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MAGE-Like 2 anticorps

MAGEL2 Reactivité: Humain ELISA, IHC Hôte: Lapin Polyclonal unconjugated
N° du produit ABIN7001382
  • Antigène Voir toutes MAGE-Like 2 (MAGEL2) Anticorps
    MAGE-Like 2 (MAGEL2)
    Reactivité
    • 33
    • 2
    • 2
    • 2
    • 2
    • 1
    • 1
    • 1
    Humain
    Hôte
    • 33
    Lapin
    Clonalité
    • 33
    Polyclonal
    Conjugué
    • 8
    • 2
    • 2
    • 2
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    Cet anticorp MAGE-Like 2 est non-conjugé
    Application
    • 13
    • 13
    • 11
    • 6
    • 6
    • 5
    • 3
    • 1
    ELISA, Immunohistochemistry (IHC)
    Attributs du produit
    Polyclonal Antibody
    Purification
    Affinity purification
    Immunogène
    Synthetic peptide of human MAGEL2
    Isotype
    IgG
    Top Product
    Discover our top product MAGEL2 Anticorps primaire
  • Indications d'application
    IHC 1:50-1:200
    Restrictions
    For Research Use only
  • Format
    Liquid
    Concentration
    0.8 mg/mL
    Buffer
    PBS with 0.05 % sodium azide and 50 % glycerol, PH7.4
    Agent conservateur
    Sodium azide
    Précaution d'utilisation
    This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
    Stock
    -20 °C
    Stockage commentaire
    Store at -20°C. Avoid freeze / thaw cycles.
  • Antigène
    MAGE-Like 2 (MAGEL2)
    Autre désignation
    MAGEL2 (MAGEL2 Produits)
    Synonymes
    anticorps NDNL1, anticorps nM15, anticorps MAGEL2, anticorps Mage-l2, anticorps ns7, anticorps MAGE family member L2, anticorps melanoma antigen, family L, 2, anticorps MAGEL2, anticorps Magel2
    Sujet
    Prader-Willi syndrome (PWS) is caused by the loss of expression of imprinted genes in chromosome 15q11-q13 region. Affected individuals exhibit neonatal hypotonia, developmental delay, and childhood-onset obesity. Necdin (NDN), a gene involved in the terminal differentiation of neurons, localizes to this region of the genome and has been implicated as one of the genes responsible for the etiology of PWS. This gene is structurally similar to NDN, is also localized to the PWS chromosomal region, and is paternally imprinted, suggesting a possible role for it in PWS.
    NCBI Accession
    NP_061939
    UniProt
    Q9UJ55
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