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Rat (Rattus) MAPK3 Kit ELISA pour Sandwich ELISA - ABIN432074
Du, Wang, Wang: Role of RhoA/MERK1/ERK1/2/iNOS signaling in ocular ischemic syndrome. dans Graefe's archive for clinical and experimental ophthalmology 2016
Data report that MPK3/MPK6 and their substrate ERF6 promote the biosynthesis of IGSs and the conversion of I3G to 4MI3G, a target of PEN2/PEN3-dependent chemical defenses in plant immunity.
Data show that the protein kinases MPK3 and MPK6 (Montrer MAPK6 Kits ELISA) can both interact with SPOROCYTELESS/NOZZLE (SPL (Montrer SGPL1 Kits ELISA)) in vitro and in vivo and can phosphorylate the SPL (Montrer SGPL1 Kits ELISA) protein in vitro.
MKK4 (Montrer MAP2K4 Kits ELISA), MKK5 (Montrer MAP2K5 Kits ELISA), MKK7 (Montrer MAP2K7 Kits ELISA), and MKK9, are responsible for the activation of MPK3 and MPK6 (Montrer MAPK6 Kits ELISA) by melatonin, indicating that melatonin-mediated innate immunity is triggered by MAPK (Montrer MAPK1 Kits ELISA) signaling through MKK4 (Montrer MAP2K4 Kits ELISA)/5/7/9-MPK3/6 cascades.
Phosphatase AP2C1, as well as AP2C1-targeted MPK3 and MPK6 (Montrer MAPK6 Kits ELISA), are important regulators of plant-nematode interaction, where the co-ordinated action of these signalling components ensures the timely activation of plant defence.
Results demonstrated the contribution of MPK3 and MPK6 (Montrer MAPK6 Kits ELISA) to riboflavin-induced resistance.
These results indicate that the MVB pathway is positively regulated by pathogen-responsive MPK3/6 through LIP5 phosphorylation and plays a critical role in plant immune system
MKK3-MPK6 is activated by blue light in a MYC2-dependent manner.
MPK3 and MPK6 (Montrer MAPK6 Kits ELISA) target a subclass of 'VQ-motif'-containing proteins to regulate immune responses.
MicroRNA biogenesis factor DRB1 (Montrer EPHX1 Kits ELISA) is a phosphorylation target of mitogen activated protein kinase (Montrer MAPK1 Kits ELISA) MPK3 in both rice and Arabidopsis
MPK3 represses the constitutive and flg22-induced expression of defence genes in Arabidopsis thaliana.
MAPK3/1 is involved in luteinizing hormone-mediated decrease of C-type natriuretic peptide (Montrer NPPC Kits ELISA) and this process is related to the EGFR (Montrer EGFR Kits ELISA) and MAPK3/1 signal pathways
Chronic hypoxia induces Egr-1 (Montrer EGR1 Kits ELISA) via activation of ERK1/2 (Montrer MAPK1/3 Kits ELISA) and contributes to pulmonary vascular remodeling.
ER Ca(2 (Montrer CA2 Kits ELISA)+) release enhances eNOS (Montrer NOS3 Kits ELISA) Ser (Montrer SIGLEC1 Kits ELISA)-635 phosphorylation and function via ERK1/2 (Montrer MAPK1/3 Kits ELISA) activation.
Thrombospondin 1 (Montrer THBS1 Kits ELISA), fibronectin (Montrer FN1 Kits ELISA), and vitronectin (Montrer VTN Kits ELISA) are differentially dependent upon RAS, ERK1/2 (Montrer MAPK1/3 Kits ELISA), and p38 (Montrer MAPK14 Kits ELISA) for induction of vascular smooth muscle cell chemotaxis.
results suggest that Nav1.7-Ca2+ influx-protein kinase C-alpha pathway activated ERK1/ERK2 and p38, which increased phosphorylation of glycogen synthase kinase-3beta, decreasing tau phosphorylation
These data suggest that Gab1-ERK1/2 (Montrer MAPK1/3 Kits ELISA) binding and their nuclear translocation play a crucial role in Egr-1 (Montrer EGR1 Kits ELISA) nuclear accumulation.
data demonstrate that hypoxia-induced adventitial fibroblast proliferation requires activation and interaction of PI3K, Akt (Montrer AKT1 Kits ELISA), mTOR (Montrer FRAP1 Kits ELISA), p70S6K (Montrer RPS6KB1 Kits ELISA), and ERK1/2 (Montrer MAPK1/3 Kits ELISA).
