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transcriptional repressor and an immediate-early gene [RGD, Feb 2006].. De plus, nous expédions NAB1 Kits (6) et NAB1 Protéines (6) et beaucoup plus de produits pour cette protéine.
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Chlamydomonas reinhardtii (C. reinhardtii) Polyclonal NAB1 Primary Antibody pour IP, WB - ABIN2487326
Blifernez, Wobbe, Niehaus, Kruse: Protein arginine methylation modulates light-harvesting antenna translation in Chlamydomonas reinhardtii. dans The Plant journal : for cell and molecular biology 2010
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Cow (Bovine) Polyclonal NAB1 Primary Antibody pour IHC, WB - ABIN2779429
Brandenberger, Wei, Zhang, Lei, Murage, Fisk, Li, Xu, Fang, Guegler, Rao, Mandalam, Lebkowski, Stanton: Transcriptome characterization elucidates signaling networks that control human ES cell growth and differentiation. dans Nature biotechnology 2004
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Human Polyclonal NAB1 Primary Antibody pour ChIP, IP - ABIN4337717
Kearney, Delgado, Eshleman, Hill, OConnor, Lenz: Type I IFNs downregulate myeloid cell IFN-γ receptor by inducing recruitment of an early growth response 3/NGFI-A binding protein 1 complex that silences ifngr1 transcription. dans Journal of immunology (Baltimore, Md. : 1950) 2013
These data suggest that type I IFN stimulation induces a rapid recruitment of a repressive Egr3/Nab1 complex that silences transcription from the ifngr1 promoter.
Data show that the suppressive effect of valproic acid on chondrocytes is not due to reduced expression or recruitment of Egr-1 to the mPGES-1 promoter and involves upregulation of NAB1.
mutations in the human NABI gene are most likely not involved in the pathogenesis of peripheral neuropathies.
Methylation pattern of the corresponding NGFI-A binding site in the human glucocorticoid receptor exon 1-F specific promoter in post mortem hippocampal tissue.
In the green unicellular alga Chlamydomonas reinhardtii, the cytosolic RNA-binding protein NAB1 represses translation of certain LHCII isoform mRNAs. Specific nitrosylation of Cys-226 decreases NAB1 activity and could be demonstrated in vitro and in vivo.
NAB1 associates with RNA transcripts through a mechanism involving its CSD that is conserved with mechanisms of sequence-specific nucleic acid recognition employed by ancestrally related bacterial cold-shock proteins (CSPs).
All examined cysteine single and double mutants exhibited a reduced antenna at photosystem II caused by a perturbed NAB1 deactivation mechanism.
Nab1 is a potent inhibitor of cardiac growth in response to pathological stimuli in vivo.
Nab1 and Nab2 proteins are necessary for Schwann cells to exit the cell cycle, downregulate suppressed cAMP-inducible protein (SCIP) expression and upregulate expression of critical myelination genes.
Egr2 directly represses genes during myelination in conjunction with NAB corepressors
transcriptional repressor and an immediate-early gene
EGR-1-binding protein 1
, EGR1 binding protein 1
, NGFI-A-binding protein 1
, transcriptional regulatory protein p54
, NGFI-A binding protein 1 (EGR1 binding protein 1)
, NGF1A-binding protein
, NGFI-A-binding protein 1-like
, nucleic acid binding protein
, transcriptional repressor TA16