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anti-Mouse (Murine) GNAI1 Anticorps:
anti-Human GNAI1 Anticorps:
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Human Polyclonal GNAI1 Primary Antibody pour ELISA, WB - ABIN561067
Sasaki, Yamasaki, Omotuyi, Mishina, Ueda: Age-dependent dystonia in striatal G?7 deficient mice is reversed by the dopamine D2 receptor agonist pramipexole. dans Journal of neurochemistry 2013
Human Polyclonal GNAI1 Primary Antibody pour IHC, IHC (p) - ABIN441599
Matsumura, Kojidani, Kamioka, Uchida, Haraguchi, Kimura, Toyoshima: Interphase adhesion geometry is transmitted to an internal regulator for spindle orientation via caveolin-1. dans Nature communications 2016
Human Polyclonal GNAI1 Primary Antibody pour WB - ABIN4890045
Hurst, Henkel, Brown, Hooks: Endogenous RGS proteins attenuate Galpha(i)-mediated lysophosphatidic acid signaling pathways in ovarian cancer cells. dans Cellular signalling 2008
Gnai1 function is impaired in the spinal cord of Ews/Ewsr1 (Montrer EWSR1 Anticorps) KO mice
LGN (Montrer GPSM2 Anticorps) and Galphai participate in a long-inferred signal that originates outside the bundle to model its staircase-like architecture, a property that is essential for direction sensitivity to mechanical deflection and hearing.
stimulation of GPR17 (Montrer GPR17 Anticorps) by the small molecule agonist MDL29,951 (2-carboxy-4,6-dichloro-1H-indole-3-propionic acid) decreases myelin basic protein (Montrer MBP Anticorps) expression levels mainly by triggering the Galphai/o signaling pathway.
Gnai1 missense mutation is responsible of hyperpigmentation in mouse model.
acidosis in inflamed tissues may be a decisive factor to regulate switching of PKA and PKCepsilon dependence via proton-sensing G-protein-coupled receptors.
Data show that guanine nucleotide-binding protein G(i) subunit alpha-1 and alpha-3 (Galphai1/3) can interact with CD14 antigen/Grb2-associated binding protein Gab1, which modulates macrophage polarization in vitro and in vivo.
By using mice deficient in individual Galphai/o G-protein subunits, authors demonstrate that Galphai1 and Galphai3 are the critical in vivo targets of ADP-ribosylation underlying vasoactive amine sensitization elicited by pertussis toxin exposure.
leucine can directly facilitate insulin (Montrer INS Anticorps) signaling through a Galphai protein-dependent intracellular signaling pathway
Inactive Galpha(i1)-GDP enhances the affinity of RGS14 for H-Ras-GTP in live cells, resulting in a ternary signaling complex that is further regulated by G protein-coupled receptors.
Mice with mutations of Gnai1 or Gnai2 (Montrer GNAI2 Anticorps) have neither fusions of ribs nor lumbar vertebrae, but loss of both Gnai3 (Montrer GNAI3 Anticorps) and one of the other two genes increases the number and severity of rib fusions without affecting the lumbar fusions.
These data indicate that, unlike in taste cells, TAS2Rs couple to the prevalent G proteins, Galphai1, Galphai2 (Montrer GNAI2 Anticorps), and Galphai3 (Montrer GNAI3 Anticorps), with no evidence for functional coupling to Galphagust.
testosterone rapidly increased whole-cell HCAEC SKCa and BKCa (Montrer KCNMA1 Anticorps) currents via a surface androgen receptor (Montrer AR Anticorps), Gi/o protein, and protein kinase A
These findings suggest that Gi1 interacts only with active GPCRs and that the well known high speed of GPCR signal transduction does not require preassembly between G proteins and GPCRs.
CGRP (Montrer S100A12 Anticorps) family of receptors displays both ligand- and RAMP-dependent signaling bias among the Galphas (Montrer GNAS Anticorps), Galphai, and Galphaq (Montrer GNAQ Anticorps)/11 pathways.
biochemical and computational data indicate that the interactions between alpha5, alpha1, and beta2-beta3 are not only vital for GDP release during G protein activation, but they are also necessary for proper GTP binding (Montrer RND2 Anticorps) (or GDP rebinding).
Data indicate that hydroxyurea (HU) induces SAR1 (Montrer IQGAP1 Anticorps) protein expression, which in turn activates gamma-globin (Montrer HBG1 Anticorps) expression, predominantly through the Gialpha/JNK (Montrer MAPK8 Anticorps)/Jun (Montrer JUN Anticorps) pathway.
Gi alpha subunit (Montrer POLG Anticorps) was found to be a key modulator of GABAB-receptors signaling in analgesia.
AC5, by binding active Galphai1, interferes with G-protein deactivation and reassembly and thereby might sensitize its own regulation.
Data indicate that focal adhesion kinase (FAK) activation and cell migration require Src, Gi/Go, COX-2 and LOXs activities.
resistin (Montrer RETN Anticorps) contributes to the pro-inflammatory state of SMC (Montrer DYM Anticorps) by the up-regulation of CX3CL1 (Montrer CX3CL1 Anticorps) and CX3CR1 (Montrer CX3CR1 Anticorps) expression via a mechanism involving NF-kB, AP-1 (Montrer FOSB Anticorps), and STAT1 (Montrer STAT1 Anticorps)/3 transcription factors, (2) resistin (Montrer RETN Anticorps) employs TLR4 (Montrer TLR4 Anticorps) and Gi-protein signaling.
Guanine nucleotide binding proteins are heterotrimeric signal-transducing molecules consisting of alpha, beta, and gamma subunits. The alpha subunit binds guanine nucleotide, can hydrolyze GTP, and can interact with other proteins. The protein encoded by this gene represents the alpha subunit of an inhibitory complex. The encoded protein is part of a complex that responds to beta-adrenergic signals by inhibiting adenylate cyclase. Two transcript variants encoding different isoforms have been found for this gene.
adenylate cyclase-inhibiting G alpha protein
, guanine nucleotide binding protein, alpha inhibiting 1
, guanine nucleotide-binding protein G(i) subunit alpha-1
, Gi1 protein alpha-subunit
, alpha-subunit of G-protein, type G-alpha-i-1
, guanine nucleotide-binding protein G(i), alpha-1 subunit
, Gi1 protein alpha subunit
, Gi-alpha-1 protein
, guanine nucleotide binding protein (G protein), alpha inhibiting 1
, Galpha i1a