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anti-Human ASCL2 Anticorps:
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Cow (Bovine) Polyclonal ASCL2 Primary Antibody pour WB - ABIN2779425
Shahib, Martaadisoebrata, Kato: Detection of HASH2 (ASCL2) gene expression in gestational trophoblastic disease. dans The Journal of reproductive medicine 2006
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We address this paradox using basic helix-loop-helix (bHLH) transcription factors ASCL1 (Montrer ASCL1 Anticorps), ASCL2, and MYOD1 (Montrer MYOD1 Anticorps), crucial mediators of lineage specification..Although the ASCL factors and MYOD1 (Montrer MYOD1 Anticorps) have some distinct DNA motif preference, it is not sufficient to explain the extent of the differential binding. All three factors can bind inaccessible chromatin and induce changes in chromatin accessibility and H3K27ac
this study shows that Ascl2 level was higher in peripheral blood mononuclear cells from Sjogren's syndrome patients compared with those from healthy controls
A novel HIF-1alpha (Montrer HIF1A Anticorps)/Ascl2/miR (Montrer MLXIP Anticorps)-200b regulatory feedback circuit in modulating EMT (Montrer ITK Anticorps)-MET plasticity of CRC (Montrer CALR Anticorps) cells, which could serve as a possible therapeutic target.
WiNTRLINC1 interacts with TCF4 (Montrer TCF4 Anticorps)/beta-catenin (Montrer CTNNB1 Anticorps) to mediate the juxtaposition of its promoter with the regulatory regions of ASCL2.
Ascl2 over-expression is associated with colorectal neoplasms.
expression of ASCL2 may identify an aggressive subgroup in lung squamous cell carcinoma
SEMA3F (Montrer SEMA3F Anticorps) functions as a suppressor of colorectal cancer metastasis by down-regulating the ASCL2-CXCR4 (Montrer CXCR4 Anticorps) signaling axis.
SNAIL1 (Montrer SNAI1 Anticorps) combines competitive displacement of ASCL2 and epigenetic mechanisms to rapidly silence the EPHB3 (Montrer EPHB3 Anticorps) tumor suppressor in colorectal cancer.
ASCL2 gene expression is regulated by miR (Montrer MLXIP Anticorps)-200a/b/c, miR (Montrer MLXIP Anticorps)-141 and miR429 levels in colon cancer.
Ascl2 directly initiates follicular T-helper cell development
More DNMT1 (Montrer DNMT1 Anticorps) mRNA was detected in the transgenic somatic cell nuclear transfer (SCNT) group than the other three groups. Hsp 70.1 mRNA was detected in the in vitro fertilzation embryos. Mash2 mRNA was present at highest levels in transgenic SCNT embryos.
Mash2 is highly conserved across species and is specifically expressed in the bovine placenta. Bovine Mash2 appears to be maternally expressed after implantation, but the paternal genome plays a role in regulating expression.
Ascl2 inhibits myogenic differentiation by targeting MRFs and facilitates the generation of postnatal satellite cells.
overexpression of the imprinted Ascl2 gene has considerable consequences for placental development, specifically for the parietal trophoblast giant cell and spongiotrophoblast lineages both of which express pregnancy-related hormones.
The ascl2 forms a transcriptional switch that is both Wnt (Montrer WNT2 Anticorps) responsive and Wnt (Montrer WNT2 Anticorps) dependent to define stem cell identity.
TSSC3 (Montrer PHLDA2 Anticorps) plays an important role in the differentiation from trophoblast stem cell to trophoblast progenitors and/or labyrinth trophoblasts through the TSSC3 (Montrer PHLDA2 Anticorps)/PI3K/AKT (Montrer AKT1 Anticorps)/MASH2 signaling pathway.
Elevated expression of Ascl2 in a Wnt (Montrer WNT2 Anticorps) signalling dependent manner specifically in the stem cell compartment of the intestine neither increases tumour formation nor diminishes survival in a well established intestinal tumour model, the Apc (Montrer APC Anticorps)(min) mouse.
Ascl2 knockdown results in tumor growth arrest by miRNA-302b-related inhibition of colon cancer progenitor cells
Partial loss of Ascl2 function affects all three layers of the mature placenta and causes intrauterine growth restriction.
Mash2 gene is preferentially expressed from the paternal allele.
The interval between Ins2 and Ascl2 is dispensable for imprinting centre function in the mouse model of Beckwith-Wiedemann Syndrome.
The lack of differential expression by microarray profiling of fetal tissues from swine parthenotes and control bi-parental fetuses supports lack of imprinting at ASCL2 in pigs.
This gene is a member of the basic helix-loop-helix (BHLH) family of transcription factors. It activates transcription by binding to the E box (5'-CANNTG-3'). Dimerization with other BHLH proteins is required for efficient DNA binding. Involved in the determination of the neuronal precursors in the peripheral nervous system and the central nervous system.
, achaete-scute complex-like 2
, achaete-scute homolog 2
, class A basic helix-loop-helix protein 45
, mammalian achaete/scute homologue 2
, transcription factor cash4
, achaete-scute complex homolog 2 (Drosophila)
, achaete-scute complex homolog-like 2
, achaete scute-like protein 2
, achaete-scute complex-like protein 2
, long transient receptor potential-related channel 5
, mammalian achaete scute homolog 2
, achaete-scute complex homolog 2