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TNFAIP1 was identified as a gene whose expression can be induced by the tumor necrosis factor alpha (TNF) in umbilical vein endothelial cells. De plus, nous expédions TNFAIP1 Protéines (8) et et beaucoup plus de produits pour cette protéine.
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Human Polyclonal TNFAIP1 Primary Antibody pour IHC (f), ICC - ABIN967193
Wolf, Marks, Sarma, Byers, Katz, Shows, Dixit: Characterization of a novel tumor necrosis factor-alpha-induced endothelial primary response gene. dans The Journal of biological chemistry 1992
Show all 2 Pubmed References
TNFAIP1 silence significantly increased the migrated and invaded cells compared to that in control, while these increases were abolished by miR (Montrer MLXIP Anticorps)-424 suppression.
CREB (Montrer CREB1 Anticorps) is a negative regulator of the TNFAIP1 gene.
TNFAIP1 plays an important role in mediating miR (Montrer MLXIP Anticorps)-15a dependent biological functions in osteosarcoma.
MiR (Montrer MLXIP Anticorps)-181a played a critical role in regulating pancreatic cancer growth and migration, likely interacting with TNFAIP1.
Results showed that the expression of TNFAIP1 protein was significantly increased in osteosarcoma tissues and associated with distant metastasis.
Expression of TNFAIP1 is regulated by the transcriptional factor Sp1 (Montrer PSG1 Anticorps).
TNFAIP1 inhibited the transcriptional activities of nuclear factor kappa B (NF-kappaB (Montrer NFKB1 Anticorps)) and activating protein-1 reporters
CK2 (Montrer CSNK2A1 Anticorps) could phosphorylate TNFAIP1 in vitro and in vivo, which facilitated the distribution of TNFAIP1 in nucleus and enhanced its interaction with PCNA (Montrer PCNA Anticorps).
The promoter region of human TNFAIP1 gene was functionally characterized.
suggest that the TNFAIP1/POLDIP2 (Montrer POLDIP2 Anticorps) complex sense-antisense architecture represents a clinically significant transcriptional structural-functional gene module associated with amplification of the genomic region on 17q11.2 in breast cancer.
Results demonstrated that TNFAIP1 was significantly upregulated by Abeta25-35 in mouse primary cortical neurons and N2a cells, and TNFAIP1 may be a key player that mediated Abeta25-35-induced neurotoxicity by inactivating the Akt (Montrer AKT1 Anticorps)/CREB (Montrer CREB1 Anticorps) signaling pathway, and in turn downregulating anti-apoptotic protein Bcl-2 (Montrer BCL2 Anticorps).
ABETA (Montrer APP Anticorps) 25-35 downregulated miR (Montrer MLXIP Anticorps)-137 and upregulated TNFAIP1 in cortical neurons and N2a cells.
Study identifies Bacurd1/Kctd13 and Bacurd2/Tnfaip1 as interacting partners to RhoA GTPase proteins which influence the development of cortical neurons.
Bacurd2 influences the multipolar-to-bipolar transition of radially migrating neurons in a cell autonomous fashion. Bacurd2 and Rnd2 (Montrer RND2 Anticorps) interact to promote radial migration within the embryonic cortex.
Estrogen and ERbeta (Montrer ESR2 Anticorps) regulate Tnfaip1 expression in mouse hippocampus.
This gene was identified as a gene whose expression can be induced by the tumor necrosis factor alpha (TNF) in umbilical vein endothelial cells. Studies of a similar gene in mouse suggest that the expression of this gene is developmentally regulated in a tissue-specific manner.
BTB/POZ domain-containing protein TNFAIP1
, tumor necrosis factor, alpha-induced protein 1 (endothelial)
, BTB/POZ domain-containing adapter for CUL3-mediated RhoA degradation protein 2
, tumor necrosis factor, alpha-induced protein 1-like
, BTB/POZ domain-containing protein TNFAIP1-like
, tumor necrosis factor induced protein 1
, tumor necrosis factor-induced protein 1