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Human Polyclonal PRAM1 Primary Antibody pour ELISA, WB - ABIN257467
Moog-Lutz, Peterson, Lutz, Eliason, Cavé-Riant, Singer, Di Gioia, Dmowski, Kamens, Cayre, Koretzky: PRAM-1 is a novel adaptor protein regulated by retinoic acid (RA) and promyelocytic leukemia (PML)-RA receptor alpha in acute promyelocytic leukemia cells. dans The Journal of biological chemistry 2001
A yopH mutant survived better in the absence of neutrophils, indicating that neutrophil inactivation by YopH by targeting PRAM-1/SKAP-HOM (Montrer SKAP2 Anticorps) and SLP-76 (Montrer LCP2 Anticorps)/Vav (Montrer VAV1 Anticorps)/PLCgamma2 (Montrer PLCG2 Anticorps) signaling hubs may be critical for Yersinia survival.
autophagy contributes to the anti-apoptotic function of the PML (Montrer PML Anticorps)-RARalpha (Montrer RARA Anticorps) protein through inhibiting the Akt (Montrer AKT1 Anticorps)/mTOR (Montrer FRAP1 Anticorps) pathway
Transgenic mice expressing PML-RAR alpha (Montrer RARA Anticorps) develop acute promyelocytic leukemia (Montrer PML Anticorps) with long latency, low penetrance, and acquired cytogenetic abnormalities.
Our results show that the pretreatment PML (Montrer PML Anticorps)-RARA (Montrer RARA Anticorps) molecular burden could therefore be used to improve risk stratification in order to develop more individualized treatment regimens for high-risk APL (Montrer FASL Anticorps) cases.
The 3-plex RT-qPCR assay is a specific, sensitive, stable, and cost-effective method that can be used for the rapid diagnosis and treatment monitoring of acute promyelocytic leukemia (Montrer PML Anticorps) with PML (Montrer PML Anticorps)-RARa (Montrer RARA Anticorps)
The MIR125B1-mediated blockade of PML (Montrer PML Anticorps)-RARA (Montrer RARA Anticorps) proteolysis was regulated via an autophagy-lysosomal pathway, contributing to the inhibition of acute promyelocytic leukemia (Montrer PML Anticorps) differentiation.
PRAM-1 is required for optimal integrin-dependent neutrophil function.
Caspase 3 (Montrer CASP3 Anticorps)-cleaved SH3 domain (Montrer ITSN1 Anticorps) of HIP-55 (Montrer DBNL Anticorps) is likely involved in PRAM-1-mediated JNK (Montrer MAPK8 Anticorps) activation upon arsenic trioxide-induced differentiation of NB4 cells.
The use of reverse-transcription polymerase chain reaction for the detection of the PML (Montrer PML Anticorps)-RARA (Montrer RARA Anticorps) and RARA (Montrer RARA Anticorps)-PML (Montrer PML Anticorps) fusion genes that allow for monitoring of acute myelocytic leukemia.
PML (Montrer PML Anticorps)-RARalpha (Montrer RARA Anticorps) fusion transcripts have been shown to be useful markers for establishing the diagnosis and for monitoring the response to treatment of acute promyelocytic leukemia (Montrer PML Anticorps).
Interaction of PRAM-1 and hSH3 domains of adhesion and degranulation promoting adapter protein (Montrer TOLLIP Anticorps) (ADAP (Montrer FYB Anticorps)) with phosphatidylcholine (Montrer SGMS2 Anticorps)-containing liposomes is observed upon incorporation of phosphatidylserine or phosphoinositides into the membrane bilayer.
hidden abnormalities and novel disease-related genomic changes occur in t(15;17)translocation in acute promyelocytic leukemia (Montrer PML Anticorps) formation of PML (Montrer PML Anticorps)-RARA (Montrer RARA Anticorps) gene
The protein encoded by this gene is similar to FYN binding protein (FYB/SLAP-130), an adaptor protein involved in T cell receptor mediated signaling. This gene is expressed and regulated during normal myelopoiesis. The expression of this gene is induced by retinoic acid and is inhibited by the expression of PML-RARalpha, a fusion protein of promyelocytic leukemia (PML) and the retinoic acid receptor-alpha (RARalpha).
PML-RARA regulated adaptor molecule 1
, PML-RARA-regulated adapter molecule 1-like
, PML-RARA-regulated adapter molecule 1
, PML-RARA target gene encoding an Adaptor Molecule-1
, PML-RAR alpha-regulated adaptor molecule 1