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APOB product is the main apolipoprotein of chylomicrons and low density lipoproteins. De plus, nous expédions APOB Anticorps (359) et APOB Protéines (18) et beaucoup plus de produits pour cette protéine.
Showing 10 out of 135 products:
Human APOB Kit ELISA pour Sandwich ELISA - ABIN2683473
Adiloglu, Can, Kinay, Aridogan: Infection with Chlamydia pneumoniae but not Helicobacter pylori is related to elevated apolipoprotein B levels. dans Acta cardiologica 2006
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Mouse (Murine) APOB Kit ELISA pour Sandwich ELISA - ABIN1874344
Hollie, Hui: Group 1B phospholipase A? deficiency protects against diet-induced hyperlipidemia in mice. dans Journal of lipid research 2011
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Human APOB Kit ELISA pour Sandwich ELISA - ABIN612664
Butt, Bernhardt, Smolenski, Kotsonis, Fröhlich, Sickmann, Meyer, Lohmann, Schmidt: Endothelial nitric-oxide synthase (type III) is activated and becomes calcium independent upon phosphorylation by cyclic nucleotide-dependent protein kinases. dans The Journal of biological chemistry 2000
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Human APOB Kit ELISA pour Sandwich ELISA - ABIN417290
Morikawa, Maranhão, Alves, Negrão, da Silva, Vinagre: Effects of anabolic androgenic steroids on chylomicron metabolism. dans Steroids 2012
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Mouse (Murine) APOB Kit ELISA pour Competition ELISA - ABIN832336
Hu, Yang, Ma, Chen, Hu, Zhao, Li, Qiu, Lu, Wang, Gao, Sha, Zheng, Wang: A lincRNA-DYNLRB2-2/GPR119/GLP-1R/ABCA1-dependent Signal Transduction Pathway Is Essential for the Regulation of Cholesterol Homeostasis and Inflammatory Reactions. dans Journal of lipid research 2014
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Human APOB Kit ELISA pour Sandwich ELISA - ABIN415222
Pramfalk, Larsson, Härdfeldt, Eriksson, Parini: Culturing of HepG2 cells with human serum improve their functionality and suitability in studies of lipid metabolism. dans Biochimica et biophysica acta 2015
Mouse (Murine) APOB Kit ELISA pour Sandwich ELISA - ABIN1113522
Smallwood, Gatti, Quizon, Weinstock, Jung, Zhao, Hua, Pomp, Bennett: High-resolution genetic mapping in the diversity outbred mouse population identifies Apobec1 as a candidate gene for atherosclerosis. dans G3 (Bethesda, Md.) 2014
Mouse (Murine) APOB Kit ELISA pour Sandwich ELISA - ABIN832333
Hu, Hu, Zhao, Li, Ma, Wu, Lu, Qiu, Sha, Wang, Gao, Zheng, Wang: An agomir of miR-144-3p accelerates plaque formation through impairing reverse cholesterol transport and promoting pro-inflammatory cytokine production. dans PLoS ONE 2014
that apos involved in TG metabolism such as apoC2 (Montrer APOC2 Kits ELISA), C3, E, and A4 (micromolar concentration), and apoB48 and apoA5 (Montrer APOA5 Kits ELISA) (single-digit nanomolar concentration) can be quantified from a single digestion mixture.
These findings indicate that maternal apo (Montrer C9orf3 Kits ELISA) B levels are significantly associated with apo (Montrer C9orf3 Kits ELISA) B levels in their pre-school age children, adjusted for confounding variables. Furthermore, the mother-child correlations in apo (Montrer C9orf3 Kits ELISA) B levels were independent of mother-child adiposity. Measurement of apo (Montrer C9orf3 Kits ELISA) B levels in mothers may identify both high-risk children and mothers who may benefit from intervention.
Individuals with apoB higher than predicted by non-HDL (Montrer HSD11B1 Kits ELISA)-C had significantly higher levels of PAI-1 (Montrer SERPINE1 Kits ELISA), which may contribute to the increased risk of future atherothrombotic events
analysis of five likely pathogenic heterozygous rare variants that include four novel nonsense mutations in APOB (p.Gln845*, p.Gln2571*, p.Cys2933* and p.Ser3718*) and a rare variant in PCSK9 (Montrer PCSK9 Kits ELISA) (Minor Allele Frequency <0.1%).
The impact of smoking on the levels of apolipoprotein B (APOB) was evaluated by analyzing data from NHANES for the years 2007-2012 for US adolescents aged 12-19 years and adults aged greater than or eqult to 20 years. The study found that smoking did not influence the observed levels of APOB for either adolescents or adults.
Identify LDLR (Montrer LDLR Kits ELISA), APOB and PCSK9 (Montrer PCSK9 Kits ELISA) novel mutations causing familial hypercholesterolemia in the central south region of China.
