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anti-Mouse (Murine) EPH Receptor B4 Anticorps:
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Mouse (Murine) Polyclonal EPH Receptor B4 Primary Antibody pour CyTOF, FACS - ABIN4899282
Foster, Gordon, Cardenas, Veiga-Fernandes, Makinen, Grigorieva, Wilkinson, Blackburn, Richie, Manley, Adams, Kioussis, Coles: EphB-ephrin-B2 interactions are required for thymus migration during organogenesis. dans Proceedings of the National Academy of Sciences of the United States of America 2010
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Human Polyclonal EPH Receptor B4 Primary Antibody pour CyTOF, FACS - ABIN4899281
Boyer-Di Ponio, El-Ayoubi, Glacial, Ganeshamoorthy, Driancourt, Godet, Perrière, Guillevic, Couraud, Uzan: Instruction of circulating endothelial progenitors in vitro towards specialized blood-brain barrier and arterial phenotypes. dans PLoS ONE 2014
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Human Monoclonal EPH Receptor B4 Primary Antibody pour ELISA, WB - ABIN1724713
Jiménez-Vega, Yepiz-Plascencia, Söderhäll, Vargas-Albores: A single WAP domain-containing protein from Litopenaeus vannamei hemocytes. dans Biochemical and biophysical research communications 2004
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Human Monoclonal EPH Receptor B4 Primary Antibody pour IHC, ELISA - ABIN969107
Chrencik, Brooun, Recht, Kraus, Koolpe, Kolatkar, Bruce, Martiny-Baron, Widmer, Pasquale, Kuhn: Structure and thermodynamic characterization of the EphB4/Ephrin-B2 antagonist peptide complex reveals the determinants for receptor specificity. dans Structure (London, England : 1993) 2006
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Human Monoclonal EPH Receptor B4 Primary Antibody pour IF, ELISA - ABIN966075
Wu, Suo, Risberg, Karlsson, Villman, Nesland: Expression of Ephb2 and Ephb4 in breast carcinoma. dans Pathology oncology research : POR 2004
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Human Monoclonal EPH Receptor B4 Primary Antibody pour IHC, ELISA - ABIN966076
Kubota: [What is drug-epidemiology?]. dans Nihon Naika Gakkai zasshi. The Journal of the Japanese Society of Internal Medicine 2007
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Human Polyclonal EPH Receptor B4 Primary Antibody pour IHC (p), WB - ABIN391924
Steinle, Meininger, Forough, Wu, Wu, Granger: Eph B4 receptor signaling mediates endothelial cell migration and proliferation via the phosphatidylinositol 3-kinase pathway. dans The Journal of biological chemistry 2002
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Mouse (Murine) Monoclonal EPH Receptor B4 Primary Antibody pour IA, IHC (fro) - ABIN2191850
Huang, Yamada, Kidoya, Naito, Nagahama, Kong, Katoh, Li, Ueno, Takakura: EphB4 overexpression in B16 melanoma cells affects arterial-venous patterning in tumor angiogenesis. dans Cancer research 2007
Human Monoclonal EPH Receptor B4 Primary Antibody pour ICC, ELISA - ABIN969108
Tachibana, Tonomoto, Hyakudomi, Hyakudomi, Hattori, Ueda, Kinugasa, Yoshimura: Expression and prognostic significance of EFNB2 and EphB4 genes in patients with oesophageal squamous cell carcinoma. dans Digestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver 2007
Human Polyclonal EPH Receptor B4 Primary Antibody pour ELISA - ABIN261662
Kumar, Masood, Spannuth, Singh, Scehnet, Kleiber, Jennings, Deavers, Krasnoperov, Dubeau, Weaver, Sood, Gill: The receptor tyrosine kinase EphB4 is overexpressed in ovarian cancer, provides survival signals and predicts poor outcome. dans British journal of cancer 2007
These findings implicate a molecular disinhibitory mechanism driving the establishment of perisomatic inhibition whereby visual experience enhances Pten signalling, resulting in the suppression of EphB4 expression; this relieves a native synaptic repulsion between PV cells and pyramidal neurons, thereby promoting the assembly of perisomatic inhibition.
