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FUBP1 encodes a ssDNA binding protein that activates the far upstream element (FUSE) of c-myc and stimulates expression of c-myc in undifferentiated cells. De plus, nous expédions Far Upstream Element (FUSE) Binding Protein 1 Anticorps (79) et Far Upstream Element (FUSE) Binding Protein 1 Protéines (10) et beaucoup plus de produits pour cette protéine.
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Low FUBP1 expression is associated with adenovirus infection.
These results suggest that the interference with the FUBP1/FUSE interaction as a further molecular mechanism that, in addition to the inactivation of TOP1 (Montrer TOP1 Kits ELISA), may contribute to the therapeutic potential of camptothecin/SN-38.
The findings demonstrate an association between FUBP1 levels and chordoma progression and prognosis, suggesting that FUBP1 can be used as a biomarker and a potential therapeutic target.
we identified cyclin J and far upstream element-binding protein 1 (FUBP1) as novel miR-16 (Montrer GDE1 Kits ELISA) targets, which mediate miR-16 (Montrer GDE1 Kits ELISA) antiproliferative effects.
FUBP1 acts as a potential oncogene in clear cell renal cell carcinoma (ccRCC) and may be considered as a novel biomarker or an attractive treatment target of ccRCC.
FBP1 (Montrer FBP1 Kits ELISA) expression in Bcell lymphoma was also associated with poor survival outcomes. Functionally, small interfering RNAmediated silencing of FBP1 (Montrer FBP1 Kits ELISA) was able to inhibit the proliferation of Bcell lymphoma cells, resulting in G0/G1 phase cell cycle arrest.
FUBP1 may potentially stimulate c-Myc (Montrer MYC Kits ELISA) expression in ESCC and its expression may promote esophageal squamous cell carcinoma progression.
With the advent of large-scale genome sequencing technology, molecular genetic alterations in FUBP1 promoter have now been identified in the majority of oligodendrogliomas
direct connection between the cellular PI3K (Montrer PIK3CA Kits ELISA)/AKT (Montrer AKT1 Kits ELISA)/mTOR (Montrer FRAP1 Kits ELISA) signaling pathway, frequently activated in human hepatocarcinogenesis, and the enrichment of oncogenic transcription factors of the FBP (Montrer FBP1 Kits ELISA) family
Concomitant overexpression of far upstream element (FUSE) binding protein (FBP) interacting repressor (FIR (Montrer PUF60 Kits ELISA)) and its splice variants induce migration and invasion of non-small cell lung cancer cells.
FBP helps to hold multiple physiologic processes to close tolerances, at least in part by constraining Myc (Montrer MYC Kits ELISA) expression.
Our data establish FUBP1 and its recognition of single-stranded genomic DNA as an important element in the transcriptional regulation of hematopoietic stem cells self-renewal.
Apoptosis-mediated cleavage of FBP1 (Montrer FBP2 Kits ELISA) and its decreased expression in epithelial cells induces cell cycle arrest, which may play an important role in colonic epithelial disruption in colitis.
FUBP1 is an authentic substrate of Parkin (Montrer PARK2 Kits ELISA) that might play an important role in development of Parkinson disease pathology along with aminoacyl-tRNA synthetase interacting multifunctional protein type 2
This gene encodes a ssDNA binding protein that activates the far upstream element (FUSE) of c-myc and stimulates expression of c-myc in undifferentiated cells. Regulation of FUSE by FUBP occurs through single-strand binding of FUBP to the non-coding strand. This protein has been shown to function as an ATP-dependent DNA helicase.
DNA helicase V
, far upstream element-binding protein 1
, hDH V
, far upstream element (FUSE) binding protein 1
, far upstream element-binding protein
, far upstream element-binding protein 1-like
, FUSE-binding protein 1
, far upstream element (FUSE) binding protein 4
, FUSE binding protein 1