Rare variants of APOB or PCSK9 (Montrer PCSK9 Kits ELISA) were identified in nine of the 22 study patients with extremely low LDL-C levels
Eicosapentaenoic acid has direct antioxidant benefits in various apoB-containing subfractions.
Association of plasma apoB with IR in obese subjects is dependent on gynoid WAT dysfunction
Serum ApoB was not significantly different between term SGA newborns and control term newborns.
enhanced VLDL TG secretion in the absence of hepatocyte ABCA1 (Montrer ABCA1 Kits ELISA) is due to altered intracellular trafficking of apolipoprotein B (apoB), resulting in augmented TG addition to nascent VLDL.
We carried out our experiment in mice deficient in the low density lipoprotein (LDL) receptor (Montrer LDLR Kits ELISA) and expressing only ApoB100 molecule (ApoB - LDLr (Montrer LDLR Kits ELISA)) where the development of atherosclerosis is known to closely mimic human atherosclerosis
The effect of hypercholesterolemia induced immune response and inflammation on progression of atherosclerosis in ApoB(tm25gy) LDLr (Montrer LDLR Kits ELISA)(tm1Her) mice, expressing only ApoB100 and deficient in the low density lipoprotein receptor (Montrer LDLR Kits ELISA).
ApoB-containing lipoproteins contribute to augmentation of AngII-induced abdominal aortic aneurysms in male mice.
Immunization with human apolipoprotein B100 (ApoB) resulted in four-fold increased accumulation of effector T cells in ApoB-containing matrigel
PCSK9 (Montrer PCSK9 Kits ELISA) markedly increases intestinal triglyceride-rich apoB production through mechanisms mediated in part by transcriptional effects on apoB.
Mice that produce apoB100 in the RPE and liver secrete lipoproteins into Bruch's membrane, but not to the extent that distinct features of AMD (Montrer AMD1 Kits ELISA) develop
The aim of this study was to characterize the ocular morphology of low-density lipoprotein receptor-deficient apolipoprotein B-100-only mice, with IGF-II overexpression.
Our data establish the role of APOB gene in severe gut (Montrer GUSB Kits ELISA) dysmotility.
Cardiac lipotoxicity may originate from direct inhibition of myocardial ApoB lipoprotein and subsequent decreased lipid export, caused by supraphysiological levels of catecholamines.
The clinicopathological phenotype of affected Holstein cattle homozygous for the causative apolipoprotein B gene (APOB) mutation associated with cholesterol deficiency is described.
Beyond malabsorption of dietary lipids, deleterious effects of apolipoprotein B deficiency on hepatic lipid metabolism, steroid biosynthesis, and cell membrane function can be expected, which may result in unspecific symptoms of reduced fertility, growth, and health.
Cholesterol deficiency results from a 1.3kbp insertion of an endogenous retrovirus (ERV2-1-LTR_BT) into exon 5 of the APOB gene at BTA11:77,959kb. The insertion is flanked by 6bp target site duplications as described for insertions mediated by retroviral integrases.
A transposable element insertion in APOB causes cholesterol deficiency in Holstein cattle
Nonesterified fatty acids significantly inhibit the expression of ApoB100, ApoE (Montrer APOE Kits ELISA), MTP (Montrer MTTP Kits ELISA), and LDLR (Montrer LDLR Kits ELISA), thereby decreasing the synthesis and assembly of VLDL and inducing TG accumulation in bovine hepatocytes.
after calving the apolipoprotein B(100) mRNA synthesis was lower, whereas microsomal triglyceride transfer protein (MTP (Montrer MTTP Kits ELISA)) and apolipoprotein E (Montrer APOE Kits ELISA) messenger RNA abundance were higher in the liver.
found association between genotypes for LDLR (Montrer LDLR Kits ELISA) and APOB polymorphisms and serum lipid levels, but none of them seem to be the causal mutation but probably represent closely linked polymorphisms
This gene product is the main apolipoprotein of chylomicrons and low density lipoproteins. It occurs in plasma as two main isoforms, apoB-48 and apoB-100: the former is synthesized exclusively in the gut and the latter in the liver. The intestinal and the hepatic forms of apoB are encoded by a single gene from a single, very long mRNA. The two isoforms share a common N-terminal sequence. The shorter apoB-48 protein is produced after RNA editing of the apoB-100 transcript at residue 2180 (CAA->UAA), resulting in the creation of a stop codon, and early translation termination. Mutations in this gene or its regulatory region cause hypobetalipoproteinemia, normotriglyceridemic hypobetalipoproteinemia, and hypercholesterolemia due to ligand-defective apoB, diseases affecting plasma cholesterol and apoB levels.
, apolipoprotein B (including Ag(x) antigen)
, apolipoprotein B-100
, apolipoprotein B48
, mutant Apo B 100
, apolipoprotein B PI
, apolipoprotein B-48
, apolipoprotein B