CLCN4 anticorps
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- Antigène Voir toutes CLCN4 Anticorps
- CLCN4 (Chloride Channel 4 (CLCN4))
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Reactivité
- Humain, Rat, Souris
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Hôte
- Lapin
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Clonalité
- Polyclonal
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Conjugué
- Cet anticorp CLCN4 est non-conjugé
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Application
- Western Blotting (WB)
- Purification
- Affinity purification
- Immunogène
- Recombinant protein of human CLCN4
- Isotype
- IgG
- Top Product
- Discover our top product CLCN4 Anticorps primaire
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- Indications d'application
- WB 1:500 - 1:2000
- Commentaires
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Abundant in skeletal muscle and also detectable in brain and heart
- Restrictions
- For Research Use only
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- Buffer
- PBS with 0.02 % sodium azide, 50 % glycerol, pH 7.3.
- Agent conservateur
- Sodium azide
- Précaution d'utilisation
- This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
- Stock
- -20 °C
- Stockage commentaire
- Store at -20C. Avoid freeze / thaw cycles.
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- Antigène
- CLCN4 (Chloride Channel 4 (CLCN4))
- Autre désignation
- CLCN4 (CLCN4 Produits)
- Synonymes
- anticorps Clc4-2, anticorps Clcn4, anticorps CLC4, anticorps ClC-4, anticorps ClC-4A, anticorps Clcn4-2, anticorps chloride voltage-gated channel 4, anticorps chloride channel, voltage-sensitive 4, anticorps CLCN4, anticorps clcn4, anticorps Clcn4
- Sujet
- The CLCN family of voltage-dependent chloride channel genes comprises nine members (CLCN1-7, Ka and Kb) which demonstrate quite diverse functional characteristics while sharing significant sequence homology. Chloride channel 4 has an evolutionary conserved CpG island and is conserved in both mouse and hamster. This gene is mapped in close proximity to APXL (Apical protein Xenopus laevis-like) and OA1 (Ocular albinism type I), which are both located on the human X chromosome at band p22.3. The physiological role of chloride channel 4 remains unknown but may contribute to the pathogenesis of neuronal disorders. Alternate splicing results in two transcript variants that encode different proteins.
- Poids moléculaire
- 84.917 kDa
- ID gène
- 1183
- UniProt
- P51793
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