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These data identify suppression of CXCL2 (Montrer CXCL2 Kits ELISA) and CXCL5 (Montrer CXCL5 Kits ELISA) chemoattractant expression by 11beta-HSD1 (Montrer HSD11B1 Kits ELISA) as a novel mechanism with potential for regulation of neutrophil recruitment to the injured myocardium, and cardiac fibroblasts as a key site for intracellular glucocorticoid regeneration during acute inflammation following myocardial injury.
IL-17RA (Montrer IL17RA Kits ELISA) regulates CXL-1 and 5 production in the lungs during the adaptive response.
STAT3 (Montrer STAT3 Kits ELISA) is required for maximal OSM (Montrer OSM Kits ELISA)-induced CXCL5 (Montrer CXCL5 Kits ELISA) expression.
CXCL5 (Montrer CXCL5 Kits ELISA) has a role in neutrophil recruitment in TH17-mediated glomerulonephritis
Since adaptive villus growth occurs despite impaired CXCL5 (Montrer CXCL5 Kits ELISA) expression and enhanced angiogenesis, this suggests that the growth of new blood vessels is not needed for resection-induced mucosal surface area expansion following massive SBR (Montrer NXF1 Kits ELISA).
CXCL5 (Montrer CXCL5 Kits ELISA) regulates pulmonary responses to infection and plays a central role in conferring clock control of inflammation.
findings demonstrated that CXCL1 (Montrer CXCL1 Kits ELISA) and CXCL5 (Montrer CXCL5 Kits ELISA) are increased in circulation with onset of T2D, are produced by islets under stress, and synergistically affect islet function, suggesting that these chemokines participate in pathogenesis of T2D.
TLR2 (Montrer TLR2 Kits ELISA)-induced epithelial-derived CXCL5 (Montrer CXCL5 Kits ELISA) is critical for polymorphonuclear leukocyte-driven destructive inflammation in pulmonary tuberculosis.
Our data suggest that the differential regulation of the chemokine CXCL5 between osteoblasts and endothelial cells upon FGF2 treatment is involved in Hematopoietic stem cell mobilization from the osteoblast niche or bone marrow to peripheral blood.
CXCL5 (Montrer CXCL5 Kits ELISA) modulated macrophage activation, increased expression of the cholesterol efflux regulatory protein ABCA1 (Montrer ABCA1 Kits ELISA), and enhanced cholesterol efflux activity in macrophages.
CXCL6 level is high in the serum of chronic hepatitis B patients.CXCL6 promotes human hepatocyte proliferation through the CXCR1-NFkappaB pathway and inhibits collagen I secretion by hepatic stellate cells.
The role of PITX2 in glaucoma may be mediated partly by regulating the expression of CXCL6 and BBS5 and thus affecting immune functions and intraocular pressure.
This is the first study to note that elevated systemic CCL5 (Montrer CCL5 Kits ELISA) and CXCL6 were associated with moderate/severe lumbar disc degeneration. These chemokines may be systemic biomarkers for the diagnosis and monitoring of disc degeneration.
The neutrophil-recruiting chemokine (Montrer CCL1 Kits ELISA) GCP-2/CXCL6 is expressed in cystic fibrosis (Montrer S100A8 Kits ELISA) airways and retains its functional properties after binding to extracellular DNA.
HIF-1alpha (Montrer HIF1A Kits ELISA) promotes HCC (Montrer FAM126A Kits ELISA) progression and metastasis by upregulating CXCL6 transcription in HCC (Montrer FAM126A Kits ELISA) cells.
Expression of the CXCL8 (Montrer IL8 Kits ELISA), CXCL6 and CXCL1 (Montrer CXCL1 Kits ELISA) genes are under the primary control of 1,25-dihydroxyvitamin D3 and its receptor.
Data suggest that expression of CXCL6 in trophoblasts is up-regulated during pregnancy development/placentation; CXCL6 expression inhibits trophoblast cell migration and invasion by down-regulating activity of MMP2 (matrix metalloproteinase 2 (Montrer MMP2 Kits ELISA)).
Overexpression of GCP-2 (Montrer FOLH1 Kits ELISA) in mesenchymal stem cells has the potential to enhance their angiogenic and survival properties.
Studies show that functional blocking of GCP-2 (Montrer FOLH1 Kits ELISA) inhibits tumor growth and metastases.
The aim of this study was to investigate the role of the transcription factors, AP-1 (Montrer FOSB Kits ELISA) and NF-kappaB (Montrer NFKB1 Kits ELISA), in IL-6 (Montrer IL6 Kits ELISA) and CXCL8 (Montrer IL8 Kits ELISA) regulation in Jurkat T-cells.
mouse homolog is a member of the CXC chemokine family, is a neutrophil chemoattractant and is rapidly induced in response to muscle injury
C-X-C motif chemokine 5
, CXC chemokine LIX
, chemokine (C-X-C motif) ligand 6 (granulocyte chemotactic protein 2)
, cytokine LIX
, small-inducible cytokine B5
, granulocyte chemotactic protein-2
, small inducible cytokine B subfamily, member 5
, small inducible cytokine subfamily B, member 15
, C-X-C motif chemokine 6
, Small inducible cytokine subfamily B (Cys-X-Cys), member b
, chemokine alpha 3
, granulocyte chemotactic protein 2
, small inducible cytokine subfamily B (Cys-X-Cys), member 6 (granulocyte chemotactic protein 2)
, small-inducible cytokine B6
, chemokine (C-X-C motif) ligand 5
, inducible cytokine subfamily B (Cys-X-Cys), member 6