HILPDA anticorps (C-Term)

N° du produit ABIN1887137

Détail du produit anti-HILPDA anticorps

Antigène: HILPDA (Hypoxia Inducible Lipid Droplet-Associated (HILPDA))

Épitope: C-Term

Reactivité: Humain, Souris

Hôte: Lapin

Clonalité: Polyclonal

Conjugué: Cet anticorp HILPDA est non-conjugé

Application: Western Blotting (WB), ELISA

Réactivité croisée (Details): At least two isoforms of HIG2 are known to exist, this antibody will detect both isoforms. HIG2 antibody is predicted to not cross-react with HIG1

Purification: Affinity chromatography purified via peptide column

Immunogène: 16 amino acid peptide near the carboxy terminus of human HIG2.

Information d'application

Restrictions: For Research Use only

Stockage

Format: Liquid

Buffer: PBS containing 0.02 % sodium azide.

Agent conservateur: Sodium azide

Précaution d'utilisation: WARNING: Reagents contain sodium azide. Sodium azide is very toxic if ingested or inhaled. Avoid contact with skin, eyes, or clothing. Wear eye or face protection when handling. If skin or eye contact occurs, wash with copious amounts of water. If ingested or inhaled, contact a physician immediately. Sodium azide yields toxic hydrazoic acid under acidic conditions. Dilute azide-containing compounds in running water before discarding to avoid accumulation of potentially explosive deposits in lead or copper plumbing.

Conseil sur la manipulation: Avoid freezing and thawing repeatly.

Stock: 4 °C/-20 °C

Stockage commentaire: Store at 4 °C for short term use.Store at -20 °C for long term preservation.

Détails sur HILPDA

Antigène: HILPDA (Hypoxia Inducible Lipid Droplet-Associated (HILPDA))

Autre désignation: HIG2 (HILPDA Produits)

Synonymes: anticorps 2310016C08Rik, anticorps AW049004, anticorps Hig2, anticorps C7orf68, anticorps HIG-2, anticorps HIG2, anticorps hypoxia inducible lipid droplet associated, anticorps Hilpda, anticorps HILPDA

Sujet: HIG1 and HIG2 (Hypoxia-inducible gene 1 and 2, respectively) are known to be induced by hypoxic conditions.HIG2 is induced by hypoxia and by glucose deprivation in cultured cells.In addition, tumor xenografts derived from human cervical cancer cells display increased expression of HIG1 and HIG2 when they are deprived of oxygen.Unlike HIG2, which is ubiquitously expressed and might be an activator and target of the canonical Wnt pathway, the function and the mechanisms underlying its regulation of HIG1 still remained unknown.The putative link between hypoxia and an oncogenic signaling pathway might play an important role in tumorigenesis.
Synonyms: Hypoxia-inducible gene 2 protein, HIG-2

NCBI Accession: NP_001092256