This study demonstrates for the first time that cyclic mechanical stretch induces the proliferation of bovine satellite cells and suppresses their myogenic differentiation through the activation of ERK (Montrer MAPK1 Kits ELISA).
findings indicate that exposure to DHEA, at concentrations found in human blood, causes vascular endothelial proliferation by a plasma membrane-initiated activity that is Gi/o and ERK1/2 (Montrer MAPK1/3 Kits ELISA) dependent.
Results suggest that estrogen receptors and the ERK1/2 (Montrer MAPK1/3 Kits ELISA) signaling pathway are involved in the anti-apoptotic action of LY117018 in vascular endothelial cells.
The results suggest that the MPK-1 (Montrer MAPK1 Kits ELISA)/ERK (Montrer MAPK1 Kits ELISA) regulatory network, including FBF-1 (Montrer FBF1 Kits ELISA), FBF-2, and LIP-1 (Montrer CENPJ Kits ELISA), controls the number of sperm by regulating the timing of the sperm-oocyte switch in C. elegans.
Cek2 (Montrer FGFR3 Kits ELISA) has a cryptic role in cell-wall biogenesis and its role is not entirely redundant to Cek1.
knockdown of SUV420H1 (Montrer SUV420H1 Kits ELISA) reduced phosphorylated ERK1 and total ERK1 proteins, and interestingly suppressed ERK1 at the transcriptional level
Secreted aspartic protease-mediated proteolytic cleavage of Msb2 is required for activation of the Cek1 mitogen activated protein kinase (Montrer MAPK1 Kits ELISA) pathway in response to environmental cues.
The authors propose that a Msb2, Cek1 and Ace2 signalling pathway addresses PMT genes as downstream targets and that different modes of regulation have evolved for PMT1 and PMT2/PMT4 genes.
Msb2 is involved in the transmission of the signal toward Cek1 mediated by the Cdc42 (Montrer CDC42 Kits ELISA) GTPase (Montrer RACGAP1 Kits ELISA).
ERK1/2 (Montrer MAPK1/3 Kits ELISA)-Akt1 (Montrer AKT1 Kits ELISA) crosstalk regulates arteriogenesis in mice and zebrafish.
eena (Montrer SH3GL1 Kits ELISA) plays an important role in the development of the myeloid cell through activation of the ERK1/ERK2 (Montrer MAPK1 Kits ELISA) pathway
ERK1 and ERK2 (Montrer MAPK1 Kits ELISA) target common and distinct gene sets, confirming diverse roles for these kinases during embryogenesis; for ERK1 different specific genes involved in dorsal-ventral patterning and subsequent embryonic cell migration were identified.
These results demonstrate that induction of Hsp70 (Montrer HSPA1A Kits ELISA) in response to heat stress is dependent on ERK (Montrer MAPK1 Kits ELISA) activation in Pac2 (Montrer PSMG2 Kits ELISA) cells.
Data define distinct roles for ERK1 and ERK2 (Montrer MAPK1 Kits ELISA) in developmental cell migration processes during zebrafish embryogenesis.
GLUL (Montrer GLUL Kits ELISA) knockdown markedly inhibited the p38 MAPK (Montrer MAPK14 Kits ELISA) and ERK1/ERK2 (Montrer MAPK1 Kits ELISA) signaling pathways in cultured breast cancer cells and reduces their proliferation.
These results suggested that HOXB7 (Montrer HOXB7 Kits ELISA) stimulates ERK1/2 (Montrer MAPK1/3 Kits ELISA) phosphorylation and provided evidence that HOXB7 (Montrer HOXB7 Kits ELISA), besides its role in transcriptional regulation, also promotes cell motility and invasiveness.
