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Aperçu des produits pour anti-ATP5J (ATP5J) Anticorps

Full name:: anti-ATP Synthase, H+ Transporting, Mitochondrial F0 Complex, Subunit F6 Anticorps (ATP5J)

Chez www.anticorps-enligne.fr sont 70 ATP Synthase, H+ Transporting, Mitochondrial F0 Complex, Subunit F6 (ATP5J) Anticorps de 15 de différents fournisseurs disponibles. De plus, nous expédions ATP5J Protéines (15) et ATP5J Kits (14) et beaucoup plus de produits pour cette protéine. Un total de 99 ATP5J produits sont actuellement listés.

Synonymes:: ATP5, ATP5A, ATPM, CF6, F6, wu:fa66e04, zgc:77541

afficher tous les anticorps	Gène	GeneID	UniProt
	ATP5J	11957	P97450
	ATP5J	522	P18859
	ATP5J	94271	P21571
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anti-ATP5J Anticorps mieux référencés

Human Polyclonal ATP5J Primary Antibody pour ICC, IF - ABIN4282288 : Uhlén, Oksvold, Älgenäs, Hamsten, Fagerberg, Klevebring, Lundberg, Odeberg, Pontén, Kondo, Sivertsson: Antibody-based protein profiling of the human chromosome 21. dans Molecular & cellular proteomics : MCP 2012 (PubMed)

Plus d’anticorps contre ATP5J partenaires d’interaction

Mouse (Murine) ATP Synthase, H+ Transporting, Mitochondrial F0 Complex, Subunit F6 (ATP5J) interaction partners

Coupling factor 6 overexpression induced salt-sensitive hypertension, complicated by systolic cardiac dysfunction, but its onset was delayed in females. Estrogen has an important role in the regulation of coupling factor 6-mediated pathophysiology.
Overexpression of coupling factor 6 attenuates exercise-induced physiological cardiac hypertrophy by downregulating Akt signaling, thereby cancelling its benefit for cardiac function in mice.
CF6 plays a crucial role in the development of insulin resistance and hypertension
coupling factor 6 induces the development of systolic dysfunction and upregulation of nicotinamide adenine dinucleotide phosphate oxidase in the heart under the high-salt diet
identification of functional elements in bi-directional promoter

Human ATP Synthase, H+ Transporting, Mitochondrial F0 Complex, Subunit F6 (ATP5J) interaction partners

AKT2 and XIST expression was identified as a potential biomarker participating in the effect of ATP5J in colorectal cancer
CF6-induced increase in apoptotic cells was blocked by immature or mature IF1, being accompanied by protein kinase B (PKB) phosphorylation. IF1 antagonizes the pro-apoptotic action of CF6 by relief of intracellular acidification and resultant phosphorylation of PKB.
Over-expression of the ATP5J gene correlates with cell migration and 5-fluorouracil sensitivity in colorectal cancer.
CF6 plays a crucial role in the development of insulin resistance and hypertension
The phenotypic range of retinal, peripheral and central nervous system disease expression is characterized in a single family with NARP syndrome from the ATPase 6 m.8993T>C mtDNA point mutation.
coupling factor 6 induces the development of systolic dysfunction and upregulation of nicotinamide adenine dinucleotide phosphate oxidase in the heart under the high-salt diet
CF6 is a novel risk factor for ischemic heart disease in end-stage renal disease. Synergism of this peptide and asymmetric dimethylarginine might contribute to its occurrence presumably by inhibition of prostacyclin and nitric oxide production.
Mutation analysis revealed the T9176C mutation in the mtATPase 6 gene (OMIM 516060) and the mutation load was above 90% in the patients with Leigh syndrome.
Plasma CF6 elevated in patients with acute myocardial infarction. At 3 days, plasma CF6 correlated positively with plasma creatinine kinase peak value and correlated negatively with left ventricular ejection fraction.
Membrane-bound ATP synthase functions as a receptor for CF6 and may have a previously unsuspected role in the genesis of hypertension by modulating the concentration of intracellular hydrogen.
T8993G allele causes severe extrapyramidal dysfunction and Leigh disease but there is no correlation between the degree of enzyme deficiency and the severity of the phenotype.
Increased CF6 may be responsible in part for decreased prostacyclin observed in coronary heart disease, in particular after PTCA and stent therapy. Potential risk factor for coronary heart disease.
in vascular endothelial cells both CF6 (coupling factor 6) and Angiotensin II downregulate PECAM-1 (platelet/endothelial cell adhesion molecule) expression via activation of c-Src kinase
Coupling factor 6 enhances Src-mediated responsiveness to angiotensin II in resistance arterioles and cells.

