anti-Forkhead Box K1 (Foxk1) Anticorps

Transcriptional regulator that binds to the upstream enhancer region (CCAC box) of myoglobin gene. De plus, nous expédions Foxk1 Kits (2) et et beaucoup plus de produits pour cette protéine.

afficher tous les anticorps Gène GeneID UniProt
Foxk1 221937 P85037
Foxk1 17425 P42128
Foxk1 304298  
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Top anti-Foxk1 Anticorps sur anticorps-enligne.fr

Showing 10 out of 53 products:

Catalogue No. Reactivité Hôte Conjugué Application Images Quantité Livraison Prix Détails
Boeuf (Vache) Lapin Inconjugué WB WB Suggested Anti-FOXK1 Antibody Titration:  0.2-1 ug/ml  Positive Control:  HepG2 cell lysate 100 μL 2 to 3 Days
$289.00
Détails
Boeuf (Vache) Lapin Inconjugué WB WB Suggested Anti-Foxk1 Antibody Titration:  0.2-1 ug/ml  Positive Control:  Mouse Uterus 100 μL 2 to 3 Days
$289.00
Détails
Boeuf (Vache) Lapin Inconjugué WB Host:  Rabbit  Target Name:  Foxk1  Sample Type:  Rat Thymus lysates  Antibody Dilution:  1.0ug/ml 100 μL 2 to 3 Days
$289.00
Détails
Boeuf (Vache) Lapin Inconjugué WB Host:  Rabbit  Target Name:  Foxk1  Sample Type:  Mouse Liver lysates  Antibody Dilution:  1.0ug/ml 100 μL 2 to 3 Days
$289.00
Détails
Cobaye Lapin Inconjugué IHC, WB Mouse Kidney WB Suggested Anti-FOXK1 Antibody Titration:  2.5ug/ml  Positive Control:  NIH/3T3 cell lysate 100 μL 2 to 3 Days
$229.00
Détails
Humain Lapin Inconjugué IHC, WB Immunohistochemical analysis of FOXK1 staining in human breast cancer formalin fixed paraffin embedded tissue section. The section was pre-treated using heat mediated antigen retrieval with sodium citrate buffer (pH 6.0). The section was then incubated wi Western blot analysis of FOXK1 expression in Hela (A), A431 (B), Jurkat (C) whole cell lysates. 200 μL 13 to 14 Days
$487.50
Détails
Humain Lapin Inconjugué ELISA, WB Western blot analysis of extracts from Jurkat cells, using FOXK1 Antibody. The lane on the right is treated with the synthesized peptide. 100 μg 2 to 3 Days
$324.50
Détails
Boeuf (Vache) Lapin Inconjugué WB Host:  Rabbit  Target Name:  FOXK1  Sample Type:  Placenta lysates  Antibody Dilution:  1.0ug/ml 100 μL 2 to 3 Days
$289.00
Détails
Humain Lapin Inconjugué ELISA, IHC, WB 100 μL 11 to 13 Days
$359.79
Détails
Cobaye Lapin Inconjugué IHC, WB 100 μL 11 to 14 Days
$493.17
Détails

Plus d’anticorps contre Foxk1 partenaires d’interaction

Human Forkhead Box K1 (Foxk1) interaction partners

  1. the miR-498 was found to target the 3'-UTR of lncRNA CASC11 and FOXK1 mRNA.

  2. in vitro and in vivo experiments, including studies of primary human cells, show how FOXK1 and/or FOXK2 are likely to act as important regulators that reprogram cellular metabolism to induce aerobic glycolysis

  3. FOXK1 was overexpressed in human hepatocellular carcinoma and positively correlated with cancer progression. DNA hypomethylation and gene copy number variation contributed to the overexpression of FOXK1.

  4. our results uncovered a novel, unexpected regulatory function of RUFY3. We identified FOXK1 as a new RUFY3-binding protein. This study shed light on the critical role of RUFY3 in inducing EMT and in the migration and invasion phenotypes caused by abnormal FOXK1 expression.

  5. LINC02163 regulates growth and epithelial-to-mesenchymal transition phenotype via miR-593-3p/FOXK1 axis in gastric cancer cells

  6. Silencing of FOXK1 expression enhanced the inhibitory effects of miR-186-5p on OS cell proliferation, migration and invasion.

  7. This work identified that CCDC43 promoted epithelial-mesenchymal transition and was a direct transcriptional target of FOXK1 in colorectal cancer cells.

  8. Knockdown of FOXK1 reduced cell viability and HK2 expression, decreased glucose consumption and lactate production in liver cancer cells. Furthermore, FOXK1 knockdown suppressed the activation of Akt/mTOR pathway. Inhibition of Akt/mTOR pathway reduced cell viability and glycolysis of liver cancer cells.

  9. Authors show that a nuclear-cytoplasmic transport system is necessary for the mTORC1-FOXK1 signal transduction. This reaction is mediated by a shuttling protein B56, which is a regulatory subunit of PP2A and plays an essential role in the mTORC1-dependent dephosphorylation of FOXK1.

  10. Results demonstrated that knockdown of FOXK1 inhibited the proliferation and metastasis of prostate cancer, at least in part, through suppressing the Wnt/beta-catenin signaling pathway.

