IL-8 anticorps

N° du produit ABIN966396

Cité dans 2 publications. Cet anticorps anti-IL-8 Monoclonal (ABIN966396) détecte spécifiquement IL-8 dans WB et ELISA. L’anticorps est réactif avec des échantillons de Humain.

Aperçu rapide pour IL-8 anticorps (ABIN966396)

Antigène: IL-8 (IL8) (Interleukin 8 (IL8))

Reactivité: Humain

Hôte: Veuillez nous consulter SVP

Clonalité: Monoclonal

Conjugué: Cet anticorp IL-8 est non-conjugé

Application: Western Blotting (WB), ELISA

Détail du produit anti-IL-8 anticorps

Specificité: Ni-NTA purified recombinant human IL-8 expressed in E.Coli. strain BL21 (DE3).

Purification: Antibodies are purified by protein A affinity chromatography

Isotype: IgG1 kappa

Information d'application

Indications d'application: Dilution: Western Blot: 1:500-1,000
ELISA: 1,000-2,000.
Determining optimal working dilutions by titration test.

Restrictions: For Research Use only

Stockage

Stock: -20 °C

Références pour anticorps anti-IL-8 (ABIN966396)

Baggiolini, Clark-Lewis: "Interleukin-8, a chemotactic and inflammatory cytokine." dans: FEBS letters, Vol. 307, Issue 1, pp. 97-101, (1992) (PubMed).

Mukaida, Shiroo, Matsushima: "Genomic structure of the human monocyte-derived neutrophil chemotactic factor IL-8." dans: Journal of immunology (Baltimore, Md. : 1950), Vol. 143, Issue 4, pp. 1366-71, (1989) (PubMed).

Détails sur IL-8

Antigène: IL-8 (IL8) (Interleukin 8 (IL8))

Autre désignation: IL-8

Sujet: Interleukin-8 (IL-8)is a member of the CXC chemokine family. This chemokine is one of the major mediators of the inflammatory response. This chemokine is secreted by several cell types. It functions as a chemoattractant, and is also a potent angiogenic factor. IL-8 is believed to play a role in the pathogenesis of bronchiolitis, a common respiratory tract disease caused by viral infection. IL-8 and other ten members of the CXC chemokine gene family form a chemokine gene cluster in a region mapped to chromosome 4q.

ID gène: 3576

Pathways: Signalisation TLR, Cellular Response to Molecule of Bacterial Origin, Regulation of G-Protein Coupled Receptor Protein Signaling, ER-Nucleus Signaling, Hepatitis C, Autophagy