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Human Polyclonal CLDN4 Primary Antibody pour IF (p), IHC (p) - ABIN680743
Wang, Chen, Liang, Yan, Lin, Liu, Luo, Huang, Li, Liu, Tang: Notch inhibition promotes fetal liver stem/progenitor cells differentiation into hepatocytes via the inhibition of HNF-1?. dans Cell and tissue research 2014
Human Polyclonal CLDN4 Primary Antibody pour ICC, IF - ABIN407633
Rho, Lampe: High-throughput screening for native autoantigen-autoantibody complexes using antibody microarrays. dans Journal of proteome research 2013
Data indiate a regulatory network in gastric cancer whereby claudin-4 expression is reduced by specific miRNAs, which are in turn bound by specific lncRNAs acting as competing endogenous RNAs (ceRNAs), resulting in increased claudin-4 expression.
This is the first study to show how TGF-beta (Montrer TGFB1 Anticorps) regulates the expression of Claudin-4 through c-Jun (Montrer JUN Anticorps) signaling and how this pathway contributes to the migratory and tumorigenic phenotype of lung tumor cells.
Claudin-4 functionally contributes to both ovarian tumor cell apoptosis resistance and migration and targeting extracellular loop interactions of claudin-4 may have therapeutic implications for reducing ovarian tumor burden.
Fluorescence-based flow cytometry and xenon magnetic resonance imaging (MRI (Montrer C7ORF49 Anticorps)) indicate binding of the biosensor specifically to claudin 4 (Cldn4)-expressing cells.
Studies indicate that Grainyhead-like transcription factor 2 (Montrer HNF1B Anticorps) (GRHL2 (Montrer GRHL2 Anticorps)) controls the expression of E-cadherin (CDH1 (Montrer CDH1 Anticorps)) required for adherens junctions and possibly regulates the expression of claudin-4 (CLDN4) in tight junctions.
Studies indicate claudin 1 (CLDN-1 (Montrer CLDN1 Anticorps)) as a target for improving epidermal drug absorption and preventing HCV infection and of claudin 4 (CLDN-4) as a target for anticancer therapeutics.
Mislocalization claudin-3 (Montrer CLDN3 Anticorps) to nucleus in colon cancer and mislocalization claudin-4 to nucleus in adenomas of the colon were detected for the first time. The potential reasons for the paradoxical expression are discussed and a review of the literature, related all the alleged mechanisms of this mislocalization is provided.
Reg (Montrer EXTL3 Anticorps) I may play a role in the maintenance of mucosal barrier function by inducing tight junction proteins such as claudins 3 and 4.
claudin-4 may represent different mechanisms of lymphatic vessel invasion with both biomarkers is related to poor prognosis
These results suggested that increase of Cldn4-expression may be involved in early molecular events during carcinogenesis of adenocarcinoma, whereas increase of Cldn7 (Montrer CLDN7 Anticorps)-expression may be associated with tumor invasion or progression.
Reg (Montrer KCNH2 Anticorps) I may play a role in the maintenance of mucosal barrier function by inducing tight junction proteins such as claudins 3 and 4.
a Grhl2 (Montrer GRHL2 Anticorps)/Ovol2 (Montrer OVOL2 Anticorps) network controls Cldn4 and Rab25 (Montrer RAB25 Anticorps) expression that facilitates lumen expansion and barrier formation in subtypes of renal epithelia
Data show that claudin-4 and claudin-7 (Montrer CLDN7 Anticorps) were observed in hepatocytes of severely damaged mouse and human livers.
The claudin-4-mediated chloride conductance can be regulated endogenously by a protease-channel-activating protease 1 (cap1 (Montrer PRSS8 Anticorps)).
Claudin 4 has little effect on normal lung physiology but may function to protect against acute lung injury in mice.
The results suggest that progressive hydronephrosis in Cldn4(-/-) mice arises from urinary tract obstruction due to urothelial hyperplasia, and that Cld4 plays an important role in maintaining the homeostatic integrity of normal urothelium.
the results indicate that Grhl2 (Montrer GRHL2 Anticorps) regulates epithelial morphogenesis through transcriptional up-regulation of Cldn3 (Montrer CLDN3 Anticorps) and Cldn4, as well as of Rab25 (Montrer RAB25 Anticorps), which increases the Cldn4 protein and its localization at TJs
Mechanism of Clostridium perfringens enterotoxin interaction with claudin-3 (Montrer CLDN3 Anticorps)/-4 protein suggests structural modifications of the toxin to target specific claudins
Claudin-3 (Montrer CLDN3 Anticorps) in the apical-most regions maintains the impermeable tight junctions during lactation, and claudin-4 contributes
Claudin-4 overexpression is associated with epigenetic derepression in gastric carcinoma.
Tumor necrosis factor-alpha (Montrer TNF Anticorps) increases claudin-1 (Montrer CLDN1 Anticorps), 4, and 7 expression in renal tubular cells, altering permeability and transepithelial electrical resistance.
Claudin-4 expression shows age-dependent change in cellular localization on pig jejunal villous epithelial cells
These findings indicate that claudin-4 and -7 may play a role in the gingiva junctional epithelium even in the absence of tight junctions.
This gene encodes an integral membrane protein, which belongs to the claudin family. The protein is a component of tight junction strands and may play a role in internal organ development and function during pre- and postnatal life. This gene is deleted in Williams-Beuren syndrome, a neurodevelopmental disorder affecting multiple systems.
, Clostridium perfringens enterotoxin receptor 1
, Williams-Beuren syndrome chromosomal region 8 protein
, clostridium perfringens enterotoxin receptor
, tight junction-associated protein
, claudin 4