ARHGAP24 anticorps (C-Term)

N° du produit ABIN5538466

Détail du produit anti-ARHGAP24 anticorps

Antigène: ARHGAP24 (rho GTPase Activating Protein 24 (ARHGAP24))

Épitope: AA 587-616, C-Term

Reactivité: Humain

Hôte: Lapin

Clonalité: Polyclonal

Conjugué: Cet anticorp ARHGAP24 est non-conjugé

Application: Western Blotting (WB)

Purification: This antibody is purified through a protein A column, followed by peptide affinity purification.

Immunogène: This ARHGAP24 antibody is generated from rabbits immunized with a KLH conjugated synthetic peptide between 587-616 amino acids from the C-terminal region of human ARHGAP24.

Isotype: Ig Fraction

Information d'application

Indications d'application: For WB starting dilution is: 1:1000

Restrictions: For Research Use only

Stockage

Format: Liquid

Concentration: 0.41 mg/mL

Buffer: Supplied in PBS with 0.09 % (W/V) sodium azide.

Agent conservateur: Sodium azide

Précaution d'utilisation: This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

Stock: 4 °C,-20 °C

Stockage commentaire: Store at 4°C for three months and -20°C, stable for up to one year. As with all antibodies care should be taken to avoid repeated freeze thaw cycles. Antibodies should not be exposed to prolonged high temperatures.

Détails sur ARHGAP24

Antigène: ARHGAP24 (rho GTPase Activating Protein 24 (ARHGAP24))

Autre désignation: ARHGAP24 (ARHGAP24 Produits)

Synonymes: anticorps FILGAP, anticorps RC-GAP72, anticorps RCGAP72, anticorps p73, anticorps p73RhoGAP, anticorps 0610025G21Rik, anticorps si:ch211-65i12.2, anticorps Rho GTPase activating protein 24, anticorps ARHGAP24, anticorps Arhgap24, anticorps arhgap24

Sujet: ARHGAPs, such as ARHGAP24, encode negative regulators of Rho GTPases (see ARHA, MIM 165390), which are implicated in actin remodeling, cell polarity, and cell migration (Katoh and Katoh, 2004 [PubMed 15254788]).

Poids moléculaire: 84 kDa

ID gène: 83478

UniProt: Q8N264

Pathways: Regulation of Cell Size, Positive Regulation of Response to DNA Damage Stimulus