ATP5J profil antigène

Antigen Summary

Mitochondrial ATP synthase catalyzes ATP synthesis, utilizing an electrochemical gradient of protons across the inner membrane during oxidative phosphorylation. It is composed of two linked multi-subunit complexes: the soluble catalytic core, F1, and the membrane-spanning component, Fo, which comprises the proton channel. The F1 complex consists of 5 different subunits (alpha, beta, gamma, delta, and epsilon) assembled in a ratio of 3 alpha, 3 beta, and a single representative of the other 3. The Fo seems to have nine subunits (a, b, c, d, e, f, g, F6 and 8). This gene encodes the F6 subunit of the Fo complex, required for F1 and Fo interactions. Alternatively spliced transcript variants encoding different isoforms have been identified for this gene. A pseudogene exists on chromosome Yp11.

Alternative names and synonyms associated with ATP5J

ATP synthase, H+ transporting, mitochondrial Fo complex, subunit F6 (atp5j) anticorps
ATP synthase, H+ transporting, mitochondrial Fo complex subunit F6 (ATP5J) anticorps
ATP synthase, H+ transporting, mitochondrial Fo complex subunit F6 L homeolog (atp5j.L) anticorps
ATP synthase, H+ transporting, mitochondrial Fo complex subunit F6 (atp5j) anticorps
ATP synthase, H+ transporting, mitochondrial F0 complex, subunit F6 (Atp5j) anticorps
ATP synthase, H+ transporting, mitochondrial F0 complex, subunit F (Atp5j) anticorps
ATP synthase, H+ transporting, mitochondrial Fo complex, subunit F6 (Atp5j) anticorps

ATP5 anticorps
ATP5A anticorps
ATPM anticorps
CF6 anticorps
F6 anticorps
wu:fa66e04 anticorps
zgc:77541 anticorps

Protein level used designations for ATP5J

ATP synthase-coupling factor 6, mitochondrial , ATP synthase, H+ transporting, mitochondrial F0 complex, subunit F6 , OXPHOS complex V coupling factor 6 , ATP synthase coupling factor VI , ATPase subunit F6 , mitochondrial ATP synthase coupling factor 6 , coupling factor 6 , mitochondrial ATP synthase, coupling factor 6 , mitochondrial ATP synthase, subunit F6 , mitochondrial ATPase coupling factor 6 , proliferation-inducing protein 36

GENE ID	SPECIES
406599	Danio rerio
418477	Gallus gallus
708411	Macaca mulatta
744512	Pan troglodytes
379616	Xenopus laevis
549796	Xenopus (Silurana) tropicalis
100113544	Nasonia vitripennis
11957	Mus musculus
522	Homo sapiens
94271	Rattus norvegicus
478393	Canis lupus familiaris
282690	Bos taurus
100172310	Pongo abelii

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Show all anti-ATP Synthase, H+ Transporting, Mitochondrial F0 Complex, Subunit F6 (ATP5J) Anticorps with Pubmed References

Mouse (Murine) ATP Synthase, H+ Transporting, Mitochondrial F0 Complex, Subunit F6 (ATP5J) interaction partners

Human ATP Synthase, H+ Transporting, Mitochondrial F0 Complex, Subunit F6 (ATP5J) interaction partners

Antigen Summary

Alternative names and synonyms associated with ATP5J

Protein level used designations for ATP5J