As endothelial progenitor cells (EPCs)derived from mouse bone marrow were cultured on substrates of increasing stiffness, the mRNA and protein levels of the specific arterial endothelial cell marker ephrinB2 (Montrer EFNB2 Anticorps) were found to increase, while the expression of the venous marker EphB4 decreased
EPHB4 is a critical regulator of early lymphatic vascular development; mutations in the gene can cause an autosomal dominant form of LRHF that is associated with a high mortality rate
These results identify EPHB4/ephrin B2 (Montrer EFNB2 Anticorps) signaling as critical to Hematopoietic stem and progenitor cells mobilization from bone marrow
EphB4 plays an irreplaceable role in bone regeneration in an inflammatory microenvironment, whereas the functional loss of ephrinB2 (Montrer EFNB2 Anticorps) can be effectively compensated, most possibly by other ephrins with similar chemical structures
Eph (Montrer EPHA1 Anticorps)-B4 stimulates eNOS (Montrer NOS3 Anticorps) phosphorylation in vitro and may mediate vein graft adaptation by regulation of eNOS (Montrer NOS3 Anticorps) activity in vivo.
These data indicate that ephrinB2 (Montrer EFNB2 Anticorps)/EphB4 signaling within the osteoblast lineage is required for late stages of osteoblast differentiation.
EphB4 promotes endochondral ossification while inhibiting osteoclast development during callus formation.
Selective inhibition of EphB4 using a functional blocking antibody results in defective lymphatic valve development.
Overexpression of bone-specific Ephb4 clearly demonstrated prevention of the development and/or progression of fibrosis in OA synovial membrane
functional expression of EphB4 is considered a promising differentiating characteristic, preferentially determined by non-invasive in vivo imaging, which may improve personalized theranostics of malignant melanoma.
The identified endothelial signalling pathway of CCM3 (Montrer PDCD10 Anticorps)-DLL4 (Montrer DLL4 Anticorps)/Notch (Montrer NOTCH1 Anticorps)-EphB4-Erk1/2 may provide an insight into mechanism of CCM3 (Montrer PDCD10 Anticorps)-ablation-mediated angiogenesis.
EphB4 overexpression is associated with resistance to dasatinib in chronic myeloid leukemia (Montrer BCL11A Anticorps).
this study uncovered the character of EPHB4-regulating endothelial activation in the pathogenesis of preeclampsia
HOXA9 (Montrer HOXA9 Anticorps) transcriptionally regulated EPHB4 expression to modulate trophoblasts migration and invasion, which may suggest a contribution of HOXA9 (Montrer HOXA9 Anticorps)-EPHB4 in the poor placentation in the pathogenesis of preeclampsia.
Inhibition of EphB4 forward signaling using soluble EphB4 protein fused (Montrer AXIN1 Anticorps) with murine serum albumin (Montrer ALB Anticorps) failed to affect eRMS model tumor progression, but did moderately slow progression in murine aRMS
EPHB4 mutations were identified in patients with multifocal capillary malformation with arteriovenous malformations.
EPHB4 is a critical regulator of early lymphatic vascular development; mutations in the gene can cause an autosomal dominant form of lymphatic-related hydrops fetalis that is associated with a high mortality rate
The expression of EPHB4 was increased in gastric cancer and increased EPHB4 expression was correlated with poor survival. Knockdown of EPHB4 promoted adhesion and exerted diverse effects on migration of gastric cancer cells.
EphB4 protein acts as a tumour promoter associated with proliferation, invasion, and angiogenesis, and may be used as a potential colorectal cancer (CRC (Montrer CALR Anticorps)) therapeutic target.
Ephrin receptors and their ligands, the ephrins, mediate numerous developmental processes, particularly in the nervous system. Based on their structures and sequence relationships, ephrins are divided into the ephrin-A (EFNA) class, which are anchored to the membrane by a glycosylphosphatidylinositol linkage, and the ephrin-B (EFNB) class, which are transmembrane proteins. The Eph family of receptors are divided into 2 groups based on the similarity of their extracellular domain sequences and their affinities for binding ephrin-A and ephrin-B ligands. Ephrin receptors make up the largest subgroup of the receptor tyrosine kinase (RTK) family. The protein encoded by this gene binds to ephrin-B2 and plays an essential role in vascular development.
EPH receptor B4
, eph receptor tyrosine kinase
, ephrin receptor EphB4
, ephrin type-B receptor 4-like
, developmental kinase 2
, ephrin type-B receptor 4
, hepatoma transmembrane kinase
, tyrosine kinase MYK-1
, soluble EPHB4 variant 1
, soluble EPHB4 variant 2
, soluble EPHB4 variant 3
, tyrosine-protein kinase TYRO11
, tyrosine-protein kinase receptor HTK