  11. demonstrated that circMAN2B2 acts as an oncogenic role in lung cancer through promoting FOXK1 expression by sponging miR-1275

  12. Knockdown of FOXK1 significantly inhibited hepatocellular carcinoma cell proliferation, migration and invasion.

  13. For FOXK1 and KCNA7, the age effect on the sperm epigenome was replicated in an independent cohort of 188 sperm samples.

  14. The higher expression of FOXK1 could indicate a poor prognosis in colorectal carcinoma (CRC) patients since FOXK1 induces epithelial-mesenchymal transition and promotes CRC cell invasion in vitro and in vivo.

  15. FOXK1 was overexpressed in esophageal cancer (EC) tissues compared with corresponding non-tumor tissues using immunohistochemistry. And high FOXK1 expression was related to poor differentiation of EC. The Kaplan-Meier curve indicated that high FOXK1 expression may result in poor prognosis of EC patients.

  16. c-jun promoted FOXK1-mediated proliferation and metastasis via orthotopic implantation.

  17. These findings implicate FOXK1 as a cell cycle and growth modulator that inhibits apoptosis in colon cancer cells. FOXK1-shRNA may serve as a novel and potent therapeutic agent, alone or with 5-FU, against colon cancer

  18. Data indicated the function of Foxk1 as a tumor suppressor in facilitating angiogenesis and metastasis in breast cancer.

  19. FOXK1 protein levels and activity are regulated by associating with JLP and PLK1

  20. Overall, these results demonstrate a novel role for FOXK1 in regulating the expression of antiviral genes via Nup98, from insects to humans.

Mouse (Murine) Forkhead Box K1 (Foxk1) interaction partners

  1. Knockdown of FoxK1 in primary hepatocytes decreased both glucose production and insulin's ability to suppress it. The findings expand the repertoire of insulin-dependent transcription factors and identify FoxK1 as a contributor to insulin signaling.

  2. in vitro and in vivo experiments, including studies of primary human cells, show how FOXK1 and/or FOXK2 are likely to act as important regulators that reprogram cellular metabolism to induce aerobic glycolysis

  3. FoxK1/K2 are reciprocally regulated to FoxO1 following insulin stimulation and play a critical role in the control of apoptosis, metabolism and mitochondrial function.

  4. Foxk1 mediates mTORC1-driven metabolic rewiring, and it is likely to be critical for metabolic diseases where improper mTORC1 signaling plays an important role.

  5. Foxk1 and Foxk2 are identified as transcriptional repressors of autophagy in muscle cells and fibroblasts.

  6. in the developing eyes, Foxk1 protein is localized in bipolar, amacrine and ganglion cells with an onset of Foxk1 expression from E15 onwards; expression pattern during development suggests that Foxk1 may have an important role in retinal cells

  7. Foxk1 promotes muscle progenitor cell proliferation by repressing Foxo4 transcriptional activity and inhibits myogenic differentiation by repressing Mef2 activity.

  8. the transcriptional repression of Foxk1 is dependent on the Sin3-Sds3 repression complex, and knockdown of Sds3 results in cell-cycle arrest.

  9. Foxk1 interacts with Sin3 through the N-terminal sin3 interacting domain

  10. Collectively, these studies define a Fhl2-Foxk1 cascade that regulates the myogenic progenitor cell activity in adult skeletal muscle and enhances our understanding of muscle regeneration.

  11. role in regulating cell cycle progression in the myogenic progenitor cell

  12. NMR study of DNA-binding domain of myocyte nuclear factor (Foxk1)

  13. These studies demonstrate that Sox15, Fhl3 and Foxk1 function to coordinately regulate the myogenic progenitor cell population and skeletal muscle regeneration.

Foxk1 profil antigène

Profil protéine

Transcriptional regulator that binds to the upstream enhancer region (CCAC box) of myoglobin gene. Has a role in myogenic differentiation and in remodeling processes of adult muscles that occur in response to physiological stimuli.

Gene names and symbols associated with anti-Forkhead Box K1 (Foxk1) Anticorps

  • forkhead box K1 (FOXK1) anticorps
  • forkhead box K1 (Foxk1) anticorps
  • forkhead box K1 (foxk1) anticorps
  • A630048H08Rik anticorps
  • AI463295 anticorps
  • ENSMUSG00000075577 anticorps
  • FOXK1 anticorps
  • FOXK1L anticorps
  • Gm10868 anticorps
  • Mnf anticorps
  • RGD1309643 anticorps
  • wu:fi75f05 anticorps
  • zgc:66237 anticorps

Protein level used designations for anti-Forkhead Box K1 (Foxk1) Anticorps

MNF , forkhead box protein K1 , myocyte nuclear factor , winged helix protein CWH-5 , forkhead box K1

GENE ID SPECIES
221937 Homo sapiens
17425 Mus musculus
304298 Rattus norvegicus
334470 Danio rerio
395463 Gallus gallus
489885 Canis lupus familiaris
100301617 Oryzias latipes
100600117 Nomascus leucogenys
100610392 Pan troglodytes
100724016 Cavia porcellus
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