ATM anticorps (pSer1981) (Biotin)

N° du produit ABIN1954960

Cet anticorps Lapin Polyclonal détecte spécifiquement ATM dans ELISA et DB. Il présente une réactivité envers Humain.

Aperçu rapide pour ATM anticorps (pSer1981) (Biotin) (ABIN1954960)

Antigène: ATM (Ataxia Telangiectasia Mutated (ATM))

Reactivité: Humain

Hôte: Lapin

Clonalité: Polyclonal

Conjugué: Cet anticorp ATM est conjugé à/à la Biotin

Application: ELISA, Dot Blot (DB)

Détail du produit anti-ATM anticorps (Biotin)

Épitope: pSer1981

Specificité: This Phospho-ATM-pS1981 antibody is generated from rabbits immunized with a KLH conjugated synthetic phosphopeptide corresponding to amino acid residues surrounding S1981 of human ATM.

Purification: Protein A purified

Isotype: IgG

Information d'application

Indications d'application: Approved: DB, ELISA

Usage: The applications listed have been tested for the unconjugated form of this product. Other forms have not been tested.

Commentaires:

Target Species of Antibody: Human

Restrictions: For Research Use only

Stockage

Format: Liquid

Concentration: Lot specific

Buffer: PBS,  pH 7.2, 0.09 % sodium azide.

Agent conservateur: Sodium azide

Précaution d'utilisation: This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

Conseil sur la manipulation: Aliquot to avoid repeated freezing and thawing.

Stock: 4 °C,-20 °C

Stockage commentaire: Short term: store at 4°C. Long term: aliquot and store -20°C for up to 6 months. Avoid freeze-thaw cycles. Protect from light.

Date de péremption: 6 months

Détails sur ATM

Antigène: ATM (Ataxia Telangiectasia Mutated (ATM))

Autre désignation: ATM

Sujet: Name/Gene ID: ATM
Subfamily: PI3/PI4
Family: Protein Kinase

Synonyms: ATM, A-T mutated, ATA, ATC, ATD, ATE, AT mutated, Ataxia telangiectasia, ATDC, Serine-protein kinase ATM, AT1, Ataxia telangiectasia mutated, TEL1, TELO1

ID gène: 472

Pathways: Signalisation p53, Apoptose, Réparation de l'ADN, Inositol Metabolic Process, Positive Regulation of Response to DNA Damage Stimulus