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transcriptional repressor and an immediate-early gene [RGD, Feb 2006].. De plus, nous expédions NAB1 Kits (6) et NAB1 Protéines (6) et beaucoup plus de produits pour cette protéine.
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Chlamydomonas reinhardtii (C. reinhardtii) Polyclonal NAB1 Primary Antibody pour IP, WB - ABIN2487326
Blifernez, Wobbe, Niehaus, Kruse: Protein arginine methylation modulates light-harvesting antenna translation in Chlamydomonas reinhardtii. dans The Plant journal : for cell and molecular biology 2010
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Cow (Bovine) Polyclonal NAB1 Primary Antibody pour IHC, WB - ABIN2779429
Brandenberger, Wei, Zhang, Lei, Murage, Fisk, Li, Xu, Fang, Guegler, Rao, Mandalam, Lebkowski, Stanton: Transcriptome characterization elucidates signaling networks that control human ES cell growth and differentiation. dans Nature biotechnology 2004
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Human Polyclonal NAB1 Primary Antibody pour ChIP, IP - ABIN4337717
Kearney, Delgado, Eshleman, Hill, OConnor, Lenz: Type I IFNs downregulate myeloid cell IFN-γ receptor by inducing recruitment of an early growth response 3/NGFI-A binding protein 1 complex that silences ifngr1 transcription. dans Journal of immunology (Baltimore, Md. : 1950) 2013
These data suggest that type I IFN stimulation induces a rapid recruitment of a repressive Egr3 (Montrer EGR3 Anticorps)/Nab1 complex that silences transcription from the ifngr1 (Montrer IFNGR1 Anticorps) promoter.
Data show that the suppressive effect of valproic acid on chondrocytes is not due to reduced expression or recruitment of Egr-1 (Montrer EGR1 Anticorps) to the mPGES-1 (Montrer PTGES Anticorps) promoter and involves upregulation of NAB1.
Methylation pattern of the corresponding NGFI-A (Montrer EGR1 Anticorps) binding site in the human glucocorticoid receptor (Montrer NR3C1 Anticorps) exon 1-F specific promoter in post mortem hippocampal tissue.
In the green unicellular alga Chlamydomonas reinhardtii, the cytosolic RNA-binding protein NAB1 represses translation of certain LHCII isoform mRNAs. Specific nitrosylation of Cys (Montrer DNAJC5 Anticorps)-226 decreases NAB1 activity and could be demonstrated in vitro and in vivo.
NAB1 associates with RNA transcripts through a mechanism involving its CSD (Montrer CSAD Anticorps) that is conserved with mechanisms of sequence-specific nucleic acid recognition employed by ancestrally related bacterial cold-shock proteins (CSPs).
All examined cysteine single and double mutants exhibited a reduced antenna at photosystem II caused by a perturbed NAB1 deactivation mechanism.
Nab1 is a potent inhibitor of cardiac growth in response to pathological stimuli in vivo.
Nab1 and Nab2 proteins are necessary for Schwann cells to exit the cell cycle, downregulate suppressed cAMP-inducible protein (SCIP) expression and upregulate expression of critical myelination genes.
Egr2 (Montrer EGR2 Anticorps) directly represses genes during myelination in conjunction with NAB corepressors
transcriptional repressor and an immediate-early gene
EGR-1-binding protein 1
, EGR1 binding protein 1
, NGFI-A-binding protein 1
, transcriptional regulatory protein p54
, NGFI-A binding protein 1 (EGR1 binding protein 1)
, NGF1A-binding protein
, NGFI-A-binding protein 1-like
, nucleic acid binding protein
, transcriptional repressor TA16