Selenoprotein S anticorps (AA 125-175)

N° du produit ABIN7449819

Cet anticorps anti-Selenoprotein S est un anticorps Lapin Polyclonal détectant Selenoprotein S dans WB et IP. Adapté pour Humain.

Aperçu rapide pour Selenoprotein S anticorps (AA 125-175) (ABIN7449819)

Antigène: Selenoprotein S (SELS)

Reactivité: Humain

Hôte: Lapin

Clonalité: Polyclonal

Conjugué: Cet anticorp Selenoprotein S est non-conjugé

Application: Western Blotting (WB), Immunoprecipitation (IP)

Détail du produit anti-Selenoprotein S anticorps

Épitope: AA 125-175

Fonction: Rabbit anti-Selenoprotein S Antibody, Affinity Purified

Purification: Affinity Purified

Immunogène: Between AA 125 and 175

Isotype: IgG

Information d'application

Indications d'application:

IP: 2 - 10 μg/mg lysate

WB: 1:1,000 - 1:5,000

Restrictions: For Research Use only

Stockage

Concentration: 1000 μg/mL

Buffer: Tris-citrate/phosphate buffer, pH 7 to 8 containing 0.09 % Sodium Azide

Agent conservateur: Sodium azide

Précaution d'utilisation: This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

Stock: 4 °C

Date de péremption: 12 months

Détails sur Selenoprotein S

Antigène: Selenoprotein S (SELS)

Autre désignation: Selenoprotein S

Sujet: Background: Selenoprotein S (SELS) is involved in the degradation process of misfolded endoplasmic reticulum (ER) luminal proteins. Selenoprotein S participates in the transfer of misfolded proteins from the ER to the cytosol, where they are destroyed by the proteasome in a ubiquitin-dependent manner. It probably acts by serving as a linker between DERL1, which mediates the retrotranslocation of misfolded proteins into the cytosol, and the ATPase complex VCP, which mediates the translocation and ubiquitination [taken from the Universal Protein Resource (UniProt) Q9BQE4].

ID gène: 55829

NCBI Accession: NP_982298

UniProt: Q9BQE4

Pathways: Cellular Response to Molecule of Bacterial Origin, ER-Nucleus Signaling, Regulation of Carbohydrate Metabolic Process, Cell RedoxHomeostasis, Negative Regulation of intrinsic apoptotic Signaling, SARS-CoV-2 Protein Interactome