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anti-Rat (Rattus) CCL5 Anticorps:
anti-Human CCL5 Anticorps:
anti-Mouse (Murine) CCL5 Anticorps:
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Human Monoclonal CCL5 Primary Antibody pour CyTOF, FACS - ABIN4900684
van Bladel, Roest, de Groot, Schutgens: Up-regulation of platelet activation in hemophilia A. dans Haematologica 2011
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Cow (Bovine) Polyclonal CCL5 Primary Antibody pour IF (p), IHC (p) - ABIN674949
Pattappa, Peroglio, Sakai, Mochida, Benneker, Alini, Grad: CCL5/RANTES is a key chemoattractant released by degenerative intervertebral discs in organ culture. dans European cells & materials 2014
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Human Polyclonal CCL5 Primary Antibody pour ELISA, WB - ABIN3043478
Li, Sun, Liao, Ma, Ma, Zhang: Regulated upon activation normal T-cell expressed and secreted originating from the epididymis differentially associates with viable and defective spermatozoa. dans Fertility and sterility 2010
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Human Monoclonal CCL5 Primary Antibody pour Inhibition, Neut - ABIN2689890
Prussin, Metcalfe: Detection of intracytoplasmic cytokine using flow cytometry and directly conjugated anti-cytokine antibodies. dans Journal of immunological methods 1996
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Human Monoclonal CCL5 Primary Antibody pour ICC - ABIN2689891
Sticherling, Küpper, Koltrowitz, Bornscheuer, Kulke, Klinger, Wilhelm, Kameyoshi, Christophers, Schröder: Detection of the chemokine RANTES in cytokine-stimulated human dermal fibroblasts. dans The Journal of investigative dermatology 1995
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Mouse (Murine) Polyclonal CCL5 Primary Antibody pour Func, ELISA - ABIN2476319
Bizanek, McGuinness, Nakanishi, Tomasz: Isolation and structure of an intrastrand cross-link adduct of mitomycin C and DNA. dans Biochemistry 1992
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Human Monoclonal CCL5 Primary Antibody pour Inhibition, ELISA - ABIN934074
Nieto, Zamora, Cantó, Garcia-Planella, Gordillo, Ortiz, Juárez, Vidal: CSF-1 regulates the function of monocytes in Crohn's disease patients in remission. dans Scientific reports 2017
Cow (Bovine) Polyclonal CCL5 Primary Antibody pour IHC (p) - ABIN674951
Li, Liu, Duan, Tian, Zhu, He, Yao, Yi, Song, Tang: Batf3-dependent CD8α+Dendritic Cells Aggravates Atherosclerosis via Th1 Cell Induction and Enhanced CCL5 Expression in Plaque Macrophages. dans EBioMedicine 2018
Rat (Rattus) Polyclonal CCL5 Primary Antibody pour WB - ABIN110550
Adamus, Manczak, Machnicki: Expression of CC chemokines and their receptors in the eye in autoimmune anterior uveitis associated with EAE. dans Investigative ophthalmology & visual science 2001
Human Monoclonal CCL5 Primary Antibody pour FACS - ABIN4898183
Lavini-Ramos, Silva, Soares-Schanoski, Monteiro, Ferreira, Pacanaro, Gomes, Batista, Faé, Kalil, Coelho: MMP9 integrates multiple immunoregulatory pathways that discriminate high suppressive activity of human mesenchymal stem cells. dans Scientific reports 2017
This study provided evidence that miR-146a targets CCL5 3' untranslated regions, downregulates its release from macrophages, and affects monocyte migration consequently. These findings drew a novel layer of posttranscriptional control of the chemokine CCL5 by miR-146a during HIV infection, which might contribute to HIV pathogenesis.
Long noncoding RNA SNHG16 regulated LPS-induced inflammation injury in WI-38 cells through competitively binding miR-146a-5p with CCL5 further mediating JNK and NF-kappaB pathways, which sheds novel light on diagnostics and therapeutics in pneumonia.
The CCL5 rs2107538 polymorphism was identified to be associated with a significantly lower risk of benign prostatic hyperplasia (BPH). This polymorphism was also associated with the development of larger prostate volumes in Chinese patients with BPH.
