Use your antibodies-online credentials, if available.
Il n’y a pas de produits dans votre liste de comparaison.
Votre panier est vide.
The protein encoded by IL19 is a cytokine that belongs to the IL10 cytokine subfamily. De plus, nous expédions IL19 Kits (53) et IL19 Protéines (39) et beaucoup plus de produits pour cette protéine.
Showing 10 out of 123 products:
Human Polyclonal IL19 Primary Antibody pour CyTOF, FACS - ABIN4899879
Huang, Wachi, Thai, Loukoianov, Tan, Forteza, Wu: Potentiation of IL-19 expression in airway epithelia by IL-17A and IL-4/IL-13: important implications in asthma. dans The Journal of allergy and clinical immunology 2008
Show all 2 Pubmed References
Human Polyclonal IL19 Primary Antibody pour ELISA, WB - ABIN2474953
Chang, Magracheva, Kozlov, Fong, Tobin, Kotenko, Wlodawer, Zdanov: Crystal structure of interleukin-19 defines a new subfamily of helical cytokines. dans The Journal of biological chemistry 2003
Mouse (Murine) Monoclonal IL19 Primary Antibody pour CyTOF, FACS - ABIN4899877
Uto-Konomi, Miyauchi, Ozaki, Motomura, Suzuki, Yoshimura, Suzuki, Cua, Kubo: Dysregulation of suppressor of cytokine signaling 3 in keratinocytes causes skin inflammation mediated by interleukin-20 receptor-related cytokines. dans PLoS ONE 2012
IL-19 was increased in the sera and skin of patients with cutaneous T-cell lymphoma and atopic dermatitis.
IL-19 is significantly positive correlated with UAE and Cystatin C (Montrer CST3 Anticorps). IL-19 may play an important role that contributes to the progression of diabetic nephropathy.
The authors identified highly significant associations with better melanoma overall survival for rs6673928, impacting IL19 expression (HR, 0.56; 95% CI, 0.41-0.77; P = 0.0002) and rs6695772, controlling the expression of BATF3 (Montrer BATF3 Anticorps) (HR, 1.64; 95% CI, 1.19-2.24; P = 0.0019).
a set of IL-17A (Montrer IL17A Anticorps)-regulated genes in keratinocytes, which recapitulate typical psoriasis genes exemplified by DEFB4A (Montrer DEFB4 Anticorps), S100A7 (Montrer S100A7 Anticorps), IL19 and CSF3 (Montrer CSF3 Anticorps), based on the differences in the expression profiles of cells stimulated with six cytokines versus cells stimulated with only five cytokines lacking IL-17A (Montrer IL17A Anticorps).
findings show that the expression of IL19 in biopsies of patients with active ulcerative (Montrer IL6 Anticorps)colitis was increased compared with patients with quiescent ulcerative colitis; microbial-driven expression of IL-19 by intestinal macrophages may contribute to the pathogenesis of inflammatory bowel disease
The SNP of IL-19 rs1798 may be associated with susceptibility to hepatitis B in children.
These data are the first to implicate the association between the interleukin-19 and type 2 diabetes mellitus with vascular complications.
IL-19 and Ang-2 (Montrer ANGPT2 Anticorps) might be involved in angiogenesis of T2 Diabetes mellitus complications.
This study demonstrated that IL-19 and IL-24 (Montrer IL24 Anticorps) are associated with the modulation of T-cell responses in filarial infections.
Data show reduced cytokines IL-10 (Montrer IL10 Anticorps) and IL-19 expression, and enhanced IL-20 (Montrer IL20 Anticorps) and il-1 beta (Montrer IL1B Anticorps) expression in chronic recurrent multifocal osteomyelitis (CRMO) monocytes.
Data, including data from studies using knockout mice, suggest that interleukin-19 suppresses hapten-dependent skin inflammation in elicitation phase of contact hypersensitivity.
Attenuated colitis in IL-19-deficient animals was associated with reduced numbers of IL-6 (Montrer IL6 Anticorps)-producing macrophages in the inflamed colonic lamina propria. Microbial-driven expression of IL-19 by intestinal macrophages may contribute to the pathogenesis of inflammatory bowel disease.
IL-19 can promote angiogenesis in the absence of hypoxia by at least two distinct mechanisms: 1) direct effects on vascular cells and 2) indirect effects by stimulation of macrophages.
IL-19 plays an anti-inflammatory role in the Th2-mediated colitis model.
microglia may self-limit their inflammatory response by producing the negative regulator IL-19.
Lack of IL-19 exacerbates intimal hyperplasia
we delineate the detailed molecular pathway for IL-4 (Montrer IL4 Anticorps) up-regulation of IL-19 in keratinocytes, which may play an important role in AD pathogenesis.
Il-19 expression is increased in astrocytes following bacterial challenge.
IL-19 is important for cutaneous wound healing because it upregulates KGF (Montrer FGF7 Anticorps) expression.
IL-19 is a potent inhibitor of experimental atherosclerosis.
The protein encoded by this gene is a cytokine that belongs to the IL10 cytokine subfamily. This cytokine is found to be preferentially expressed in monocytes. It can bind the IL20 receptor complex and lead to the activation of the signal transducer and activator of transcription 3 (STAT3). A similar cytokine in mouse is reported to up-regulate the expression of IL6 and TNF-alpha and induce apoptosis, which suggests a role of this cytokine in inflammatory responses. Alternatively spliced transcript variants encoding the distinct isoforms have been described.
, melanoma differentiation associated protein-like protein
, melanoma differentiation-associated protein-like protein
, interleukin 20