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RASD1 encodes a member of the Ras superfamily of small GTPases and is induced by dexamethasone. De plus, nous expédions RASD1 Protéines (7) et RASD1 Kits (6) et beaucoup plus de produits pour cette protéine.
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Studies indicate potential roles for Rasd1 small G protein (Montrer RAC2 Anticorps) and leptin (Montrer LEP Anticorps) in TRPC4 (Montrer TRPC4 Anticorps) cation channel (Montrer TRPV1 Anticorps) activation.
Data indicate that protein kinase A (PKA) phosphorylates only serine-253 amino acid on activator of G-protein signaling 1 protein Dexras1 (RASD1).
Loss of RASD1 is associated with prostate cancer.
Study showed that Rasd1 and NonO (Montrer NONO Anticorps) interact at the CRE-site of specific target genes.
Rasd1 modulates endogenous renin (Montrer REN Anticorps) gene expression by interacting with Ear2 (Montrer NR2F6 Anticorps).
A glucocorticoid response element (GRE) was identified in the 3'-flanking region (2.3 kb downstream of poly(A) signal) of the human Dexras1 gene. A point mutation within the 15-bp GRE abolished this glucocorticoid responsiveness
yeast two hybrid analysis and co-immunoprecipitation studies in mammalian cells demonstrate a direct interaction between AGS1 (Montrer TREX1 Anticorps) and the G(beta1) subunit of heterotrimeric G proteins
does not alter the membrane translocation of protein kinase C delta (Montrer PKCd Anticorps).
Dexras1 plays a critical role in shaping the photic phase response curve (PRC (Montrer PPRC1 Anticorps)) and the signaling events through which it regulates clock entrainment in transgenic mice.
dex-ras1 mRNA is not induced by glucocorticoid stimulation in HEK293 cells
This study demonstrated that provide unprecedented evidence for a selective epigenetic up-regulation of Dexras 1 gene expression in the striatum of EAE mice.
RASD1 is a novel factor in Metaphase I-Metaphase II oocyte transition and may be involved in regulating the progression of cytokinesis and spindle formation
Results suggest a potential role for Dexras1 in modulating a selective subset of psychiatric symptoms, possibly via its interaction with NMDARs and/or other disease-related binding-partners
two mechanisms of Rasd1 induction in the hypothalamus, were identified.
Lysosomal iron modulates NMDA receptor-mediated excitation via small GTPase (Montrer RACGAP1 Anticorps), Dexras1
Dexras1 mediates adipogenesis and diet-induced obesity.
Dexras1 is involved in the circadian clock regulation
Dexras1 appears to mediate N-methylaspartate (NMDA)-elicited neurotoxicity via nitric oxide and iron influx.
These results indicate that the absence of Dexras1 sensitizes the suprachiasmatic nucleus to perturbations resulting from restricted feeding.
Data suggest that Dexras1 regulates multiple photic and nonphotic signal-transduction pathways, thereby playing an essential role modulating species-specific characteristics of circadian entrainment.
This gene encodes a member of the Ras superfamily of small GTPases and is induced by dexamethasone. The encoded protein is an activator of G-protein signaling and acts as a direct nucleotide exchange factor for Gi-Go proteins. This protein interacts with the neuronal nitric oxide adaptor protein CAPON, and a nuclear adaptor protein FE65, which interacts with the Alzheimer's disease amyloid precursor protein. This gene may play a role in dexamethasone-induced alterations in cell morphology, growth and cell-extracellular matrix interactions. Epigenetic inactivation of this gene is closely correlated with resistance to dexamethasone in multiple myeloma cells. Alternatively spliced transcript variants encoding different isoforms have been found for this gene.
activator of G-protein signaling 1
, dexamethasone-induced Ras-related protein 1
, ras-related protein
, DEXRAS1 (Dexras1)