This study explored the interaction of CCL5 with GPR75, an orphan receptor of the Gqalpha family of GPCRs, which appears to be expressed more abundantly in neuron-like cells than astrocytes.
data show that a genetic variant of CCL5 confers protection against Chagas heart disease
CCL5 deficiency could reverse obliterative changes in pulmonary arteries via caveolin-1-dependent amplification of BMPR2 signaling.
loss of CCL5 could enhance CXCL1 expression in hepatocytes and activate CXCL1-CXCR2 axis in neutrophils to augment their hepatic infiltration
these findings show that IL-6 and CCL5 signaling, which promote crosstalk between triple-negative breast cancer (TNBC) and lymphatic vessels, are key enhancers of TNBC tumor growth and metastasis. Furthermore, these results demonstrate that a drug combination inhibiting these pathways may be a promising therapy for TNBC patients
CCL5/CCR5 axis may be involved in the perineural invasion of salivary adenoid cystic carcinoma cells
In burn wounds, RANTES/CCL5 levels were on average 14.86pg/ml in young vs 4.26pg/ml in aged (p=0.026).
Combination of IP-10 and RANTES could be potentially used as diagnostic and monitoring biomarker in pulmonary tuberculosis management.
Our findings reveal the structural determinants involved in the recognition of CCL5 by the CCR5 N terminus. These findings, together with existing structural data, provide a complete structural framework with which to understand the specificity of receptor:chemokine interactions.
Genetic variants in the CCL5/CCR5 pathway may serve as prognostic markers and may predict severe hand-foot skin reaction in metastatic colorectal cancer patients receiving regorafenib therapy.
The secretion of CCL5 and CCL2 was stimulated in undifferentiated and decidualized ESCs by the combination of TNF-alpha and IFN-gamma under non-apoptotic as well as apoptotic (with Fas-stimulation in parallel) conditions. TNF-alpha or IFN-gamma alone did not have this effect. The stimulatory influence of TNF-alpha plus IFN-gamma on CCL5 and CCL2 in ESCs was also seen on the transcriptional level.
this study shows that EMSY is increased and activates TSLP & CCL5 expression in eosinophilic esophagitis
These CCL5 derivatives may now be tested against several inflammation-related pathologies where the CCL5:CCR5 axis plays a relevant role.
The CCL5 In1.1T/C polymorphism may modulate pulmonary early-onset tuberculosis risk.
We use CPRC prostate cancer model and demonstrate that endothelial cells secrete large amount of CCL5 and induces autophagy by suppressing AR expression in prostate cancer cell lines. Consequently, elevated autophagy accelerates focal adhesions proteins disassembly and promoted prostate cancer invasion. Inhibition of both CCL5/CCR5 signaling and autophagy significantly reduces metastasis in vivo
CCL5, from endothelial cells, acts in a paracrine fashion on triple-negative breast cancer (TNBC) cells to enhance their migration, invasion, and metastasis. CCL5, in turn, accelerates TNBC cell secretion of PAI-1 and promotes TNBC cell metastasis, thus forming a positive feedback loop. Moreover, this enhanced metastatic ability is reversible and dependent on CCL5 signaling via the chemokine receptor, CCR5.
high concentration of plasma CCL5 may promote EMT of breast cancer cells. Plasma CCL5 could be a promised candidate to predict chemotherapy response in NCT of LABC.
Our study provides the first evidence of an exercise-reducible myokine, CCL5, in the mouse skeletal muscle.
our results demonstrate a sequence of early events elicited by CCL5 chemoattracting macrophage that result in inflammatory aggravation of hepatic IRI.
the results show that Broadleaf Mahonia is a novel effective antiinflammatory herb in vitro and ex vivo, and that CCL5 may be closely associated with Granulomatous lobular mastitispathogenesis
miR-155 enhances venous neointima formation through the autocrine and paracrine effects of smooth muscle-like cell-derived RANTES in a nuclear factor kappaB-dependent manner in arteriovenous shunts.
Macrophage subtypes enhanced the osteogenesis in transwell setting and the transition from M1 to M2 was associated with an increase in bone anabolic factors CCL2/MCP-1, CCL5/RANTES and IGF-1 in vitro.
Taken together, our data suggest that CCR5 regulates insulin signaling in hypothalamus which contributes to systemic insulin sensitivity and glucose metabolism.
identifies the essential role of the chemoattractive cytokine CCL5 for liver disease progression and especially hepatocellular carcinoma development
Breast cancer cell CCL5 mediates bone marrow independent angiogenesis via paracrine signaling.
