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Human Polyclonal ABCG5 Primary Antibody pour IF (p), IHC (p) - ABIN708371
Wang, Xiaoling, Pingting, Shuqiang, Yuaner: Chronic unpredictable mild stress combined with a high-fat diets aggravates atherosclerosis in rats. dans Lipids in health and disease 2014
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Human Monoclonal ABCG5 Primary Antibody pour FACS, IHC - ABIN1724857
Johnson, Lee, Pickert, Urbatsch: Bile acids stimulate ATP hydrolysis in the purified cholesterol transporter ABCG5/G8. dans Biochemistry 2010
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Human Polyclonal ABCG5 Primary Antibody pour IHC, IHC (p) - ABIN4277248
Ahn, Jang, Jun, Lee, Shin: Expression of liver X receptor correlates with intrahepatic inflammation and fibrosis in patients with nonalcoholic fatty liver disease. dans Digestive diseases and sciences 2014
Human Polyclonal ABCG5 Primary Antibody pour WB - ABIN4892532
Huang, Wang, Quan, Wang, Yang, Zhong: Lactobacillus acidophilus ATCC 4356 prevents atherosclerosis via inhibition of intestinal cholesterol absorption in apolipoprotein E-knockout mice. dans Applied and environmental microbiology 2014
Case Reports: compound heterozygous for nonsense mutations in ABCG5 responsible for sitosterolemia.
ABCG5 gene variants were not associated with cholelithiasis in patients with Gaucher disease type 1.
Genetic variations in ABCG5, CYP7A1 (Montrer CYP7A1 Anticorps), and DHCR7 (Montrer DHCR7 Anticorps) may contribute to differing responses of serum cholesterol to dairy intake among healthy adults.
ABCG5 Gene Variants are associated with Sitosterolemia and Familial Mediterranean Fever (Montrer MEFV Anticorps).
first case of a Mexican family with sitosterolemia carrying two new ABCG5 gene mutations
Genetic polymorphism within the ABCG5 gene is a risk factor for diabetes.
crystallization in lipid bilayers to determine the X-ray structure of human G5G8 in a nucleotide-free state at 3.9 A resolution, generating the first atomic model of an ABC (Montrer ABCB6 Anticorps) sterol transporter
ATP-binding cassette (ABC (Montrer ABCB6 Anticorps)) transporters G5 (ABCG5) and G8 (ABCG8 (Montrer ABCG8 Anticorps)) form an obligate heterodimer that limits intestinal absorption and facilitates biliary secretion of cholesterol and phytosterols.
ABCG5/8 variants are associated with susceptibility to coronary heart disease.
Sitosterolemia is caused by a genetic defect of sterolins (ABCG5/ABCG8 (Montrer ABCG8 Anticorps)) mapped to the STSL locus. Polymorphic variations in STSL have been linked to lipid levels and gallstone disease
high expression levels of both ATP-binding cassette sub-family G member 5 and 8 (ABCG5 and ABCG8 (Montrer ABCG8 Anticorps)) were present in bovine liver and digestive tract samples, and in the mammary gland
ABCG5 and ABCG8 (Montrer ABCG8 Anticorps) mRNA levels were significantly increased in cholesterol group and less increased in myriocin group, relative to that in normal group.
The ABCG5/G8-independent pathway plays an important role in regulating biliary cholesterol secretion, and gallstone formation, which works independently of the ABCG5/G8 pathway.
ABCG5/G8 mediate mass biliary cholesterol secretion but not from a reverse cholesterol transport-relevant pool.
AdGRP78 reduced expression of lipogenic genes and plasma triglycerides in the db/db (Montrer LEPR Anticorps) strain. Both G5 and G8 protein levels increased as did total biliary cholesterol
The data demonstrate that Abcg5/Abcg8 (Montrer ABCG8 Anticorps) deficiency reduces the uptake and secretion of both dietary triacylglycerols and cholesterol by the intestine, suggesting a novel role for the sterol transporter in the formation and secretion of chylomicrons.
Sitosterolemia is caused by a genetic defect of sterolins (ABCG5/ABCG8 (Montrer ABCG8 Anticorps)) mapped to the STSL (Montrer ABCG8 Anticorps) locus. Polymorphic variations in STSL (Montrer ABCG8 Anticorps) have been linked to lipid levels and gallstone disease
The absence of an ABCG5/ABCG8 (Montrer ABCG8 Anticorps) expression.
Mice with deficient Abcg2 have features of inflammatory DCM and that the reversibility of myocardial T cell infiltration provides a novel model for investigating the progression of myocardial fibrosis.
biliary cholesterol mass secretion under maximal bile salt-stimulated conditions is fully dependent on ABCG5/G8
This study is the first to report such toxic effects of phytosterol accumulation in ABCG5/G8 knockout mice.
The protein encoded by this gene is a member of the superfamily of ATP-binding cassette (ABC) transporters. ABC proteins transport various molecules across extra- and intra-cellular membranes. ABC genes are divided into seven distinct subfamilies (ABC1, MDR/TAP, MRP, ALD, OABP, GCN20, White). This protein is a member of the White subfamily. The protein encoded by this gene functions as a half-transporter to limit intestinal absorption and promote biliary excretion of sterols. It is expressed in a tissue-specific manner in the liver, colon, and intestine. This gene is tandemly arrayed on chromosome 2, in a head-to-head orientation with family member ABCG8. Mutations in this gene may contribute to sterol accumulation and atheroschlerosis, and have been observed in patients with sitosterolemia.
ATP-binding cassette, sub-family G (WHITE), member 5 (sterolin 1)
, ATP-binding cassette sub-family G member 5
, sterolin 1
, ATP-binding cassette transporter
, ATP-binding cassette, sub-family G (WHITE), member 5
, ATP-binding cassette sub-family G member 5-like
, ATP-binding cassette, subfamily G, member 5
, ATP-binding cassette sub-family G (WHITE) member 5 (sterolin 1)