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Human VEGFC Kit ELISA pour Sandwich ELISA - ABIN411382
Zhao, Sun, Li: Expression and clinical significance of STAT3, P-STAT3, and VEGF-C in small cell lung cancer. dans Asian Pacific journal of cancer prevention : APJCP 2012
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Human VEGFC Kit ELISA pour Sandwich ELISA - ABIN625369
Yue, Xu, Zhu, Zhang, Liu: Over-expression of nerve growth factor-β in human cholangiocarcinoma QBC939 cells promote tumor progression. dans PLoS ONE 2013
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Human VEGFC Kit ELISA pour Sandwich ELISA - ABIN414935
You, Bei, Cheng, Cheng: Expression of COX-2 and VEGF-C in cholangiocarcinomas at different clinical and pathological stages. dans Genetics and molecular research : GMR 2015
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Mouse (Murine) VEGFC Kit ELISA pour Sandwich ELISA - ABIN415601
Zhang, Wang, Gao, Guo, Chen, Wang, Gao, Rao, Chen: Alternatively activated RAW264.7 macrophages enhance tumor lymphangiogenesis in mouse lung adenocarcinoma. dans Journal of cellular biochemistry 2009
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Mouse (Murine) VEGFC Kit ELISA pour Sandwich ELISA - ABIN365459
Shimizu, Shibata, Shintani, Ishii, Murohara: Therapeutic lymphangiogenesis with implantation of adipose-derived regenerative cells. dans Journal of the American Heart Association 2012
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Human VEGFC Kit ELISA pour Sandwich ELISA - ABIN365328
Tang, Jiang, Guo, Si, Jiao: Expression and significance of vascular endothelial growth factor A and C in leukemia central nervous system metastasis. dans Leukemia research 2013
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Vegfc acts through ERK (Montrer MAPK1 Kits ELISA) to induce sprouting and differentiation of trunk lymphatic progenitors.
data not only reveal a non-canonical function of Mt2 (Montrer MT2 Kits ELISA) in angiogenesis, but also propose Mt2 (Montrer MT2 Kits ELISA) as a novel regulator of vegfc expression.
Vegfc signaling increases mafba (Montrer MAFB Kits ELISA) expression to control downstream transcription
Vegfc is dispensable for facial lymphatic sprouting but not for the complete development of the facial lymphatic network.
In the embryo, phenotypes driven by increased Vegfc are suppressed in the absence of Ccbe1 (Montrer CCBE1 Kits ELISA), and Vegfc-driven sprouting is enhanced by local Ccbe1 (Montrer CCBE1 Kits ELISA) overexpression. Moreover, Vegfc- and Vegfr3 (Montrer FLT4 Kits ELISA)-dependent Erk (Montrer MAPK1 Kits ELISA) signaling is impaired in the absence of Ccbe1 (Montrer CCBE1 Kits ELISA).
Vegfc has an essential role in lymphangiogenesis [review]
The parallel growth of motoneuron axons with the dorsal aorta depends on Vegfc/Vegfr3 (Montrer FLT4 Kits ELISA) signaling in zebrafish.
Vegfc acts in two distinct modes during development: as a paracrine factor secreted from arteries to guide closely associated lymphatic vasculature and as an autocrine factor to drive migratory persistence during angiogenesis.
Rspo1-Wnt-VegfC-Vegfr3 signaling plays a crucial role as an endothelial-autonomous permissive cue for developmental angiogenesis.
Here, we show that vascular endothelial growth factor C (Vegfc), an angiogenic as well as a lymphangiogenic factor, is unexpectedly involved in this process in zebrafish.
VEGF-C and VEGF-C156S genes have roles in the pro-lymphangiogenic growth factor therapy of lymphedema
Transcription of the vascular endothelial growth factor C gene (VEGF-C) and translation of the corresponding protein were significantly up-regulated in swine umbilical vein endothelial cells with classical swine fever virus acute infection.
No difference in bioactivity was detected between porcine relaxin-1 (Montrer RLN1 Kits ELISA) and recombinant human relaxin-2 (Montrer RLN1 Kits ELISA) in either mice or rats.
During progressive ischemia, functional and metabolic benefits of intramyocardial VEGF-C gene transfer were apparent. VEGF-C-induced collateral formation occurred at the site of gene transfer
Studied the effect of recombinant human vascular endothelial growth factor (VEGF)-C on lymphangiogenesis, inflammation, and fibrosis in the mouse kidney using the unilateral ureteral obstruction (UUO); lymphangiogenesis was significantly induced in the UUO+VEGF-C group. In lymphatic endothelial cells, VEGF-C increased the activity and proliferation of such cells and expression of VCAM-1.
In multivariate analysis, only serum VEGF-A (Montrer VEGFA Kits ELISA) correlated to diabetes duration, whereas VEGF-C only correlated to HbA1c and fasting blood glucose.
This study reports that human dendritic cells produce VEGF-C, a cytokine with potent pro-lymphangiogenic activity when stimulated with IFN-gamma (Montrer IFNG Kits ELISA)
Association of coexpressed high levels of VEGF-C and active MMP-9 (Montrer MMP9 Kits ELISA) with lymphatic spreading and local invasiveness of Papillary thyroid carcinoma (PTC (Montrer F9 Kits ELISA)) suggests their potential usefulness as predictive biomarkers of aggressive PTC (Montrer F9 Kits ELISA) behavior.
