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The Fanconi anemia complementation group (FANC) currently includes FANCA, FANCB, FANCC, FANCD1 (also called BRCA2), FANCD2, FANCE, FANCF, FANCG, FANCI, FANCJ (also called BRIP1), FANCL, FANCM and FANCN (also called PALB2). De plus, nous expédions Fanconi Anemia, Complementation Group C Protéines (6) et et beaucoup plus de produits pour cette protéine.
Showing 10 out of 113 products:
Human Polyclonal FANCC Primary Antibody pour WB - ABIN251003
Yung, Tilgner, Ledran, Habibollah, Neganova, Singhapol, Saretzki, Stojkovic, Armstrong, Przyborski, Lako: Brief report: human pluripotent stem cell models of fanconi anemia deficiency reveal an important role for fanconi anemia proteins in cellular reprogramming and survival of hematopoietic progenitors. dans Stem cells (Dayton, Ohio) 2013
Polyclonal FANCC Primary Antibody pour WB - ABIN540662
Freie, Ciccone, Li, Plett, Orschell, Srour, Hanenberg, Schindler, Lee, Clapp: A role for the Fanconi anemia C protein in maintaining the DNA damage-induced G2 checkpoint. dans The Journal of biological chemistry 2004
Human Polyclonal FANCC Primary Antibody pour ELISA, ICC - ABIN4310504
Moeller, Yordy, Williams, Giri, Raju, Molkentine, Byers, Heymach, Story, Lee, Sturgis, Weber, Garden, Ang, Schwartz: DNA repair biomarker profiling of head and neck cancer: Ku80 expression predicts locoregional failure and death following radiotherapy. dans Clinical cancer research : an official journal of the American Association for Cancer Research 2011
The finding that FANCC overexpression reduced betacell apoptosis advances the potential for an alternative approach to the treatment of Diabetes mellitus caused by FANCC defects
Lung adenocarcinomas in both male and female patients were associated with (a) genotypic polymorphisms of FANCC and FANCD1 (Montrer BRCA2 Anticorps).
Israeli ATM (Montrer ATM Anticorps), BLM (Montrer BLM Anticorps), and FANCC heterozygous mutation carriers are not at an increased risk for developing cancer.
FANCC interacts and co-localizes with STMN1 (Montrer STMN1 Anticorps) at centrosomes during mitosis. We also showed that FANCC is required for STMN1 (Montrer STMN1 Anticorps) phosphorylation.
FANCC interferes with UNC5A's functions in apoptosis and suggest that FANCC may participate in developmental processes through association with the dependence receptor UNC5A (Montrer UNC5A Anticorps).
The successful in vitro repair of the mutated Fanconi anemia (Montrer PALB2 Anticorps) FANCC gene using the CRISPR/Cas9 system has been described.
Data indicate that TLR-indu (Montrer IL1B Anticorps)ced IL-1beta overproduction in FANCA- and FANCC-deficie (Montrer CRK Anticorps)nt mononuclear phagocyte cell lines and primary cells requires activation of the inflammasome.
deregulations of the FANCC-mediated DNA damage repair pathway and the PTCH1 (Montrer PTCH1 Anticorps)-associated sonic hedgehog (Montrer SHH Anticorps) pathway are associated with the development of early dysplastic head and neck lesions.
we identified faults in two genes, Fanconi C and Bloom helicase( FANCC and BLM (Montrer BLM Anticorps)), in six families. Faults in these genes appear to increase the risk of developing breast cancer
FANCC polymorphisms might be associated with the obstructive symptoms in allergic diseases.
Genetic deletion of Fancc blocks the autophagic clearance of viruses (virophagy) and increases susceptibility to lethal viral encephalitis. Fanconi anemia complementation group C (FANCC) protein interacts with Parkin (Montrer PARK2 Anticorps), is required in vitro and in vivo for clearance of damaged mitochondria, and decreases mitochondrial reactive oxygen species (ROS (Montrer ROS1 Anticorps)) production and inflammasome activation.
Data show that Fanconi anemia (Montrer PALB2 Anticorps), complementation group C protein knockout (Fancc -/-) mice develop hematopoietic chromosomal instability followed by leukemia in an age-dependent manner.
Loss of Fancc Impairs Antibody-Secreting Cell Differentiation in Mice through Deregulating the Wnt (Montrer WNT2 Anticorps) Signaling Pathway
Combined deficiency of Foxo3a (Montrer FOXO3 Anticorps) and Fancc or Fancd2 (Montrer FANCD2 Anticorps) not only impairs the self-renewal capacity but also markedly increases the apoptosis of neural stem and progenitor cells (NSPCs), leading to defective neurogenesis.
Using mice deficient in both Mus81 (Montrer MUS81 Anticorps) and the FA pathway protein FancC, we show both proteins cooperate in parallel pathways, as concomitant loss of FancC and Mus81 (Montrer MUS81 Anticorps) triggered cell-type-specific proliferation arrest, apoptosis and DNA damage accumulation in utero.
Loss of FANCC leads to a drastic increase in stalled/collapsed forks in cells carrying an Mcm4 (Montrer MCM4 Anticorps) missense mutation.
FANCC is most likely to be critical for resistance to DNA cross-linking drug-induced DNA damage in cells transformed by JAK2 (Montrer JAK2 Anticorps) V617F mutant.
Data indicate that IL-1beta (Montrer IL1B Anticorps) overproduction from FANCC-deficient macrophages is p38 (Montrer CRK Anticorps) dependent.
Loss of FANCC expression results in an impaired emergency granulopoiesis response in a transgenic mouse model of Fanconi anemia (Montrer PALB2 Anticorps).
Compromised hematopoiesis in Fancc(-/-) animals is developmentally programmed and does not arise de novo in bone marrow.
The Fanconi anemia complementation group (FANC) currently includes FANCA, FANCB, FANCC, FANCD1 (also called BRCA2), FANCD2, FANCE, FANCF, FANCG, FANCI, FANCJ (also called BRIP1), FANCL, FANCM and FANCN (also called PALB2). The previously defined group FANCH is the same as FANCA. Fanconi anemia is a genetically heterogeneous recessive disorder characterized by cytogenetic instability, hypersensitivity to DNA crosslinking agents, increased chromosomal breakage, and defective DNA repair. The members of the Fanconi anemia complementation group do not share sequence similarity\; they are related by their assembly into a common nuclear protein complex. This gene encodes the protein for complementation group C.
Fanconi anemia, complementation group C protein
, Fanconi anemia C
, Fanconi anemia, complementation group C
, fanconi anemia group C protein-like
, Fanconi anemia group C protein-like
, Fanconi anemia group C protein
, Fanconi anemia group C protein homolog