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FOXD3 belongs to the forkhead family of transcription factors which is characterized by a distinct forkhead domain. De plus, nous expédions FOXD3 Kits (12) et FOXD3 Protéines (4) et beaucoup plus de produits pour cette protéine.
Showing 10 out of 84 products:
Human Monoclonal FOXD3 Primary Antibody pour ELISA, WB - ABIN966161
Hromas, Moore, Johnston, Socha, Klemsz: Drosophila forkhead homologues are expressed in a lineage-restricted manner in human hematopoietic cells. dans Blood 1993
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Human Monoclonal FOXD3 Primary Antibody pour ELISA, WB - ABIN969152
Saleem, Banerjee-Basu, Berry, Baxevanis, Walter: Analyses of the effects that disease-causing missense mutations have on the structure and function of the winged-helix protein FOXC1. dans American journal of human genetics 2001
Human Monoclonal FOXD3 Primary Antibody pour IHC, ELISA - ABIN969153
Gregory, Barlow, McLay, Kaul, Swarbreck, Dunham, Scott, Howe, Woodfine, Spencer, Jones, Gillson, Searle, Zhou, Kokocinski, McDonald, Evans, Phillips, Atkinson, Cooper, Jones, Hall, Andrews, Lloyd et al.: The DNA sequence and biological annotation of human chromosome 1. ... dans Nature 2006
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Human Monoclonal FOXD3 Primary Antibody pour ICC, IF - ABIN256525
Huang, Kesselman, Kizub, Guerrero-Preston, Ratovitski: Phospho-?Np63?/microRNA feedback regulation in squamous carcinoma cells upon cisplatin exposure. dans Cell cycle (Georgetown, Tex.) 2013
Foxd3 rescues the prdm1a (Montrer PRDM1 Anticorps) loss-of-function neural crest phenotype.
These results reveal dynamic and differential regulation of FoxD3 in distinct neural crest subpopulations, suggesting that heterogeneity is encrypted at the regulatory level
analysis of how a FoxD3 gene trap line reveals neural crest precursor movement and a role for FoxD3 in their specification
tfap2a (Montrer TFAP2A Anticorps) and foxd3 are expressed during gastrulation prior to neural crest induction in distinct, complementary, domains; tfap2a (Montrer TFAP2A Anticorps) is expressed in the ventral non-neural ectoderm and foxd3 in the dorsal mesendoderm and ectoderm
analysis of a Foxd3/mitfa (Montrer MITF Anticorps) transcriptional switch that governs whether a bi-potent pigment precursor will attain either an iridophore or a melanophore fate
foxd3 is an essential Nodal-dependent regulator of zebrafish mesoderm development.
Decrement of function of foxd3 and/or sox10, two genes important for the development and specification of neural crest, resulted in a reduction and/or loss of GnRH cells of the midbrain, as well as a reduction in the number of terminal nerve GnRH cells.
zebrafish Foxd3 is necessary for the differentiation of a subset of neural crest cell fates, perhaps in precursors of particular neural crest lineages.
Foxd3, a well-known regulator in neural crest development, is also involved in myf5 (Montrer MYF5 Anticorps) regulation
Foxd3 selectively specifies premigratory neural crest cells for a neuronal, glial or cartilage fate by inducing the expression of lineage-associated transcription factors in these cells and regulating their subsequent migration.
FOXD3 knockdown resulted in enhanced ATC (Montrer SRPK1 Anticorps) proliferation, invasion and migration and diminished cellular apoptosis. Further, we showed that FOXD3 regulated expression of E-cadherin (Montrer CDH1 Anticorps) by modulating MAPK (Montrer MAPK1 Anticorps)/EKR signaling pathway that promotes EMT (Montrer ITK Anticorps) and metastasis during thyroid carcinogenesis.
Results show that FOXD3 expression was reduced in colon cancer cells. Its knockdown dramatically increased the proliferation of tumor cells, enhanced cell invasiveness and inhibited cell apoptosis. The study indicates that FOXD3 may play a protective role in human colon formation by regulating EGFR (Montrer EGFR Anticorps)/Ras/Raf (Montrer RAF1 Anticorps)/MEK (Montrer MAP2K1 Anticorps)/ERK (Montrer EPHB2 Anticorps) signal pathway.
