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Aperçu des produits pour anti-DDIT4 (DDIT4) Anticorps

Full name:
anti-DNA-Damage-Inducible Transcript 4 Anticorps (DDIT4)
Chez www.anticorps-enligne.fr sont 89 DNA-Damage-Inducible Transcript 4 (DDIT4) Anticorps de 15 de différents fournisseurs disponibles. De plus, nous expédions DDIT4 Protéines (13) et DDIT4 Kits (1) et beaucoup plus de produits pour cette protéine. Un total de 113 DDIT4 produits sont actuellement listés.
Synonymes:
5830413E08Rik, AA415483, dig2, REDD-1, REDD1, rtp801, sb:cb821, zgc:64114

Réactivités les plus populaires pour anti-DDIT4 (DDIT4) Anticorps

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anti-Human DDIT4 Anticorps:

anti-Mouse (Murine) DDIT4 Anticorps:

anti-Rat (Rattus) DDIT4 Anticorps:

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anti-DDIT4 Anticorps mieux référencés

  1. Human Polyclonal DDIT4 Primary Antibody pour IHC, ELISA - ABIN1003101 : Ellisen, Ramsayer, Johannessen, Yang, Beppu, Minda, Oliner, McKeon, Haber: REDD1, a developmentally regulated transcriptional target of p63 and p53, links p63 to regulation of reactive oxygen species. dans Molecular cell 2002 (PubMed)
    Show all 4 references for ABIN1003101

  2. Human Polyclonal DDIT4 Primary Antibody pour ELISA, WB - ABIN1003102 : Shoshani, Faerman, Mett, Zelin, Tenne, Gorodin, Moshel, Elbaz, Budanov, Chajut, Kalinski, Kamer, Rozen, Mor, Keshet, Leshkowitz, Einat, Skaliter, Feinstein: Identification of a novel hypoxia-inducible factor 1-responsive gene, RTP801, involved in apoptosis. dans Molecular and cellular biology 2002 (PubMed)
    Show all 4 references for ABIN1003102

  3. Human Polyclonal DDIT4 Primary Antibody pour ICC, IF - ABIN4304577 : Stadler, Rexhepaj, Singan, Murphy, Pepperkok, Uhlén, Simpson, Lundberg: Immunofluorescence and fluorescent-protein tagging show high correlation for protein localization in mammalian cells. dans Nature methods 2013 (PubMed)

  4. Human Polyclonal DDIT4 Primary Antibody pour ICC, IF - ABIN439647 : Potts, McMillan, Rosales, Kim, Ou, Toombs, Brekken, Minden, MacMillan, White: Mode of action and pharmacogenomic biomarkers for exceptional responders to didemnin B. dans Nature chemical biology 2015 (PubMed)

Plus d’anticorps contre DDIT4 partenaires d’interaction

Human DNA-Damage-Inducible Transcript 4 (DDIT4) interaction partners

  1. The production of superoxide anion in nockout-Rtp801 mouse lung fibroblasts (MLF) was lower than that in Rtp801 Wt cells after cigarette smoke extract treatment, and it was inhibited in Wt MLF by silencing nicotinamide adenine dinucleotide phosphate oxidase (Montrer DUOX2 Anticorps)-4 (Nox4 (Montrer NOX4 Anticorps)) expression with small interfering Nox4 (Montrer NOX4 Anticorps) RNA.

  2. Changes in REDD1 mRNA and protein have been observed in skeletal muscle under various physiological conditions (e.g., nutrient consumption and resistance exercise) and pathological conditions (e.g., sepsis, alcoholism, diabetes, obesity) suggesting a role for REDD1 in regulating mTORC1-dependent skeletal muscle protein metabolism. [Review]

  3. a novel STAT3 (Montrer STAT3 Anticorps)-dependent mechanism of both IL-6 (Montrer IL6 Anticorps)-induced activation of mTOR (Montrer FRAP1 Anticorps) and IL-6 (Montrer IL6 Anticorps)-dependent reversion of stress-induced inhibition of mTOR (Montrer FRAP1 Anticorps) activity, is reported.

  4. findings implicate REDD1 as a crucial regulator of mTORC1 activity in iron-depleted cells

  5. C/EBPbeta (Montrer CEBPB Anticorps) promotes autophagy in PC3 (Montrer PCSK1 Anticorps) cells by augmenting REDD1 expression.

  6. These data highlight the central role of REDD1 in regulating both protein synthesis and autophagy in skeletal muscle during sepsis.

