APOA2 anticorps
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- Antigène Voir toutes APOA2 Anticorps
- APOA2 (Apolipoprotein A-II (APOA2))
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Reactivité
- Rat, Souris
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Hôte
- Lapin
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Clonalité
- Polyclonal
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Conjugué
- Cet anticorp APOA2 est non-conjugé
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Application
- Immunohistochemistry (IHC), Immunofluorescence (IF)
- Réactivité croisée
- Rat
- Purification
- Affinity purification
- Immunogène
- Recombinant protein corresponding to Mouse Apolipoprotein A II
- Top Product
- Discover our top product APOA2 Anticorps primaire
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- Indications d'application
- IHC/IF (M,R) 1:400-1:1300
- Restrictions
- For Research Use only
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- Format
- Liquid
- Buffer
- PBS, pH 7.4, 0.02 % sodium azide
- Agent conservateur
- Sodium azide
- Précaution d'utilisation
- This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
- Stock
- -20 °C
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- Antigène
- APOA2 (Apolipoprotein A-II (APOA2))
- Autre désignation
- Apolipoprotein A II/ApoA-II (APOA2 Produits)
- Synonymes
- anticorps Apo-AII, anticorps ApoA-II, anticorps apoAII, anticorps Alp-2, anticorps ApoAII, anticorps Apoa-2, anticorps Hdl-1, anticorps APOAII, anticorps apoa2, anticorps apoA-II, anticorps cb1032, anticorps wu:fb57h11, anticorps zgc:193613, anticorps apolipoprotein A2, anticorps apolipoprotein A-II, anticorps APOA2, anticorps Apoa2, anticorps apoa2
- Sujet
- Apolipoprotein A II is the second most abundant protein of the high density lipoprotein particles. The apolipoprotein A II gene consists of 4 exons and 3 introns. The four exons encode the 5' untranslated region, pre peptide, a short N terminal domain and a C terminal domain composed of a variable number of lipid binding amphipathic helices. Familial apolipoprotein A II deficiency may result from a splice junction alteration which blocks splicing of intron 3 from the primary transcript and result in the formation of a non functional mRNA.
- ID gène
- 11807
- NCBI Accession
- NP_001292478
- UniProt
- P09813
- Pathways
- Regulation of Lipid Metabolism by PPARalpha, Production of Molecular Mediator of Immune Response, Negative Regulation of Transporter Activity, Lipid Metabolism
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