FOXO4 (Montrer FOXO4 Anticorps) and FOXD3 were shown independently predictive of overall survival in gastric cancer
FOXD3/miR (Montrer MLXIP Anticorps)-214/MED19 (Montrer MED19 Anticorps) axis is important for the regulation of growth, invasion and metastasis of colorectal cancer
total of 1799 differentially methylated regions were identified including SLC6A3, Rab40C, ZNF584, and FOXD3 whose significant methylation differences were confirmed in breast cancer patients through quantitative real-time polymerase chain reaction.Methylation of those aforementioned genes in white blood cells of our young patients may highlight their potential as early epimarkers
Results showed that silencing FoxD3 in lung cancer cell lines stimulated cell growth and inhibited cell apoptosis.
FOXD3 is sufficient but not necessary to drive PAX3 (Montrer PAX3 Anticorps) expression in melanoma cells.
The present study finds that the aspirin-FOXD3-OLA1P2-STAT3 (Montrer STAT3 Anticorps) axis exhibits exciting anticancer effects and provides new insights into the chemopreventive mechanisms underlying aspirin use
FOXD3 might serve as an independent prognostic biomarker and a potential therapeutic target for high-grade gliomas, which warrant further investigation.
Down-regulation of FOXD3 is associated with metastasis in hepatocellular carcinoma.
FOXD3-AS1 (Montrer ARSB Anticorps) serves as a sponge or as a competing endogenous noncoding RNA for miR (Montrer MLXIP Anticorps)-150, restricting its capability to promote cell growth and thereby exaggerating hyperoxia-induced lung epithelial cell death
suggest that CHD7 (Montrer CHD3 Anticorps), Oct3/4 (Montrer POU5F1 Anticorps), Sox2 (Montrer SOX2 Anticorps), and Nanog (Montrer NANOG Anticorps) directly induce FoxD3 expression when stimulated by BMP2 (Montrer BMP2 Anticorps)/Wnt3a (Montrer WNT3A Anticorps) signaling, that FoxD3 promotes Sox10 (Montrer SOX10 Anticorps) expression, and that histone H3K4 methylation play important roles in this process of neural crest-derived stem cell formation
this study shows that Foxd3 suppresses the production of IL-10 (Montrer IL10 Anticorps)+ Breg cells by directly binding the IL-10 (Montrer IL10 Anticorps) promoter
Foxd3 poises enhancers in pluripotent stem cells by recruiting multiple epigenetic enzymes that together simultaneously initiate and repress enhancer activity.
analysis of a cycle of activation and deactivation of Foxd3 required for exit from naive pluripotency and subsequent primordial germ cell specification
Foxd3 suppresses NFAT (Montrer NFATC1 Anticorps)-mediated differentiation to maintain self-renewal of embryonic stem cells
In a xenograft tumor model, FOXD3 overexpression inhibits tumor growth and angiogenesis.
Data indicate that homeobox b5 (Hoxb5 (Montrer HOXB5 Anticorps)) regulated the neural crest (NC)development by directly inducing Forkhead box D3 gene (Foxd3).
Foxd3 induced mutant ESCs (Montrer NR2E3 Anticorps) precociously express genes required for mesoderm induction, but they are likely unable to differentiate into skeletal muscle.
Data indicate that growth factor receptor (Montrer RYK Anticorps) protein binding protein 2 (Grb2 (Montrer GRB2 Anticorps)) is upregulated and regulated by Forkhead Box D3 (Foxd3), and pregulated Grb2 (Montrer GRB2 Anticorps) interacts with huntingtin (Htt (Montrer HTT Anticorps)).
This gene belongs to the forkhead family of transcription factors which is characterized by a distinct forkhead domain. Mutations in this gene cause autoimmune susceptibility 1.
fork head domain protein 6
, forkhead box protein D3
, mother superior
, forkhead box D3
, HNF3/FH transcription factor genesis
, HNF-3/forkhead homolog 2
, hepatocyte nuclear factor 3 forkhead homolog 2
, winged helix protein CWH-3
, winged-helix protein CWH-3