Tel:
+49 (0)241 95 163 153
Fax:
+49 (0)241 95 163 155
E-Mail:
orders@anticorps-enligne.fr

RETNLB anticorps

RETNLB Reactivité: Humain ELISA, IHC Hôte: Lapin Polyclonal unconjugated
N° du produit ABIN2422115
  • Antigène Voir toutes RETNLB Anticorps
    RETNLB (Resistin Like beta (RETNLB))
    Reactivité
    • 50
    • 18
    Humain
    Hôte
    • 64
    • 2
    • 1
    • 1
    Lapin
    Clonalité
    • 65
    • 3
    Polyclonal
    Conjugué
    • 30
    • 11
    • 5
    • 3
    • 2
    • 2
    • 2
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    Cet anticorp RETNLB est non-conjugé
    Application
    • 51
    • 33
    • 13
    • 13
    • 8
    • 4
    • 4
    • 4
    • 2
    • 2
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    ELISA, Immunohistochemistry (IHC)
    Purification
    Affinity purification
    Immunogène
    Recombinant protein of human RETNLB
    Isotype
    IgG
    Top Product
    Discover our top product RETNLB Anticorps primaire
  • Indications d'application
    IHC 1:50-1:200
    Restrictions
    For Research Use only
  • Format
    Liquid
    Concentration
    0.3 mg/mL
    Buffer
    PBS with 0.05 % sodium azide and 50 % glycerol, PH7.4
    Agent conservateur
    Sodium azide
    Conseil sur la manipulation
    Avoid freeze / thaw cycles.
    Stock
    -20 °C
    Stockage commentaire
    Store at -20°C. Avoid freeze / thaw cycles.
  • Antigène
    RETNLB (Resistin Like beta (RETNLB))
    Autre désignation
    RETNLB (RETNLB Produits)
    Synonymes
    anticorps FIZZ1, anticorps FIZZ2, anticorps HXCP2, anticorps RELM-beta, anticorps RELMb, anticorps RELMbeta, anticorps XCP2, anticorps RETNLB, anticorps 9030012B21Rik, anticorps Fizz2, anticorps Relmb, anticorps Xcp3, anticorps resistin like beta, anticorps RETNLB, anticorps Retnlb
    Sujet
    Resistin-like betais aproteinthat in humans is encoded by theRETNLBgene. An infusion of either resistin or RELMB in rats rapidly induced severe hepatic but not peripheral insulin resistance. Increases in circulating resistin or RELMB levels markedly stimulated hepatic glucose production despite the presence of fixed physiologic insulin levels. This enhanced rate of glucose output was due to increased flux through glucose-6-phosphatase. The results supported the notion that a novel family of fat- and gut-derived circulating proteins modulates hepatic insulin action.
    Pathways
    Hormone Activity
Vous êtes ici:
Support technique