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The protein encoded by CEND1 is a neuron-specific protein. De plus, nous expédions Cell Cycle Exit and Neuronal Differentiation 1 Protéines (6) et beaucoup plus de produits pour cette protéine.
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The detection of ALT, AFP (Montrer AFP Anticorps), AFP (Montrer AFP Anticorps)-L3, and GP73 (Montrer GOLM1 Anticorps) has a certain guiding significance to predict the risk of hepatocellular carcinoma in hepatic cirrhosis patients
The mechanism of hepatocarcinogenesis and a promising blueprint for miR (Montrer MLXIP Anticorps)-493-5p-GP73 (Montrer GOLM1 Anticorps) axis-oriented treatment of HCC (Montrer FAM126A Anticorps).
GP73 (Montrer GOLM1 Anticorps) unglycosylation is associated with hepatocellular carcinoma cell motility and invasiveness.
The GOLPH2 (Montrer GOLM1 Anticorps) is a novel marker for non-small-cell lung cancer.
Findings suggest that GP73 (Montrer GOLM1 Anticorps) silencing through siRNA delivery may provide a low-toxicity therapy for the inhibition of tumor proliferation and metastasis.
Both gain- and loss-of-function studies determine that GOLM1 (Montrer GOLM1 Anticorps) acts as a key oncogene (Montrer RAB1A Anticorps) by promoting HCC (Montrer FAM126A Anticorps) growth and metastasis.
A cytokine QTL at the NAA35 (Montrer MAK10 Anticorps)-GOLM1 (Montrer GOLM1 Anticorps) locus markedly modulated interleukin (IL)-6 (Montrer IL6 Anticorps) production in response to multiple pathogens and was associated with susceptibility to candidemia.
knock-down of GP73 (Montrer GOLM1 Anticorps) down-regulates HCV infection and replication in Huh7-MAVSR cells and primary human hepatocytes
Studies indicate that golgi membrane protein 1 (GOLM1 (Montrer GOLM1 Anticorps)) may promote HCC (Montrer FAM126A Anticorps) by regulation of epidermal growth factor receptor (EGFR (Montrer EGFR Anticorps)) recycling, and suggest that therapeutic targeting of GOLM1 (Montrer GOLM1 Anticorps) may be a potential strategy for combating hepatocellular carcinoma (HCC (Montrer FAM126A Anticorps)) metastasis.
these studies revealed that GP73 (Montrer GOLM1 Anticorps) promotes hepatitis C virus (HCV) secretion by directly mediating the interaction of ApoE (Montrer APOE Anticorps) with HCV replication complex through binding with HCV NS5A.
data suggest that common reprogramming mechanisms exist driving the conversion of lineage-distant somatic cell types to neurons and reveal a critical role for CEND1 in NEUROG2 (Montrer NEUROG2 Anticorps)-driven astrocytic reprogramming.
results suggest that functional interactions between BM88/Cend1, RanBPM and Dyrk1B (Montrer DYRK1B Anticorps) affect the balance between cellular proliferation and differentiation in Neuro 2a cells
This study suggested that Cend1 is involved in Ahi1 (Montrer AHI1 Anticorps)-associated hypothalamic neuronal differentiation in early development, giving us fresh insight into the mechanism behind the delayed development in Joubert syndrome.
Our results suggest that Cend1 is required for normal cerebellar development.
Transplantation of BM88/Cend1-overexpressing neural progenitor cells exerts beneficial effects on tissue regeneration by enhancing the number of generated neurons and restricting the formation of astroglial scar, in a mouse model of cortical brain injury.
BM88 is widely expressed in the embryonic CNS and PNS, in both nestin (Montrer NES Anticorps)-positive neuroepithelial cells and post-mitotic betaIII-tubulin (Montrer TUBB3 Anticorps) positive neurons.
Results implicate BM88 in the synchronization of cell cycle exit and differentiation of neuronal precursors in the developing nervous system.
BM88/Cend1 participates in cell cycle control and neuronal differentiation mechanisms during neonatal SVZ neurogenesis and becomes crucial for the transition from neuroblasts to mature neurons when reaching high levels
BM88 plays an essential role in regulating cell cycle exit and differentiation of Neuro 2a cells toward a neuronal phenotype
The protein encoded by this gene is a neuron-specific protein. The similar protein in pig enhances neuroblastoma cell differentiation in vitro and may be involved in neuronal differentiation in vivo. Multiple pseudogenes have been reported for this gene.
, cell cycle exit and neuronal differentiation protein 1
, cell cycle exit and neuronal differentiation 1