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Insulin Kit ELISA

Ce kit ELISA Colorimetric est conçu pour la mesure quantitative de Humain Insulin. Il y a 2 publications disponibles.
N° du produit ABIN1326860

Aperçu rapide pour Insulin Kit ELISA (ABIN1326860)

Antigène

Voir toutes Insulin (INS) Kits ELISA
Insulin (INS)

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Humain

Méthode de détection

Colorimetric

Type de méthode

Sandwich ELISA

Application

ELISA
  • Analytical Method

    Quantitative
  • Plaque

    Pre-coated

    Restrictions

    For Research Use only
  • Stock

    4 °C
  • Bakry, Shoeib, El Shafiee, Hassan: "Androgenetic alopecia, metabolic syndrome, and insulin resistance: Is there any association? A case-control study." dans: Indian dermatology online journal, Vol. 5, Issue 3, pp. 276-81, (2014) (PubMed).

    Sui, Yi, Yao, Liang, Sun, Hu, Uc, Nelson, Ode, Philipson, Engelhardt, Norris: "Quantifying insulin sensitivity and entero-insular responsiveness to hyper- and hypoglycemia in ferrets." dans: PLoS ONE, Vol. 9, Issue 3, pp. e90519, (2014) (PubMed).

  • Antigène Voir toutes Insulin (INS) Kits ELISA

    Insulin (INS)

    Autre désignation

    Insulin

    Sujet

    Insulin is the principal hormone responsible for the control of glucose metabolism. It is synthesized in the _-cells of the islets of Langerhans as the precursor, proinsulin, which is processed to form C-peptide and insulin. Both are secreted in equimolar amounts into the portal circulation. The mature insulin molecule comprises two polypeptide chains, the A chain and B chain (21 and 30 amino acids respectively). The two chains are linked together by two inter-chain disulphide bridges. There is also an intra-chain disulphide bridge in the A chain. Insulin concentrations are severely reduced in insulin-dependent diabetes mellitus (IDDM) and some other conditions such as hypopituitarism. Insulin levels are raised in non-insulin-dependent diabetes mellitus (NIDDM), obesity, insulinoma and some endocrine dysfunctions such as Cushing

    Pathways

    Signalisation NF-kappaB, Signalisation RTK, Positive Regulation of Peptide Hormone Secretion, Peptide Hormone Metabolism, Hormone Activity, Carbohydrate Homeostasis, ER-Nucleus Signaling, Regulation of Carbohydrate Metabolic Process, Feeding Behaviour, Autophagy, Negative Regulation of intrinsic apoptotic Signaling, Brown Fat Cell Differentiation, Positive Regulation of fat Cell Differentiation
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