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The protein encoded by EHD3 is a member of the parathyroid hormone family. De plus, nous expédions EHD3 Protéines (6) et beaucoup plus de produits pour cette protéine.
Showing 10 out of 32 products:
Human Monoclonal EHD3 Primary Antibody pour IF, IHC (p) - ABIN565474
Dumas, Lê-Bury, Marie-Anaïs, Herit, Mazzolini, Guilbert, Bourdoncle, Russell, Benichou, Zahraoui, Niedergang: The HIV-1 protein Vpr impairs phagosome maturation by controlling microtubule-dependent trafficking. dans The Journal of cell biology 2015
these data suggest that PTHrP(12-48) is a bioactive breast cancer-derived peptide that locally regulates the differentiation of hematopoietic cells and the activity of osteoclasts within the tumor-bone marrow microenvironment, perhaps to facilitate tumor control of bone.
RSK activation via ERK modulates human colon cancer cells response to PTHrP.
Data provide evidence that EHD3 might undergo its tumor suppressor functions at least in part through promoting the sorting of activated EGFR (Montrer EGFR Anticorps) to the lysosomal degradative compartment and spatio-temporal regulation of EGFR (Montrer EGFR Anticorps) signaling via ERK (Montrer EPHB2 Anticorps) and AKT (Montrer AKT1 Anticorps).
this is the first time that mutation recurrence has been detected in PTHLH associated with brachydactyly type E and we have observed that, as remarked before, this syndrome has high intrafamilial as well as interfamilial variability
Antagonistic antibodies against the death receptors demonstrated that Apo2L/TRAIL mediated its apoptotic signals through activation of the TRAIL-R2 in PTHrP expressing breast cancer cells
Amino PTHrP and carboxyl PTHrP can vary independently in different lung carcinomas. Carboxyl PTHrP may temper the stimulatory effect of amino PTHrP on cancer progression.
PTHLH is a gastrin (Montrer GAST Anticorps)-regulated growth factor that might contribute to gastric epithelial cell homeostasis.
Duplication of PTHLH co-segregates with osteochondroplasia with a combined brachydactyly type E/A1 in a three-generation pedigree.
Data shown here expand the knowledge on the phenotypic presentation of PTHLH mutations, highlighting significant clinical variability and incomplete penetrance of the PTHLH-related symptoms.
We showed that the supplementation of the osteogenic differentiation medium with PTHrP inhibited the alkaline phosphatase activity and the expression of the transcription factor DLX3 (Montrer DLX3 Anticorps), but the depletion of PTHrP did not support the differentiation of DFCs.We showed that SUFU (Suppressor Of Fused Homolog (Montrer SUFUH Anticorps)) was not regulated during the osteogenic differentiation in DFCs
EHD3 and phosphatidic acid cooperatively regulate membrane deformation and trafficking from early endosomes.
CaV3.1 (Montrer CACNA1G Anticorps) and CaV3.2 (Montrer CACNA1H Anticorps) are substrates for EHD3-dependent protein trafficking in heart
Early steps in primary cilium assembly require EHD1/EHD3-dependent ciliary vesicle formation.
EHD3 is a critical component of protein trafficking in heart and is essential for the proper membrane targeting of select cellular proteins that maintain excitability.
EHD-mediated endocytic traffic of key surface receptors such as VEGFR2 (Montrer KDR Anticorps) is essential for physiological control of glomerular function.
EHD3: a protein that resides in recycling tubular and vesicular membrane structures and interacts with EHD1 (Montrer EHD1 Anticorps).
Early steps in primary cilium assembly require EHD1/EHD3 (Montrer EHD1 Anticorps)-dependent ciliary vesicle formation.
The protein encoded by this gene is a member of the parathyroid hormone family. This hormone regulates endochondral bone development and epithelial-mesenchymal interactions during the formation of the mammary glands and teeth. This hormone is involved in lactation possibly by regulating the mobilization and transfer of calcium to the milk. The receptor of this hormone, PTHR1, is responsible for most cases of humoral hypercalcemia of malignancy. Four alternatively spliced transcript variants encoding two distinct isoforms have been observed. There is also evidence for alternative translation initiation from non-AUG (CUG and GUG) start sites, in-frame and downstream of the initiator AUG codon, to give rise to nuclear forms of this hormone.
EH domain containing 3
, EH domain-containing protein 3
, PAST homolog 3
, PTH-related protein
, parathyroid hormone-like related protein
, parathyroid hormone-related protein
, EH-domain containing protein 2
, EH-domain containing 3
, EH domain-containing protein 3-like
, LOW QUALITY PROTEIN: EH domain-containing protein 3