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anti-Human GRIP1 Anticorps:
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Human Polyclonal GRIP1 Primary Antibody pour WB - ABIN1742325
Van Damme, Bogaert, Dewil, Hersmus, Kiraly, Scheveneels, Bockx, Braeken, Verpoorten, Verhoeven, Timmerman, Herijgers, Callewaert, Carmeliet, Van Den Bosch, Robberecht: Astrocytes regulate GluR2 expression in motor neurons and their vulnerability to excitotoxicity. dans Proceedings of the National Academy of Sciences of the United States of America 2007
Dog (Canine) Polyclonal GRIP1 Primary Antibody pour WB - ABIN2779681
Sugatani, Nishitani, Yamakawa, Yoshinari, Sueyoshi, Negishi, Miwa: Transcriptional regulation of human UGT1A1 gene expression: activated glucocorticoid receptor enhances constitutive androstane receptor/pregnane X receptor-mediated UDP-glucuronosyltransferase 1A1 regulation with glucocorticoid receptor-interacting protei dans Molecular pharmacology 2005
GRIP1 is phosphorylated at an N-terminal serine cluster by cyclin-dependent kinase-9 (CDK9), which is recruited into GC-induced GR:GRIP1:CDK9 hetero-complexes, producing distinct GRE-specific GRIP1 phospho-isoforms.
Data suggest that molecules in the erythropoietin-producing hepatocellular receptor B family (EPHB) / ephrinB (EFNB) signalling pathways, specifically ephrin B3 and GRIP1, are involved blood pressure regulation.
GRIP1 was identified as one of the most important differentially expressed, topologically significant proteins in the protein-protein interaction network in Alzheimer's disease.
the Trip6-GRIP1-myosin VI interaction and its regulation on F-actin network play a significant role in dendritic morphogenesis
In 15 of 590 families, we identified recessive mutations in the genes FRAS1, FREM2, GRIP1, FREM1, ITGA8, and GREM1, all of which function in the interaction of the ureteric bud and the metanephric mesenchyme.
In three unrelated families with parental consanguinity, GRIP1 mutations were found to segregate with Fraser syndrome in an autosomal recessive manner.
Gain-of-function glutamate receptor interacting protein 1 variants alter GluA2 recycling and surface distribution in patients with autism
GRIP1 splice forms interact with gephyrin and play a role in synaptic function at GABAergic and glycinergic synapses in cultured hippocampal neurons.
after regulated endocytosis, binding to GRIP/ABP stabilizes the internalized receptors in an intracellular pool and prevents them from being recycled back to the plasma membrane or entering a degradative pathway.
GRIP1c 4-7 plays a role not only in glutamatergic synapses but also in GABAergic synapses
study showed neither single marker nor haplotype analysis revealed an association between variants at GRIP1 locus & schizophrenia; suggests it is unlikely that the GRIP1 polymorphisms investigated play a substantial role in schizophrenia susceptibility
Supramodular nature of GRIP1 revealed by the structure of its PDZ12 tandem in complex with the carboxyl tail of Fras1.
GRIP1 is a critical regulator of immunometabolism, which engages distinct transcriptional mechanisms to coordinate the balance between macrophage populations and ultimately promote metabolic homeostasis.
GRIP1 binds to apoER2 and EphrinB2 to induce activity-dependent AMPA receptor insertion at the synapse.
Study performed social behavioral testing on neuron-specific Grip1/2-double knockout (DKO) and wild type (WT) mice and determined the expression profile of key signaling proteins in AMPAR, mGluR, mTOR, and GABA pathways in brain. Data Grip1/2-mediated AMPA signaling in regulating social behaviors and disturbance of glutamate- and GABA-signaling in specialized brain regions in autism-related social behavioral deficits.
subcellular redistribution of GRIP1 and a change in the binding of GRIP1 to GluA2 during synaptic scaling, was observed.
This study demonistrated that deletion of accumbal GRIP1 increases vulnerability to cue-induced cocaine relapse without altering the reinforcing properties of the drug or the response to natural rewards
a regulatory role for GRIP1 during microtubule-based transport and suggest a crucial function for 14-3-3 proteins in controlling kinesin-1 motor attachment during neuronal development
Data indicate that glucocorticoid receptor (GR):GRIP1 use distinct mechanisms to repress inflammatory genes at different stages of the transcription cycle.
GRIP1 and 2 regulate activity-dependent AMPA receptor recycling via exocyst complex interactions.
GRIP1 scaffolding protein is required for the formation and integrity of the dermo-epidermal junction and reveal the importance of PDZ domains in the organization of supramolecular structures essential for mammalian embryonic development.
Glutamate-receptor-interacting protein GRIP1 directly steers kinesin to dendrites.
morphology of octopus cell (OC) somata in the cochlear nucleus B6 and B6Cast mice, and the immunocytochemistry of GluR1 (glutamate receptor subunit 1) and GRIP-C (glutamate receptor interacting protein-C terminus).
investigated whether there is a link between growth inhibitory effect and transcriptional activation of PPARgamma in several gastrointestinal tumour cell lines [GRIP1tau]
These results indicate that the GRIP family comprises an essential molecular component for cerebellar LTD.
An isoform of Grip1 interacts with the N-terminus of Dlx2.
This gene encodes a member of the glutamate receptor interacting protein family. The encoded scaffold protein binds to and mediates the trafficking and membrane organization of a number of transmembrane proteins. Alternatively spliced transcript variants encoding different isoforms have been described.
glutamate receptor-interacting protein 1
, eye blebs
, AMPA receptor-interacting protein GRIP1
, glutamate receptor interacting protein 1
, glutamate receptor-interacting protein 1-like