Data show that VEGF-C, VEGF-D (Montrer Figf Kits ELISA), and VEGFR-3 (Montrer FLT4 Kits ELISA) were expressed in a substantial percentage of breast carcinomas.
By treating LECs with VEGF (Montrer VEGFA Kits ELISA)-C156S and analyzing subsequent changes in gene expression, we identified several 'immediate early (Montrer JUN Kits ELISA)' transcription factors that showed a rapid transient upregulation VEGFR-3 (Montrer FLT4 Kits ELISA) stimulation. these results reveal an important and unanticipated role of HOXD10 (Montrer HOXD10 Kits ELISA) in the regulation of VEGFR-3 (Montrer FLT4 Kits ELISA) signaling in lymphatic endothelial cells, and in the control of lymphangiogenesis and permeability.
In colon cancer samples, there was a positive correlation between the expression of integrin alpha4 and VEGF-C. Integrin alpha4 and VEGF-C were significantly associated with the clinicopathological parameters (LMVD, Duke's stage, and lymph node metastasis). patients with high integrin alpha4 or VEGF-C expression had significantly shorter overall survival and tumor-free survival time.
High VEGFC expression is associated with angiogenesis and lymphangiogenesis.
Adiponectin promoted VEGF-C expression and secretion in human chondrosarcoma cells.
HuR (Montrer ELAVL1 Kits ELISA) plays important roles in determining malignant aggressiveness and outcome in prostate cancer (PCa (Montrer FLVCR1 Kits ELISA)), especially in androgen-independent PCa (Montrer FLVCR1 Kits ELISA) cells, via the regulation of cell proliferation, migration, and expression of VEGF-A (Montrer VEGFA Kits ELISA), -C, and COX-2 (Montrer COX2 Kits ELISA).
lymphangiogenesis is regulated by two distinct proteolytic mechanisms of ligand activation: one in which VEGFC activation by ADAMTS3 (Montrer Adamts2 Kits ELISA) and CCBE1 (Montrer CCBE1 Kits ELISA) spatially and temporally patterns developing lymphatics, and one in which VEGFD (Montrer Figf Kits ELISA) activation by a distinct proteolytic mechanism may be stimulated during inflammatory lymphatic growth
These results reveal an unexpected role for VEGF-C, a major lymphangiogenic growth factor, in the transition to fetal liver erythropoiesis.
Results suggest that interleukin-6 (IL-6 (Montrer IL6 Kits ELISA)) increases VEGF-C induction and lymphangiogenesis may involve, at least in part, Src (Montrer SRC Kits ELISA)-FAK (Montrer PTK2 Kits ELISA)-STAT3 (Montrer STAT3 Kits ELISA) cascade in lymphatic endothelial cells (LECs).
Data show that heparanase-1 (HPA-1 (Montrer HPSE Kits ELISA)) induced shedding of heparan sulfate chain from syndecan-1 (SDC-1 (Montrer SDC1 Kits ELISA)) facilitated the release of vascular endothelial growth factor C (VEGF-C) from SDC-1 (Montrer SDC1 Kits ELISA)/VEGF-C complex into the medium of hepatocarcinoma cell.
Data show that in the MCF-7 breast cancer cell line, only MT1X metallothioneins (MTs (Montrer NEU2 Kits ELISA)) positively correlated with vascular endothelial growth factor C (VEGFC).
The findings in this study strongly suggest the following: i) that VEGF-C promotes the proliferative activity and migratory ability of mesenchymal stem cell ; and ii) VEGF-C and Tgfb (Montrer TGFB1 Kits ELISA) reciprocally regulate mesenchymal stem cell commitment to differentiation into lymphatic endothelial or osteoblastic phenotypes, respectively.
The authors show that VEGF-C is necessary for perinatal lymphangiogenesis, but required for adult lymphatic vessel maintenance only in the intestine.
MT1-MMP (Montrer MMP14 Kits ELISA) directly cleaves LYVE-1 (Montrer LYVE1 Kits ELISA) on lymphatic endothelial cells to inhibit LYVE-1 (Montrer LYVE1 Kits ELISA)-mediated lymphangiogenic responses and restrains the production of VEGF-C.
HA increases lymphangiogenesis in renal fibrosis model and also stimulates vascular endothelial cell growth factor (Montrer FGF1 Kits ELISA)-C production from macrophages through Toll-like receptor 4 (Montrer TLR4 Kits ELISA)-dependent signal pathway
Results showed that the VEGF-C/VEGFR-3 (Montrer FLT4 Kits ELISA) system underlies the protective effect of ischemic preconditioning against forebrain ischemia in the mouse hippocampus
The protein encoded by this gene is a member of the platelet-derived growth factor/vascular endothelial growth factor (PDGF/VEGF) family, is active in angiogenesis and endothelial cell growth, and can also affect the permeability of blood vessels. This secreted protein undergoes a complex proteolytic maturation, generating multiple processed forms which bind and activate VEGFR-3 receptors. Only the fully processed form can bind and activate VEGFR-2 receptors. This protein is structurally and functionally similar to vascular endothelial growth factor D.
vascular endothelial growth factor C
, vascular endothelial growth factor c
, FLT4 ligand DHM
, vascular endothelial growth factor-related protein
, flt4 ligand
, vascular endothelial growth factor C isoform 129
, vascular endothelial growth factor C isoform 184