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Human VEGFC Kit ELISA pour Sandwich ELISA - ABIN414935
You, Bei, Cheng, Cheng: Expression of COX-2 and VEGF-C in cholangiocarcinomas at different clinical and pathological stages. dans Genetics and molecular research : GMR 2015
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Mouse (Murine) VEGFC Kit ELISA pour Sandwich ELISA - ABIN415601
Zhang, Wang, Gao, Guo, Chen, Wang, Gao, Rao, Chen: Alternatively activated RAW264.7 macrophages enhance tumor lymphangiogenesis in mouse lung adenocarcinoma. dans Journal of cellular biochemistry 2009
Show all 2 references for ABIN415601
data not only reveal a non-canonical function of Mt2 (Montrer MT2 Kits ELISA) in angiogenesis, but also propose Mt2 (Montrer MT2 Kits ELISA) as a novel regulator of vegfc expression.
Vegfc signaling increases mafba (Montrer MAFB Kits ELISA) expression to control downstream transcription
Vegfc is dispensable for facial lymphatic sprouting but not for the complete development of the facial lymphatic network.
In the embryo, phenotypes driven by increased Vegfc are suppressed in the absence of Ccbe1 (Montrer CCBE1 Kits ELISA), and Vegfc-driven sprouting is enhanced by local Ccbe1 (Montrer CCBE1 Kits ELISA) overexpression. Moreover, Vegfc- and Vegfr3 (Montrer FLT4 Kits ELISA)-dependent Erk (Montrer MAPK1 Kits ELISA) signaling is impaired in the absence of Ccbe1 (Montrer CCBE1 Kits ELISA).
Vegfc has an essential role in lymphangiogenesis [review]
The parallel growth of motoneuron axons with the dorsal aorta depends on Vegfc/Vegfr3 (Montrer FLT4 Kits ELISA) signaling in zebrafish.
Vegfc acts in two distinct modes during development: as a paracrine factor secreted from arteries to guide closely associated lymphatic vasculature and as an autocrine factor to drive migratory persistence during angiogenesis.
Rspo1-Wnt-VegfC-Vegfr3 signaling plays a crucial role as an endothelial-autonomous permissive cue for developmental angiogenesis.
Here, we show that vascular endothelial growth factor C (Vegfc), an angiogenic as well as a lymphangiogenic factor, is unexpectedly involved in this process in zebrafish.
The development of lymphatic vessels in zebrafish embyros depends on Vegfc signaling.
VEGF-C and VEGF-C156S genes have roles in the pro-lymphangiogenic growth factor therapy of lymphedema
Transcription of the vascular endothelial growth factor C gene (VEGF-C) and translation of the corresponding protein were significantly up-regulated in swine umbilical vein endothelial cells with classical swine fever virus acute infection.
No difference in bioactivity was detected between porcine relaxin-1 (Montrer RLN1 Kits ELISA) and recombinant human relaxin-2 (Montrer RLN1 Kits ELISA) in either mice or rats.
During progressive ischemia, functional and metabolic benefits of intramyocardial VEGF-C gene transfer were apparent. VEGF-C-induced collateral formation occurred at the site of gene transfer
This paper quantifies the lymphatic microvessel density (LMD) in benign and malignant salivary gland tumors and analyzes the relationship between LMD and tumor expression of vascular endothelial growth factors C (VEGF-C) and the proliferative index.
KAI1 (Montrer CD82 Kits ELISA)-induced decreases in VEGFC expression are mediated via Src (Montrer SRC Kits ELISA)/STAT3 (Montrer STAT3 Kits ELISA) signaling pathways in pancreatic cancer cells.
The results of this study suggest that the more aggressive biological behavior of squamous cell carcinoma of the tongue in young patients may be related to a higher expression of VEGF-C.
This study suggests that NRP1 (Montrer NELL1 Kits ELISA) expression and LVD are independent factors that are likely to predict the risk of LN metastasis in squamous cell carcinoma (SCC (Montrer CYP11A1 Kits ELISA))of the tongue, whereas the expression of VEGFC, VEGFR3 (Montrer FLT4 Kits ELISA), CCR7 (Montrer CCR7 Kits ELISA), and SEMA3E (Montrer SEMA3E Kits ELISA) are nonindependent predictive factors
High expression of VEGFC is associated with peritoneal dissemination in gastric cancer.
