Aperçu des produits pour anti-CCL5 (CCL5) Anticorps

Human Chemokine (C-C Motif) Ligand 5 (CCL5) interaction partners

1. KLF5-regulating cancer-associated fibroblasts affect gastric cancer cells progression by CCL5 secretion and activation of CCR5.

2. Data show that plasminogen activator inhibitor-1 (PAI-1 (Montrer SERPINE1 Anticorps)) and chemokine (Montrer CCL1 Anticorps) CCL5 (CCL5) overexpression promoted cell proliferation and migration in breast cancer cells.

3. Among infants with lower CCL5 levels, the Haemophilus-dominant microbiota profile was associated with a higher risk of intensive care use and hospital length-of-stay >/=3 days compared to the Moraxella-dominant profile. Conversely, among those with higher CCL5 levels, there were no significant associations between the microbiota profiles and these severity outcomes

4. The current study suggests that TLR3 (Montrer TLR3 Anticorps) signaling induces CCL5 expression via NF-kappaB (Montrer NFKB1 Anticorps) and IRF3 (Montrer IRF3 Anticorps) in bile duct cells, and this pathway may be involved in the pathogenesis of BA.

5. Study demonstrates that increased CCL5 expression was restricted to human mesenchymal glioblastoma (GBM) and suggests that CCL5 functions in an autocrine growth-promoting circuit, and establish a new receptor responsible for CCL5 function in mesenchymal glioblastoma cells.

6. Study showed that bone stromal cells promoted prostate cancer progression through the secretion of CCL5. In vitro co-culture of bone stromal cells with prostate cancer cells induced the expression of CCL5, which promoted prostate cancer cell migration. CCL5 was found to have a key role in the progression of prostate cancer in the bone metastasis microenvironment.

7. identifies the essential role of the chemoattractive cytokine CCL5 for liver disease progression and especially hepatocellular carcinoma development

8. Breast cancer cell CCL5 mediates bone marrow independent angiogenesis via paracrine signaling.

9. the present study has demonstrated a novel pathway involving CCl5/CCR1/beta-catenin/Slug, via which human Mesenchymal stem cells promotes colorectal cancer development.

10. The present study suggest that TT genotype of CCL5 In1.1T/C (rs2280789) polymorphism play an important role to increased CCL5 expression in T cell which may enhanced Th1 (Montrer TH1L Anticorps) immunity and help in protection against tuberculosis

Mouse (Murine) Chemokine (C-C Motif) Ligand 5 (CCL5) interaction partners

1. miR (Montrer MLXIP Anticorps)-155 enhances venous neointima formation through the autocrine and paracrine effects of smooth muscle-like cell-derived RANTES in a nuclear factor kappaB-dependent manner in arteriovenous shunts.

2. Macrophage subtypes enhanced the osteogenesis in transwell setting and the transition from M1 to M2 was associated with an increase in bone anabolic factors CCL2/MCP-1 (Montrer CCL2 Anticorps), CCL5/RANTES and IGF-1 (Montrer IGF1 Anticorps) in vitro.

3. Taken together, our data suggest that CCR5 regulates insulin (Montrer INS Anticorps) signaling in hypothalamus which contributes to systemic insulin (Montrer INS Anticorps) sensitivity and glucose metabolism.

4. identifies the essential role of the chemoattractive cytokine CCL5 for liver disease progression and especially hepatocellular carcinoma development

5. Breast cancer cell CCL5 mediates bone marrow independent angiogenesis via paracrine signaling.

6. Studied the effects of CCL5-CCR5 interactions in breast cancer metabolism, and findings suggest that CCL5-CCR5 interactions in the tumor microenvironment modulate metabolic events during tumor onset to promote tumorigenesis.

7. RANTES levels were associated with TMS (Montrer PYCARD Anticorps) measures of cortical synaptic excitability, but not with long-term potentiation (LTP (Montrer SCP2 Anticorps))-like plasticity.

8. study found that the inflammatory chemokine (Montrer CCL1 Anticorps) CCL5 is mostly retained (75%) during the resolution of zymosan A peritonitis in mice; CCL5 exerts a novel proresolving role on macrophages when acting in concert with apoptotic PMN (Montrer TBCE Anticorps)-expressed D6.

9. A robust up-regulation of RANTES within the brain was seen in a mouse model of tick-borne encephalitis.

10. CCL5 paradoxically limits macrophage accumulation in the injured kidney during renin (Montrer REN Anticorps) angiotensin system (RAS) activation by constraining the proinflammatory actions of CCL2 (Montrer CCL2 Anticorps).

Pig (Porcine) Chemokine (C-C Motif) Ligand 5 (CCL5) interaction partners

1. These results represent an important molecular mechanism whereby H. parasuis induced RANTES in the inflammatory response.

2. Foot-and-mouth disease virus leader proteinase inhibits dsRNA-induced RANTES transcription.

3. we cloned the nucleotide sequence of the 5'-flanking region of the porcine RANTES (poRANTES) gene and characterized the regulatory elements that activate transcription

4. The Toll (Montrer TLR4 Anticorps)-like receptor signaling pathway is involved in porcine reproductive and respiratory syndrome virus-induced RANTES activation.

5. These results suggest that porcine RANTES can play an important role in xenotransplant rejection, through participating in the interaction between porcine endothelial cells and human monocytes.

Cow (Bovine) Chemokine (C-C Motif) Ligand 5 (CCL5) interaction partners

1. CCL5 but not CCL2 (Montrer CCL2 Anticorps) mainly attract bovine classical monocytes and promote their differentiation into LPS (Montrer IRF6 Anticorps)-hypo-responsive macrophages.

2. decrease in sensitivity of HIV variants to RANTES neutralization during the course of progressive infection, but not during follow-up of long term survivors; data suggest a role for RANTES neutralization sensitivity of HIV-1 in AIDS pathogenesis

Guinea Pig Chemokine (C-C Motif) Ligand 5 (CCL5) interaction partners

1. In the pathogenesis of allergic rhinitis, substance P (Montrer TAC1 Anticorps) induces expression of RANTES in nasal mucosa.

Rhesus Monkey Chemokine (C-C Motif) Ligand 5 (CCL5) interaction partners

1. CCL3 (Montrer CCL3 Anticorps), CCL4 (Montrer CCL4 Anticorps) and CCL5 gene expression was evaluated in response to simian-human immunodeficiency virus (SHIV) infection in a rhesus macaque model.