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Both CLU (Montrer CLU Anticorps) and PLXNA4 have been genetically associated with Alzheimer disease (AD) risk and our data thus provide a direct relationship between two AD risk genes. Our data suggest that increasing the levels of PLXNA4 or targeting CLU (Montrer CLU Anticorps)-PLXNA4 interactions may have therapeutic value in AD.
Data indicate that plexin A1 (Montrer PLXNA1 Anticorps)-4 (PLXNA1 (Montrer PLXNA1 Anticorps)-4) mediation of neuroanatomical traits can be detected using in vivo neuroimaging techniques.
There is a significant association between Alzheimer's disease risk and SNPs in PLXNA4.
Rare variants in PLXNA4 and Parkinson's disease.
Plexin-A4 promotes tumor progression and tumor angiogenesis by enhancement of VEGF (Montrer VEGFA Anticorps) and bFGF (Montrer FGF2 Anticorps) signaling.
These findings implicate Semaphorin-6A (Montrer SEMA6A Anticorps) - Plexin-A2 (Montrer Plxna2 Anticorps)/Plexin-A4 interactions in dorsal lateral geniculate nucleus axon guidance and in the spatiotemporal organization of guidepost cell populations in the mammalian subpallium.
These findings suggest that by interacting with PlexA4, TrkA (Montrer NTRK1 Anticorps) plays a crucial role in redirecting local Sema3A (Montrer SEMA3A Anticorps) signaling to retrograde axonal transport, thereby regulating dendritic GluA2 (Montrer GRIA2 Anticorps) localization and patterning.
Distinct cytoplasmic domains in Plexin-A4 mediate diverse responses to semaphorin 3A (Montrer SEMA3A Anticorps) in developing mammalian neurons.
results suggest that Plexin-A4 is involved in the precise positioning of oligodendrocyte precursor cells in developing cerebral cortex.
null mutations in Sema6A (Montrer SEMA6A Anticorps) or PlexinA4 (PlexA4) exhibit a pronounced defect in OPL (Montrer ZIC1 Anticorps) stratification of horizontal cell axons without any apparent deficits in bipolar cell dendrite or photoreceptor axon targeting.
Findings suggest plexin-A4 and Sema3A (Montrer SEMA3A Anticorps) as new intervention points for treating sepsis.
suggesting that a Semaphorin6A-Plexin-A4 cis (Montrer CISH Anticorps) interaction serves as an inhibitory mechanism.
plexin-A4 was expressed in the developing nervous system with a pattern different to that of other members of the plexin-A subfamily (plexin-A1 (Montrer PLXNA1 Anticorps), plexin-A2 (Montrer Plxna2 Anticorps) and plexin-A3 (Montrer PLXNA3 Anticorps))
PlexA4 expressed in oligodendrocyte precursor cells acts as a mediator of semaphorin signals
Involved in the development of primary sensory neurons especially in branching of the peripheral axons. Interacts with the SLIT2 signaling specifically to promote axonal branching of Rohon-Beard neurons and the trigeminal sensory ganglion neurons.
, plexin A4, A
, plexin A4
, putative plexin 2