High ERK1 expression is associated with castration-resistant prostate cancer.
combined use of butyrate and highly specific Syk (Montrer SYK Kits ELISA) inhibitor BAY61-3606 does not enhance differentiation and apoptosis of colonocytes. Instead, BAY completely abolishes butyrate-induced differentiation and apoptosis in a Syk (Montrer SYK Kits ELISA)- and ERK1/2 (Montrer MAPK1/3 Kits ELISA)-dependent manner.
new findings indicating that canonical FGFR (Montrer FGFR2 Kits ELISA)-ERK1/2 (Montrer MAPK1/3 Kits ELISA) signaling entrapped hBMSCs in a pre-committed state and arrested further maturation of committed precursors.
mutually exclusive transcriptional regulation by AP-1 (cjun (Montrer JUN Kits ELISA)/cfos) and non-canonical NF-kappaB (Montrer NFKB1 Kits ELISA) (RelB (Montrer RELB Kits ELISA)/p52 (Montrer FKBP4 Kits ELISA)) downstream of MEK (Montrer MAP2K1 Kits ELISA)-ERK (Montrer EPHB2 Kits ELISA) and NIK (Montrer MAP3K14 Kits ELISA)-IKK-alpha (Montrer CHUK Kits ELISA)-NF-kappaB2 (p100 (Montrer CUX1 Kits ELISA)) phosphorylation, respectively was responsible for persistent Ccl20 (Montrer CCL20 Kits ELISA) expression in the colonic cells.
LPS (Montrer IRF6 Kits ELISA)-activated ERK1,2 was at least partly involved in the observed effects on periodontal ligament stem cell differentiation capacity, acquisition of myofibroblastic attributes, and changes of their immunomodulatory features.
The findings indicate that ERK (Montrer EPHB2 Kits ELISA) and JNK (Montrer MAPK8 Kits ELISA) signaling pathways, as well as NF-kappaB (Montrer NFKB1 Kits ELISA)-mediated signaling are important contributors to the pathogenesis of Kashin-Beck disease.
The antitumor activity of scopoletin may be due to its strong anti-angiogenic effect, which may be mediated by its effective inhibition of ERK1, VEGF-A (Montrer VEGFA Kits ELISA), and FGF-2 (Montrer FGF2 Kits ELISA).
High ERK1 expression is associated with melanoma.
Cortical neuron-specific deletion of extracellular signal-regulated kinases Erk1 or Erk2 (Montrer MAPK1 Kits ELISA) significantly increased the duration of wakefulness.
pERK1/2 is a regulator of CD44 (Montrer CD44 Kits ELISA) expression, and increased CD44 (Montrer CD44 Kits ELISA) expression leads to a pro-sclerotic and migratory parietal epithelial cell phenotype in focal segmental glomerulosclerosis.
mmLDL increased the serum concentrations and expression of ICAM-1 (Montrer ICAM1 Kits ELISA) and VCAM-1 (Montrer VCAM1 Kits ELISA) by activating the ERK1/2 (Montrer MAPK1/3 Kits ELISA) pathway, resulting in the expression of ETB (Montrer EDNRB Kits ELISA) receptors and the enhancement of contractile function in vascular smooth muscle.
Angiotensin II regulates dendritic cells through activation of p65 NF-kappaB (Montrer NFkBP65 Kits ELISA), ERK1, ERK2 (Montrer MAPK1 Kits ELISA) and STAT1 (Montrer STAT1 Kits ELISA) pathways.
MAPK3/1 participates in primordial follicle activation through mTORC1-KITL (Montrer KITLG Kits ELISA) signaling.
At low oxLDL levels LOX-1 (Montrer OLR1 Kits ELISA) activates the protective Oct-1 (Montrer POU2F1 Kits ELISA)/SIRT1 (Montrer SIRT1 Kits ELISA) pathway, while at higher levels of the lipoprotein switches to the thrombogenic ERK1/2 (Montrer MAPK1/3 Kits ELISA) pathway.
Studies indicate that progesterone receptor (Montrer PGR Kits ELISA) transgenic (Pgrcre/+) mitogen inducible gene 6 (Mig (Montrer CXCL9 Kits ELISA)-6over) phosphatase and tensin homolog protein (Ptenf/f) knockout mice exhibited an increase of phospho-ERK1/2 (Montrer MAPK1/3 Kits ELISA) and its target genes.