Studied the effects of CCL5-CCR5 interactions in breast cancer metabolism, and findings suggest that CCL5-CCR5 interactions in the tumor microenvironment modulate metabolic events during tumor onset to promote tumorigenesis.
RANTES levels were associated with TMS measures of cortical synaptic excitability, but not with long-term potentiation (LTP)-like plasticity.
study found that the inflammatory chemokine CCL5 is mostly retained (75%) during the resolution of zymosan A peritonitis in mice; CCL5 exerts a novel proresolving role on macrophages when acting in concert with apoptotic PMN-expressed D6.
A robust up-regulation of RANTES within the brain was seen in a mouse model of tick-borne encephalitis.
CCL5 paradoxically limits macrophage accumulation in the injured kidney during renin angiotensin system (RAS) activation by constraining the proinflammatory actions of CCL2.
This study showed that RANTES is important in the regulation of vascular dysfunction through modulation of perivascular inflammation.
IL-6 Mediates Macrophage Infiltration after Irradiation via Up-regulation of CCL2/CCL5 in Non-small Cell Lung Cancer
Blocking antibodies against RANTES and eotaxin reduced the infiltration of CD4(+) and CD8(+) T cells into the nigra, attenuated nigral expression of proinflammatory molecules, and suppressed nigral activation of glial cells. These findings paralleled dopaminergic neuronal protection, normalized striatal neurotransmitters, and improved motor functions in MPTP-intoxicated mice.
CCL5 deficiency resulted in reduced neointima formation after carotid artery injury and thrombosis.
RANTES produced by renal tubular cells act as a key chemokine in acute kidney injury and HIF-1alpha regulated LncRNA-PRINS might be involved in RANTES production.
Mycobacterium chelonae activates the gene expressions of chemokine (C-C motif) ligand 2 (CCL2) and CCL5 in murine bone marrow-derived macrophages and in vivo mouse model.
These results represent an important molecular mechanism whereby H. parasuis induced RANTES in the inflammatory response.
Foot-and-mouth disease virus leader proteinase inhibits dsRNA-induced RANTES transcription.
we cloned the nucleotide sequence of the 5'-flanking region of the porcine RANTES (poRANTES) gene and characterized the regulatory elements that activate transcription
The Toll-like receptor signaling pathway is involved in porcine reproductive and respiratory syndrome virus-induced RANTES activation.
These results suggest that porcine RANTES can play an important role in xenotransplant rejection, through participating in the interaction between porcine endothelial cells and human monocytes.
CCL5 but not CCL2 mainly attract bovine classical monocytes and promote their differentiation into LPS-hypo-responsive macrophages.
decrease in sensitivity of HIV variants to RANTES neutralization during the course of progressive infection, but not during follow-up of long term survivors; data suggest a role for RANTES neutralization sensitivity of HIV-1 in AIDS pathogenesis
In the pathogenesis of allergic rhinitis, substance P induces expression of RANTES in nasal mucosa.
CCL3, CCL4 and CCL5 gene expression was evaluated in response to simian-human immunodeficiency virus (SHIV) infection in a rhesus macaque model.
This gene is one of several CC cytokine genes clustered on the q-arm of chromosome 17. Cytokines are a family of secreted proteins involved in immunoregulatory and inflammatory processes. The CC cytokines are proteins characterized by two adjacent cysteines. The cytokine encoded by this gene functions as a chemoattractant for blood monocytes, memory T helper cells and eosinophils. It causes the release of histamine from basophils and activates eosinophils. This cytokine is one of the major HIV-suppressive factors produced by CD8+ cells. It functions as one of the natural ligands for the chemokine receptor CCR5 and it suppresses in vitro replication of the R5 strains of HIV-1, which use CCR5 as a coreceptor.
chemokine (C-C motif) ligand 5
, small inducible cytokine A5
, C-C motif chemokine 5
, T-cell-specific protein RANTES
, regulated upon activation normal T-cell expressed and secreted
, small-inducible cytokine A5
, T-cell specific protein p288
, beta-chemokine RANTES
, eosinophil chemotactic cytokine
, regulated upon activation, normally T-expressed, and presumably secreted
, small inducible cytokine subfamily A (Cys-Cys), member 5
, chemokine ah294
, small inducible cytokine A5 RANTES
, chemokine CCL5/RANTES
, chemokine ligand 5