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anti-Rat (Rattus) Tumor Protein p73 Anticorps:
anti-Human Tumor Protein p73 Anticorps:
anti-Mouse (Murine) Tumor Protein p73 Anticorps:
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Human Monoclonal Tumor Protein p73 Primary Antibody pour ChIP, CyTOF - ABIN4343232
Shimodaira, Yoshioka-Yamashita, Kolodner, Wang: Interaction of mismatch repair protein PMS2 and the p53-related transcription factor p73 in apoptosis response to cisplatin. dans Proceedings of the National Academy of Sciences of the United States of America 2003
Show all 49 Pubmed References
Human Monoclonal Tumor Protein p73 Primary Antibody pour ChIP, ICC - ABIN252619
Costanzo, Merlo, Pediconi, Fulco, Sartorelli, Cole, Fontemaggi, Fanciulli, Schiltz, Blandino, Balsano, Levrero: DNA damage-dependent acetylation of p73 dictates the selective activation of apoptotic target genes. dans Molecular cell 2002
Show all 28 Pubmed References
Human Polyclonal Tumor Protein p73 Primary Antibody pour IHC (p), IP - ABIN151880
Levy, Adamovich, Reuven, Shaul: The Yes-associated protein 1 stabilizes p73 by preventing Itch-mediated ubiquitination of p73. dans Cell death and differentiation 2007
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Human Monoclonal Tumor Protein p73 Primary Antibody pour IP, WB - ABIN967629
Jost, Marin, Kaelin: p73 is a simian [correction of human] p53-related protein that can induce apoptosis. dans Nature 1997
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Human Polyclonal Tumor Protein p73 Primary Antibody pour IHC - ABIN966779
Yuan, Shioya, Ishiko, Sun, Gu, Huang, Lu, Kharbanda, Weichselbaum, Kufe: p73 is regulated by tyrosine kinase c-Abl in the apoptotic response to DNA damage. dans Nature 1999
Human Polyclonal Tumor Protein p73 Primary Antibody pour IF (p), IHC (p) - ABIN675279
Liu, Yang, Jing, Ren, Wei, Zhang, Zhang, Duan, Zhou, Sun: Silica nanoparticle exposure inducing granulosa cell apoptosis and follicular atresia in female Balb/c mice. dans Environmental science and pollution research international 2018
Human Monoclonal Tumor Protein p73 Primary Antibody pour WB - ABIN252620
Shao, Tanaka, Gribi, Yu: Thioredoxin-related regulation of NO/NOS activities. dans Annals of the New York Academy of Sciences 2002
Human Monoclonal Tumor Protein p73 Primary Antibody pour GS, ICC - ABIN269466
King, Reddi, Ponnamperuma, Gerdes, Weinberg: Dysregulated ΔNp63α negatively regulates the maspin promoter in keratinocytes via blocking endogenous p73 binding. dans Molecular carcinogenesis 2014
Tumor protein p73 (TAp73) and kallmann syndrome 1 sequence (Montrer KAL1 Anticorps) protein (KAI1 (Montrer CD82 Anticorps)) expression levels are positively correlated in colorectal cancer.
the present study verified a novel molecular link between miRNA663b and TP73, and indicated that miRNA663b may be a critical therapeutic target in breast cancer.
role of p73 in autophagy induction under nitrosative stress in K562 cells
DeltaNp73 was abundantly expressed in the atopic dermatitis epidermis and increased the release of TSLP (Montrer TSLP Anticorps) via NF-kappaB (Montrer NFKB1 Anticorps) activation.
PRIMA-1 could cause the demethylation of TP73, through DNMT1 (Montrer DNMT1 Anticorps) depletion, to subsequently enhance the unfolded protein response
Data found that P73 G4C14-to-A4T14 polymorphism was significantly associated with non-small cell lung cancer risk in Chinese population.
In p53 (Montrer TP53 Anticorps)-deficient breast cancers, compensatory mechanism of NFkB repression by p63 (Montrer RPE65 Anticorps) and p73 during genotoxic stress could lead to complex effects that would influence all aspects of tumor progression.
DeltaNp73 had no leukemic transformation capacity by itself and apparently did not cooperate with the PML (Montrer PML Anticorps)/RARA (Montrer RARA Anticorps) fusion protein to induce a leukemic phenotype in a murine BM transplantation model.
In colorectal tumor cells RPL26 (Montrer RPL26 Anticorps) regulates p73 expression via two distinct mechanisms: protein stability and mRNA translation.
Data show that a dominant-negative effect is widely spread within the p53 (Montrer TP53 Anticorps)/p63 (Montrer RPE65 Anticorps)/p73 family as all p53 (Montrer TP53 Anticorps) loss-of-function hotspot mutants and several of the isoforms of p53 (Montrer TP53 Anticorps) and p73 tested exhibit a dominant-negative potential.
p73 is required for appropriate BMP-induced mesenchymal-to-epithelial transition during somatic cell reprogramming.
P73 (Montrer ARHGAP24 Anticorps) role in differentiating stem cells.RASSF1A promotes a YAP (Montrer YAP1 Anticorps)-p73 (Montrer ARHGAP24 Anticorps) transcriptional programme that enables differentiation.
Absence of TAp73 leads to activation of TGF-beta (Montrer TGFB1 Anticorps) signaling through a Sma (Montrer ACTA2 Anticorps) and Mad-related proteins-independent pathway, favoring oncogenic transforming growth factor-beta effects and epithelial-to-mesenchymal transition.
Findings reinforce the role of TAp73 as tumor suppressor gene and indicate that the regulation of cellular metabolism by TAp73 contributes to its tumor suppressor function.
cells expressing both p63 (Montrer CKAP4 Anticorps) and p73 (Montrer ARHGAP24 Anticorps) exist in mouse epidermis and hair follicle and that hetero-tetramer complexes can be detected by immunoprecipitation in differentiating keratinocytes.
both p53 (Montrer TP53 Anticorps) and p73 (Montrer ARHGAP24 Anticorps) are critical in apoptosis induced by DNA damage and differentiation.
New function of p73 (Montrer ARHGAP24 Anticorps), independent of p53 (Montrer TP53 Anticorps), in the neurogenic architecture of the SVZ of rodent brain.
these results therefore highlight an unanticipated role for p53 (Montrer TP53 Anticorps) family proteins in a regulatory network that integrates essential Wnt (Montrer WNT2 Anticorps)-Tcf (Montrer HNF4A Anticorps) and nodal-Smad (Montrer SMAD1 Anticorps) inputs.
TAp73 as necessary and sufficient for basal body docking, axonemal extension, and motility during the differentiation of Motile multiciliated cell progenitors.
p73 (Montrer ARHGAP24 Anticorps) drives multiciliogenesis, both through transcriptional activation of a master ciliogenesis transcription factor FoxJ1 (Montrer FOXJ1 Anticorps) and through regulation of multiple genes central to ciliogenesis.
This gene encodes a member of the p53 family of transcription factors involved in cellular responses to stress and development. It maps to a region on chromosome 1p36 that is frequently deleted in neuroblastoma and other tumors, and thought to contain multiple tumor suppressor genes. The demonstration that this gene is monoallelically expressed (likely from the maternal allele), supports the notion that it is a candidate gene for neuroblastoma. Many transcript variants resulting from alternative splicing and/or use of alternate promoters have been found for this gene, but the biological validity and the full-length nature of some variants have not been determined.
transformation related protein 73
, tumor protein p73
, tumor protein p73-like
, p53-like transcription factor
, p53-related protein