Gpr182 reduction led to increased activation of ERK1/2 in basal and challenge models, demonstrating a potential role for this orphan GPCR in regulating the proliferative capacity of the intestine.
ERK1 underexpression is associated with obesity.
retinoschisin (Montrer RS1 Kits ELISA) is a novel regulator of MAP kinase (Montrer MAPK1 Kits ELISA) signalling and exerts an anti-apoptotic effect on retinal cells.
Early activation of MAPK p44/42 is involved in deoxynivalenol -induced disruption of intestinal barrier function and tight junction network signaling.
Pseudorabies virus glycoprotein gE-mediated ERK 1/2 phosphorylation also occurs in epithelial cells and in these cells, gE-mediated ERK 1/2 signaling is associated with degradation of the pro-apoptotic protein Bim (Montrer BCL2L11 Kits ELISA).
Treatment with ERK (Montrer MAPK1 Kits ELISA) inhibitors or ERK1/2 (Montrer MAPK1/3 Kits ELISA) knockdown significantly suppressed porcine epidemic diarrhea virus progeny production.
This study reveals a new function of the gE glycoprotein of pseudorabies virus and suggests that pseudorabies virus, through activation of ERK1/2 (Montrer MAPK1/3 Kits ELISA) signaling, has a substantial impact on T cell behavior.
CSF2 (Montrer CSF2 Kits ELISA) stimulates proliferation of trophectoderm cells by activation of the PI3K-and ERK1/2 (Montrer MAPK1/3 Kits ELISA) MAPK (Montrer MAPK1 Kits ELISA)-dependent MTOR (Montrer FRAP1 Kits ELISA) signal transduction cascades.
PGRN (Montrer GRN Kits ELISA) inhibits adipogenesis in porcine preadipocytes partially through ERK (Montrer MAPK1 Kits ELISA) activation mediated PPARgamma (Montrer PPARG Kits ELISA) phosphorylation.
Porcine circovirus type 2 (PCV2) might induce autophagy via the AMPK (Montrer PRKAA1 Kits ELISA)/ERK (Montrer MAPK1 Kits ELISA)/TSC2 (Montrer TSC2 Kits ELISA)/mTOR (Montrer FRAP1 Kits ELISA) signaling pathway in the host cells, representing a pivotal mechanism for PCV2 pathogenesis
Data show that proinflammatory cytokines induction was ERK1/2 (Montrer MAPK1/3 Kits ELISA) and JNK1 (Montrer MAPK8 Kits ELISA)/2 dependent.
Saccharomyces cerevisiae inhibits the Enterotoxigenic Escherichia coli-induced expression of pro-inflammatory transcripts and this inhibition was associated to a decrease of ERK1/2 (Montrer MAPK1/3 Kits ELISA) and p38 MAPK (Montrer MAPK14 Kits ELISA) phosphorylation
ERK1 phosphorylation in response to Insulin-like Growth Factor-1 (Montrer IGF1 Kits ELISA) does not require activation of the Insulin-like Growth Factor-1 receptor tyrosine kinase (Montrer IGF1R Kits ELISA)
The protein encoded by this gene is a member of the MAP kinase family. MAP kinases, also known as extracellular signal-regulated kinases (ERKs), act in a signaling cascade that regulates various cellular processes such as proliferation, differentiation, and cell cycle progression in response to a variety of extracellular signals. This kinase is activated by upstream kinases, resulting in its translocation to the nucleus where it phosphorylates nuclear targets. Alternatively spliced transcript variants encoding different protein isoforms have been described.
MAP kinase isoform p44
, MAPK 1
, extracellular signal-regulated kinase 1
, extracellular signal-related kinase 1
, insulin-stimulated MAP2 kinase
, microtubule-associated protein 2 kinase
, MAP kinase 3
, p44 MAP kinase
, pp42/MAP kinase
, mitogen-activated protein kinase 3
, MAP kinase 12
, MAPK 12
, extracellular signal-regulated kinase 6
, mitogen-activated protein kinase 12
, stress-activated protein kinase 3
, MAP kinase 1
, MAPK 3
, mitogen-activated 3
, mitogen-activated protein kinase 1
, extracellular signal-regulated kinase-1
, likely protein kinase