Bcl-2 anticorps (Biotin)

N° du produit ABIN2590010

Cet anticorps Lapin Polyclonal détecte spécifiquement Bcl-2 dans WB et ELISA. Il présente une réactivité avec des échantillons de Humain.

Aperçu rapide pour Bcl-2 anticorps (Biotin) (ABIN2590010)

Antigène: Bcl-2 (BCL2) (B-Cell CLL/lymphoma 2 (BCL2))

Reactivité: Humain

Hôte: Lapin

Clonalité: Polyclonal

Conjugué: Cet anticorp Bcl-2 est conjugé à/à la Biotin

Application: Western Blotting (WB), ELISA

Détail du produit anti-Bcl-2 anticorps (Biotin)

Specificité: Specifically detects C-terminal Bcl-2 from humans. There is no cross-reactivity with Caspase-3, Caspase-8 or Livin.

Purification: Protein G purified

Immunogène: synthetic peptide corresponding to the C-terminal region of Bcl-2 conjugated with KLH

Type of Immunogen: Synthetic peptide - KLH conjugated

Isotype: IgG

Information d'application

Indications d'application: Approved: ELISA (1:64000), WB (1:5000)

Usage: The applications listed have been tested for the unconjugated form of this product. Other forms have not been tested.

Commentaires:

Target Species of Antibody: Human

Restrictions: For Research Use only

Stockage

Format: Liquid

Concentration: Lot specific

Buffer: PBS without preservatives

Agent conservateur: Without preservative

Conseil sur la manipulation: Avoid repeated freezing and thawing

Stock: 4 °C,-20 °C

Stockage commentaire: May be stored at 4°C for short-term only. For long-term storage and to avoid freeze-thaw cycles, aliquot and store at -20°C. Aliquots are stable for at least 6 months at -20°C.

Détails sur Bcl-2

Antigène: Bcl-2 (BCL2) (B-Cell CLL/lymphoma 2 (BCL2))

Autre désignation: BCL2 / Bcl-2

Sujet: Name/Gene ID: BCL2
Family: Apoptosis

Synonyms: BCL2, Apoptosis regulator Bcl-2, B-cell CLL/lymphoma 2, B-cell lymphoma protein 2, Bcl-2, PPP1R50

ID gène: 596

UniProt: P10415

Pathways: Signalisation MAPK, Signalisation PI3K-Akt, Apoptose, Caspase Cascade in Apoptosis, Regulation of Muscle Cell Differentiation, Cell-Cell Junction Organization, Skeletal Muscle Fiber Development, Autophagy, Smooth Muscle Cell Migration, Negative Regulation of intrinsic apoptotic Signaling