  7. Findings from this study propose a REDD1-regulated mechanism in T2D skeletal muscle that may contribute to whole body insulin (Montrer INS Anticorps) resistance and may be a target to improve insulin (Montrer INS Anticorps) action in insulin (Montrer INS Anticorps)-resistant individuals.

  8. REDD1 knockout (KO) mice, all skin compartments, epidermal stem, and progenitor cells were protected from atrophic effects of glucocorticoids.

  9. MiR (Montrer MLXIP Anticorps)-630 reduced apoptosis by downregulating several apoptotic modulators, PARP3 (Montrer PARP3 Anticorps), DDIT4, and EP300 (Montrer EP300 Anticorps).

  10. REDD1 and p-AKT (Montrer AKT1 Anticorps) over-expression may serve as a prognostic biomarker in ovarian cancer, but KRAS mutations and REDD1 protein over-expression were not correlated in OC.

Mouse (Murine) DNA-Damage-Inducible Transcript 4 (DDIT4) interaction partners

  1. These data suggest that loss of REDD1 augments the rate of the OV-induced increase in muscle mass by altering multiple protein balance pathways.

  2. The production of superoxide anion in nockout-Rtp801 mouse lung fibroblasts (MLF) was lower than that in Rtp801 Wt cells after cigarette smoke extract treatment, and it was inhibited in Wt MLF by silencing nicotinamide adenine dinucleotide phosphate oxidase (Montrer DUOX1 Anticorps)-4 (Nox4 (Montrer NOX4 Anticorps)) expression with small interfering Nox4 (Montrer NOX4 Anticorps) RNA.

  3. REDD1 is required for normal insulin (Montrer INS Anticorps)-stimulated signaling, and a subtle balance exists between MEK1 (Montrer MAP2K1 Anticorps)/2, REDD1, and mTOR (Montrer FRAP1 Anticorps)

  4. study suggests that VDR (Montrer CYP27B1 Anticorps) regulates Ddit4 expression during epidermal homeostasis and the wound healing process, while elevated Ddit4 represents an early growth-arresting stress response within VDR (Montrer CYP27B1 Anticorps)(-/-) follicles.

  5. REDD1 knockout T cells exhibit a defect in proliferation and cell survival, although markers of activation appear normal. These findings demonstrate a previously unappreciated role for REDD1 in T cell function.

  6. Reactive oxygen species regulation through REDD1/TXNIP (Montrer TXNIP Anticorps) is physiological rheostat controlling stress-induced autophagy.

  7. LPS (Montrer TLR4 Anticorps) induces REDD1 expression by two distinct CREB (Montrer CREB1 Anticorps)-mediated mechanisms

  8. Glucocorticoids induce skin atrophy and activate REDD1 expression.

  9. REDD1 expression limits the nutrient-induced stimulation of protein synthesis and activation of mTORC1 signaling during periods of feed deprivation.

  10. REDD1 is necessary for hyperglycemia mediated effects on VEGF expression in the retina of diabetic mice.

Zebrafish DNA-Damage-Inducible Transcript 4 (DDIT4) interaction partners

  1. Redd1 alters dorsoventral patterning by antagonizing the Wnt (Montrer WNT2 Anticorps)/beta-catenin (Montrer CTNNB1 Anticorps) signaling pathway.

DDIT4 profil antigène

Antigen Summary

HIF-1-responsive gene that may protect some types of cells from hypoxia and H(2)O(2)-triggered apoptosis

Alternative names and synonyms associated with DDIT4

  • DNA-damage-inducible transcript 4 (ddit4) anticorps
  • DNA-damage-inducible transcript 4 (DDIT4) anticorps
  • DNA-damage-inducible transcript 4 (Ddit4) anticorps
  • 5830413E08Rik anticorps
  • AA415483 anticorps
  • dig2 anticorps
  • REDD-1 anticorps
  • REDD1 anticorps
  • rtp801 anticorps
  • sb:cb821 anticorps
  • zgc:64114 anticorps

Protein level used designations for DDIT4

DNA damage-inducible transcript 4 protein , REDD-1 , DNA-damage-inducible transcript 4 protein , protein regulated in development and DNA damage response 1 , DNA-damage-inducible transcript 4 , HIF-1 responsive protein RTP801 , HIF-1 responsive RTP801 , dexamethasone-induced gene 2 protein , regulated in development and DNA damage response 1

GENE ID SPECIES
394782 Xenopus (Silurana) tropicalis
450520 Pan troglodytes
489038 Canis lupus familiaris
54541 Homo sapiens
74747 Mus musculus
140942 Rattus norvegicus
100153821 Sus scrofa
529235 Bos taurus
379481 Xenopus laevis
378866 Danio rerio
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