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anti-Human DVL1 Anticorps:
anti-Mouse (Murine) DVL1 Anticorps:
anti-Rat (Rattus) DVL1 Anticorps:
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Pig (Porcine) DVL1 Primary Antibody pour ELISA, WB - ABIN1782135
Spate, Brown, Redel, Whitworth, Murphy, Prather: Dickkopf-related protein 1 inhibits the WNT signaling pathway and improves pig oocyte maturation. dans PLoS ONE 2014
Human Polyclonal DVL1 Primary Antibody pour ELISA, WB - ABIN560673
Turashvili, Bouchal, Baumforth, Wei, Dziechciarkova, Ehrmann, Klein, Fridman, Skarda, Srovnal, Hajduch, Murray, Kolar: Novel markers for differentiation of lobular and ductal invasive breast carcinomas by laser microdissection and microarray analysis. dans BMC cancer 2007
Human Polyclonal DVL1 Primary Antibody pour IF (p), IHC (p) - ABIN670671
Hua, Xu, He, Jiang, Ye, Pan: Wnt4/?-catenin signaling pathway modulates balloon-injured carotid artery restenosis via disheveled-1. dans International journal of clinical and experimental pathology 2015
Human Polyclonal DVL1 Primary Antibody pour IHC (p), WB - ABIN4268104
Jung, Lee, Kim, Dhong, Cho, Roh: Role of Wnt signaling pathway in progression of sinonasal inverted papilloma to squamous cell carcinoma. dans American journal of rhinology & allergy 2015
Lack of DVL (Montrer DVL2 Anticorps) prevents NEK2 (Montrer NEK2 Anticorps)-controlled dissolution of loose centrosomal linker and subsequent centrosomal separation. Increased DVL (Montrer DVL2 Anticorps) levels, in contrast, sequester centrosomal NEK2 (Montrer NEK2 Anticorps) and mimic monopolar spindle defects induced by a dominant negative version of this kinase.
These results identify USP4 (Montrer USP4 Anticorps) as a novel regulator of Dvl (Montrer DVL2 Anticorps) in Wnt (Montrer WNT2 Anticorps)/beta-catenin (Montrer CTNNB1 Anticorps) signal and show its involvement in Wnt3a (Montrer WNT3A Anticorps)-induced osteoblast differentiation
Authors conclude that the planar cell polarity (PCP (Montrer PRCP Anticorps)) pathway contributes significantly to the motility and hence the invasiveness of glioblastoma multiforme (GBM) cells, and that Nrdp1 (Montrer RNF41 Anticorps) acts as a negative regulator of PCP (Montrer PRCP Anticorps) signaling by inhibiting Dvl (Montrer DVL2 Anticorps) through a novel polyubiquitination mechanism.
The DEP (Montrer ZDHHC21 Anticorps) domain of Dishevelled (Montrer DVL2 Anticorps) undergoes a conformational switch, from monomeric to swapped dimer, to trigger DIX domain-dependent polymerization and signaling to beta-catenin (Montrer CTNNB1 Anticorps).
two mutually exclusive functions of the DEP (Montrer ZDHHC21 Anticorps) domain in Wnt (Montrer WNT2 Anticorps) signal transduction - binding to Frizzled to recruit Dishevelled (Montrer DVL2 Anticorps) to the receptor complex, is reported.
we show that Wnt5a (Montrer WNT5A Anticorps) rapidly represses rDNA gene transcription in breast cancer cells and generates a chromatin state with reduced transcription of rDNA by RNA polymerase I (Montrer POLR1C Anticorps) (Pol I). These effects were specifically dependent on Dishevelled1 (DVL1), which accumulates in nucleolar organizer regions (NORs) and binds to rDNA regions of the chromosome.
Data show that dishevelled (Montrer DVL2 Anticorps) proteins DVL1, 2 and 3 were exclusively expressed in chronic lymphatic leukaemia (CLL) cells as compared to normal peripheral blood mononuclear cells (PBMCs).
The secreted frizzled-related protein (Montrer SFRP2 Anticorps) and disheveled protein family members appear to be actively involved in the pathogenesis of primary testicular germ cell tumors.
