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Drosophila that have mutations in their mago nashi (grandchildless) gene produce progeny with defects in germplasm assembly and germline development. De plus, nous expédions Mago-Nashi Homolog, Proliferation-Associated (Drosophila) Protéines (14) et et beaucoup plus de produits pour cette protéine.
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Human Polyclonal MAGOH Primary Antibody pour ELISA, WB - ABIN268734
Zhao, Colaizzo-Anas, Nowak, Shows, Elliott, Aplan: The mammalian homologue of mago nashi encodes a serum-inducible protein. dans Genomics 1998
Results provide evidence that MAGOH binding to the C-terminal region of Y14 disrupts Y14 nucleolar localization.
findings show 2 genes MAGOH and MAGOHB are expressed in mammals; in macrophages, expression of MAGOHB but not MAGOH mRNA increases after LPS stimulation; both MAGOH proteins interact with other exon junction complex (EJC) components, incorporate into mRNA-bound EJCs and activate nonsense-mediated decay
The stable association of multiprotein exon junction complex core with RNA is maintained by inhibition of eIF4AIII ATPase activity by MAGOH-Y14.
crystal structure of a tetrameric exon junction core complex containing the DEAD-box adenosine triphosphatase eukaryotic initiation factor 4AIII bound to an ATP analog, MAGOH, Y14, a fragment of MLN51, and a polyuracil mRNA mimic
These results indicate that MAGOH regulates the transcriptional activation of STAT3 by interfering complex formation between STAT3 and Y14.
we first show that Eif4a3 haploinsufficiency phenocopies aberrant neurogenesis and microcephaly of Magoh and Rbm8a mutant mice.we show that genetic ablation of one downstream pathway, p53, significantly rescues microcephaly of all 3 EJC mutant
These results point to a central role for Magoh in melanocyte development.
mouse magoh is involved in cyclin-dependent kinase regulation.
Magoh haploinsufficiency causes microcephaly because of intermediate neural progenitors depletion and neuronal apoptosis.
Zygotic mago nashi is expressed into the dorsal-marginal region during gastrulation.
MAPK is the primary functional target of mago in eye development; in cultured cells, Mago knockdown disproportionately affects other large genes located in heterochromatin
Mago Nashi, Tsunagi/Y14, and Ranshi form a complex that influences oocyte differentiation in Drosophila melanogaster
We have determined the crystal structure of the complex between Drosophila melanogaster Y14 and Mago at a resolution of 2.5 A. The structure reveals an atypical mode of protein-protein recognition mediated by an RNA-binding domain (RBD).
mago nashi is required for germline stem cell differentiation, but surprisingly mago nashi functions independently of tsunagi/Y14 in this process
Drosophila that have mutations in their mago nashi (grandchildless) gene produce progeny with defects in germplasm assembly and germline development. This gene encodes the mammalian mago nashi homolog. In mammals, mRNA expression is not limited to the germ plasm, but is expressed ubiquitously in adult tissues and can be induced by serum stimulation of quiescent fibroblasts.
protein mago nashi homolog
, mago-nashi-like proliferation-associated protein
, mago-nashi homolog, proliferation-associated
, mago-nashi homolog, proliferation-associated (Drosophila)
, mago nashi
, CG9401 gene product from transcript CG9401-RA
, mago nishi
, mago nashi homolog