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In vertebrates, the genes encoding the class of transcription factors called homeobox genes are found in clusters named A, B, C, and D on four separate chromosomes. De plus, nous expédions HOXA9 Protéines (10) et et beaucoup plus de produits pour cette protéine.
Showing 10 out of 121 products:
Human Polyclonal HOXA9 Primary Antibody pour ICC, IF - ABIN4319699
Molino, Jabès, Bonnet, Gaudin, Bernard, Benech, Khrestchatisky: Gene expression comparison reveals distinct basal expression of HOX members and differential TNF-induced response between brain- and spinal cord-derived microvascular endothelial cells. dans Journal of neuroinflammation 2016
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Human Monoclonal HOXA9 Primary Antibody pour IF, ELISA - ABIN561318
Storlie, Jackson, Hutchinson, Grose: Delayed biosynthesis of varicella-zoster virus glycoprotein C: upregulation by hexamethylene bisacetamide and retinoic acid treatment of infected cells. dans Journal of virology 2006
Cow (Bovine) Polyclonal HOXA9 Primary Antibody pour WB - ABIN2778526
Whelan, Ludwig, Bertrand: HoxA9 induces insulin-like growth factor-1 receptor expression in B-lineage acute lymphoblastic leukemia. dans Leukemia 2008
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Mouse (Murine) Polyclonal HOXA9 Primary Antibody pour ELISA, WB - ABIN4319694
Hu, Fong, Ferrell, Largman, Shen: HOXA9 modulates its oncogenic partner Meis1 to influence normal hematopoiesis. dans Molecular and cellular biology 2009
MiR (Montrer MLXIP Anticorps)-182/HOXA9 was involved in the process of RUNX3 (Montrer RUNX3 Anticorps)-mediated GC tumor growth.
The present study demonstrated that miR (Montrer MLXIP Anticorps)-139-5p, functioning as a suppressor in the tumorigenesis process, suppressed oral squamous carcinoma cells mobility by targeting HOXA9 directly.
NHA9 (NUP98 (Montrer NUP98 Anticorps)-HOXA9 fusion protein) deregulates the expression of key leukemic genes, including MEIS1 (Montrer MEIS1 Anticorps)-HOXA9-PBX3 complex, through the enhancer binding and the direct interaction of the fusion protein with HDAC (Montrer HDAC3 Anticorps) and p300 (Montrer EP300 Anticorps) transcriptional regulators
Results show that a combination of HOXA9 and HOXA10 (Montrer HOXA10 Anticorps) promoter methylation markers is significantly associated with the prognosis of breast cancer patients a subtype-independent manner.
knockdown of HOXA9 abrogated NAC1induced drug resistance.
HOXA9 expression is upregulated in three-dimensional organotypic culture of claudin-low breast cancer cells.
HOXA9 transcriptionally regulated EPHB4 (Montrer EPHB4 Anticorps) expression to modulate trophoblasts migration and invasion, which may suggest a contribution of HOXA9-EPHB4 (Montrer EPHB4 Anticorps) in the poor placentation in the pathogenesis of preeclampsia.
Overexpression of HOXA4 (Montrer HOXA4 Anticorps) and HOXA9 contributes to self-renewal and overpopulation of stem cells in colorectal cancers.
HOTTIP/WDR5 (Montrer WDR5 Anticorps)/HOXA9/Wnt (Montrer WNT2 Anticorps) axis contributes to pancreatic tumor stem cell stemness.
Beta-catenin (Montrer CTNNB1 Anticorps) regulates expression of downstream targets of a key transcriptional memory gene, Hoxa9 that is highly enriched in myeloid leukemia (Montrer BCL11A Anticorps) cancer stem cells and helps sustain leukemic self-renewal.
The analysis points to a critical role for Hoxa9 and PU.1 in distal regulation of c-myb (Montrer MYB Anticorps) expression in murine myeloid cells during iL-6 (Montrer IL6 Anticorps)-induced cell differentiation.
the Hoxa9- and Meis1 (Montrer MEIS1 Anticorps)-associated upregulation of Flt3 (Montrer FLT3 Anticorps) is a passive event with regard to leukemia development in mice and with limited relevance to the AML (Montrer RUNX1 Anticorps) pathology.
the cohesin complex regulates PRC2 targeting to silence Hoxa7 and Hoxa9 and negatively regulate self-renewal. Our studies identify a novel epigenetic mechanism underlying leukemogenesis in AML patients with cohesin mutations.
data delineate an altered epigenetic stress response in activated satellite cells from aged mice, which limits satellite cell function and muscle regeneration by Hoxa9-dependent activation of developmental pathways
Pbx3 contributes to Hoxa9 leukemogenesis through stabilization of the Meis1 (Montrer MEIS1 Anticorps) protein.
IGF-1 (Montrer IGF1 Anticorps) is a direct HOXA9 target important for hematopoietic transformation.
deficiency of tumor suppressor prep1 accelerates the onset of meis1 (Montrer MEIS1 Anticorps)- hoxa9 leukemogenesis
Hoxa9 collaborates with E2A (Montrer TCF3 Anticorps)-PBX1 (Montrer PBX1 Anticorps) in mouse B cell leukemia in association with Flt3 (Montrer FLT3 Anticorps) activation and decrease of B cell gene expression.
results suggest a previously unidentified role for C/EBPalpha (Montrer CEBPA Anticorps) in maintaining the proliferation required for Hoxa9/Meis1 (Montrer MEIS1 Anticorps)-mediated leukemogenesis
In vertebrates, the genes encoding the class of transcription factors called homeobox genes are found in clusters named A, B, C, and D on four separate chromosomes. Expression of these proteins is spatially and temporally regulated during embryonic development. This gene is part of the A cluster on chromosome 7 and encodes a DNA-binding transcription factor which may regulate gene expression, morphogenesis, and differentiation. This gene is highly similar to the abdominal-B (Abd-B) gene of Drosophila. A specific translocation event which causes a fusion between this gene and the NUP98 gene has been associated with myeloid leukemogenesis. Read-through transcription exists between this gene and the upstream homeobox A10 (HOXA10) gene.
, homeobox protein Hox-A9
, XIHbox 6
, homeobox protein Hox-1G
, homeodomain protein HOXA9
, homeo box A9
, homeobox protein Hox-1.7
, Homeobox gene A7
, homeobox protein Hox-1.1
, homeobox protein Hox-A7
, homeobox protein R5
, homeobox protein A9