SMNDC1 Protein (Myc-DYKDDDDK Tag)

N° du produit ABIN2732252

Détail du produit

Antigène: SMNDC1 (Survival Motor Neuron Domain Containing 1 (SMNDC1))

Type de proteíne: Recombinant

Origine: Humain

Source: HEK-293 Cells

Purification/Conjugué: Cette SMNDC1 protéine est marqué à la Myc-DYKDDDDK Tag.

Application: Antibody Production (AbP), Standard (STD)

Attributs du produit:

• Recombinant human SMNDC1 protein expressed in HEK293 cells.
• Produced with end-sequenced ORF clone

Pureté: > 80 % as determined by SDS-PAGE and Coomassie blue staining

Information d'application

Indications d'application: Recombinant human proteins can be used for:
Native antigens for optimized antibody production
Positive controls in ELISA and other antibody assays

Commentaires:

The tag is located at the C-terminal.

Restrictions: For Research Use only

Stockage

Concentration: 50 μg/mL

Buffer: 25 mM Tris.HCl, pH 7.3, 100 mM glycine, 10 % glycerol.

Stock: -80 °C

Stockage commentaire: Store at -80°C. Thaw on ice, aliquot to individual single-use tubes, and then re-freeze immediately. Only 2-3 freeze thaw cycles are recommended.

Détail du antigène

Antigène: SMNDC1 (Survival Motor Neuron Domain Containing 1 (SMNDC1))

Autre désignation: Smndc1 (SMNDC1 Produits)

Synonymes: smnr Protein, spf30 Protein, SMNDC1 Protein, SMNR Protein, SPF30 Protein, TDRD16C Protein, wu:fb37h07 Protein, wu:fc23a07 Protein, 2410004J23Rik Protein, 4933440I19Rik Protein, survival motor neuron domain containing 1 Protein, smndc1 Protein, SMNDC1 Protein, Bm1_41545 Protein, Smndc1 Protein

Sujet: This gene is a paralog of SMN1 gene, which encodes the survival motor neuron protein, mutations in which are cause of autosomal recessive proximal spinal muscular atrophy. The protein encoded by this gene is a nuclear protein that has been identified as a constituent of the spliceosome complex. This gene is differentially expressed, with abundant levels in skeletal muscle, and may share similar cellular function as the SMN1 gene.

Poids moléculaire: 26.5 kDa

NCBI Accession: NP_005862