TNF-alpha (Montrer TNF Kits ELISA) mediates VEGF-C expression, which plays a critical role in the pathogenesis of pterygia.
a potential link between the upregulation of Syk (Montrer SYK Kits ELISA) and VEGF-C expression and lung adenocarcinoma.
The summarizes the structure and function features of pathway-related molecules of VEGFC/D-VEGFR3 (Montrer FLT4 Kits ELISA)/NRP2 (Montrer NELL2 Kits ELISA) axis, stages of various tumors and their molecular mechanisms and significances in tuthe expression changes of these molecules in different anatomic organs or histopathologic types or development lymphatic metastasis.
Data show that WNT1-inducible signaling pathway protein-1 (WISP)-1 (Montrer WISP1 Kits ELISA)/CCN4 (Montrer WISP1 Kits ELISA) expression was correlated with vascular endothelial growth factor-C (VEGF-C) expression in Oral squamous cell carcinoma (OSCC) specimens.
VEGF-C overexpression shows an unfavorable prognosis for EC patients.
Data show that in the MCF-7 breast cancer cell line, only MT1X metallothioneins (MTs (Montrer NEU2 Kits ELISA)) positively correlated with vascular endothelial growth factor C (VEGFC).
The findings in this study strongly suggest the following: i) that VEGF-C promotes the proliferative activity and migratory ability of mesenchymal stem cell ; and ii) VEGF-C and Tgfb (Montrer TGFB1 Kits ELISA) reciprocally regulate mesenchymal stem cell commitment to differentiation into lymphatic endothelial or osteoblastic phenotypes, respectively.
The authors show that VEGF-C is necessary for perinatal lymphangiogenesis, but required for adult lymphatic vessel maintenance only in the intestine.
MT1-MMP (Montrer MMP14 Kits ELISA) directly cleaves LYVE-1 (Montrer LYVE1 Kits ELISA) on lymphatic endothelial cells to inhibit LYVE-1 (Montrer LYVE1 Kits ELISA)-mediated lymphangiogenic responses and restrains the production of VEGF-C.
HA increases lymphangiogenesis in renal fibrosis model and also stimulates vascular endothelial cell growth factor (Montrer FGF1 Kits ELISA)-C production from macrophages through Toll-like receptor 4 (Montrer TLR4 Kits ELISA)-dependent signal pathway
Results showed that the VEGF-C/VEGFR-3 (Montrer FLT4 Kits ELISA) system underlies the protective effect of ischemic preconditioning against forebrain ischemia in the mouse hippocampus
Vascular endothelial growth factor C/VEGFR-3 (Montrer FLT4 Kits ELISA) signaling modifies HS and CCL21 (Montrer CCL21 Kits ELISA) gradients around lymphatics, regulating lymphocyte migration.
Coronary artery stem development first requires VEGF-C to stimulate vessel growth around the outflow tract.
Data show that the expression of lymphatic vessel endothelial hyaluronan receptor 1 (LYVE 1 (Montrer LYVE1 Kits ELISA)) was similar with vascular endothelial growth factor C (VEGF-C), but its peak appeared 1-2 d later than that of VEGF-C.
reveal the evolutionary conservation of the lymphatic-like phenotype of the Schlemm's canal (SC), implicate VEGF-C and VEGFR-3 (Montrer FLT4 Kits ELISA) as critical regulators of SC lymphangiogenesis
The protein encoded by this gene is a member of the platelet-derived growth factor/vascular endothelial growth factor (PDGF/VEGF) family, is active in angiogenesis and endothelial cell growth, and can also affect the permeability of blood vessels. This secreted protein undergoes a complex proteolytic maturation, generating multiple processed forms which bind and activate VEGFR-3 receptors. Only the fully processed form can bind and activate VEGFR-2 receptors. This protein is structurally and functionally similar to vascular endothelial growth factor D.
vascular endothelial growth factor C
, vascular endothelial growth factor c
, FLT4 ligand DHM
, vascular endothelial growth factor-related protein
, flt4 ligand
, vascular endothelial growth factor C isoform 129
, vascular endothelial growth factor C isoform 184