-family F (GCN20), member 1
, tumour necrosis factor alpha
, putative tumour necrosis factor alpha
, LPS-induced TNF-alpha factor homolog
, TNF-alpha factor
, lipid-induced TNF-alpha factor
, lipopolysaccharide-induced tumor necrosis factor-alpha factor homolog
, small integral membrane protein of lysosome/late endosome
, tissue necrosis factor-alpha
, tumor necrosis factor, alpha
, Tumor necrosis factor ligand superfamily member 2
, TNF-alpha 1