Data show that receptor for activated C kinase 1 (RACK1 (Montrer GNB2L1 Anticorps)) interacts with Dishevelled (Dvl (Montrer DVL2 Anticorps)) proteins and promotes their lysosomal degradation, and this effect is enhanced by autophagy induction.
TMEM88 stimulated triple negative breast cancer cell invasion by interacting with DVLl.
Data show that the peptide binding pocket of the Dishevelled 1 (Dvl1) PDZ domain (Montrer INADL Anticorps) is stabilized upon CXXC finger 5 protein (CXXC5 (Montrer CXXC5 Anticorps)) binding.
Here we show that the peptide-binding pocket of the Dvl PDZ domain (Montrer INADL Anticorps) can be occupied by Dvl's own highly conserved C-terminus, inducing a closed conformation.
The interaction of Dvl1 with Syt-1 (Montrer SYT1 Anticorps), which is regulated by Wnts, modulates neurotransmitter release.
findings indicate that Dvl-1 may be an underlying participant of oxidative stress induced (Montrer SQSTM1 Anticorps) by selenium deficiency
These results support a novel cell-autonomous postsynaptic role for Dact1 (Montrer DACT1 Anticorps), in cooperation with Dishevelled-1 and possibly Disrupted in Schizophrenia-1 (Montrer DISC1 Anticorps), in the formation of synapses on cortical interneuron dendrites.
we show that Dvl-1 expression is restricted to OSNs in the dorsal recess of the nasal cavity, and labels a unique subpopulation of glomeruli. Dvl-2 (Montrer DVL2 Anticorps) and Dvl-3 (Montrer DVL3 Anticorps) have a widespread distribution in both the olfactory epithelium and olfactory bulb .
Fuzzy appears to control subcellular localization of the core PCP (Montrer BMP1 Anticorps) protein Dishevelled (Montrer DVL2 Anticorps), recruiting it to Rab8 (Montrer RAB8A Anticorps)-positive vesicles and to the basal body and cilium. We show that loss of Fuzzy results in inhibition of PCP (Montrer BMP1 Anticorps) signaling
A Wnt5a (Montrer WNT5A Anticorps)--> Dvl PCP (Montrer BMP1 Anticorps) signaling cascade may regulate actin polymerization and protrusive cell behavior in the caudal (Montrer CAD Anticorps) splanchnic mesoderm to promote second heart field deployment, outflow tract lengthening and cardiac looping.
Findings indicate that Daple (Montrer CCDC88C Anticorps) interacts with Dishevelled (Montrer DVL2 Anticorps) to direct the Dishevelled (Montrer DVL2 Anticorps)/protein kinase (Montrer CDK7 Anticorps) lambda protein complex to activate Rac (Montrer AKT1 Anticorps), which in turn mediates the non-canonical Wnt (Montrer WNT2 Anticorps) signalling pathway required for cell migration.
The antagonistic functions of Vangl2 (Montrer VANGL2 Anticorps) and Dvl1 allow sharpening of planar cell polarity signaling locally on the tips of the filopodia to sense directional cues, Wnts, eventually causing turning of growth cones.
Different Dvl (Montrer DVL2 Anticorps) proteins (Dvl1, Dvl2 (Montrer DVL2 Anticorps), Dvl3 (Montrer DVL3 Anticorps)) and the composition of dishevelled (Montrer DVL2 Anticorps)-beta-arrestin protein complexes contribute to the specific activation of individual branches of Wnt (Montrer WNT2 Anticorps) signaling in Xenopus gastrulation.
DVL1, the human homolog of the Drosophila dishevelled gene (dsh) encodes a cytoplasmic phosphoprotein that regulates cell proliferation, acting as a transducer molecule for developmental processes, including segmentation and neuroblast specification. DVL1 is a candidate gene for neuroblastomatous transformation. The Schwartz-Jampel syndrome and Charcot-Marie-Tooth disease type 2A have been mapped to the same region as DVL1. The phenotypes of these diseases may be consistent with defects which might be expected from aberrant expression of a DVL gene during development.
DSH homolog 1
, dishevelled 1 (homologous to Drosophila dsh)
, dishevelled, dsh homolog 1
, segment polarity protein dishevelled homolog DVL-1
, dishevelled 1
, dishevelled, dsh homolog 2, like